Zobrazeno 1 - 10
of 19
pro vyhledávání: '"Yue-Qin Qin"'
Autor:
Chainllie Young, Kevin A. Roth, Barbara J. Klocke, Tim West, David M. Holtzman, Joann Labruyere, Yue-Qin Qin, Krikor Dikranian, John W. Olney
Publikováno v:
Neurobiology of Disease, Vol 20, Iss 2, Pp 608-614 (2005)
Acute, transient exposure to ethanol causes a widespread pattern of caspase-3 activation and neuroapoptosis in the developing rodent brain. To determine whether caspase-3 activation is an essential step in ethanol-induced developmental neuroapoptosis
Externí odkaz:
https://doaj.org/article/ddbf78aae8964682938b87f19d085287
Publikováno v:
IEEE Systems Journal. 16:5035-5043
Autor:
Shi, Wei-Kang, Deng, Rui-Cheng, Wang, Peng-Fei, Yue, Qin-Qin, Liu, Qi, Ding, Kun-Ling, Yang, Mei-Hui, Zhang, Hong-Yu, Gong, Si-Hua, Deng, Min, Liu, Wen-Run, Feng, Qiu-Ju, Xiao, Zhu-Ping, Zhu, Hai-Liang
Publikováno v:
In Bioorganic & Medicinal Chemistry 1 October 2016 24(19):4519-4527
Autor:
Vesna Jevtovic-Todorovic, Yue-Qin Qin, Haihui Wang, Tatyana Tenkova, Joann Labruyere, John W. Olney, Chainllie Young
Publikováno v:
British Journal of Pharmacology. 146:189-197
Recently, it was reported that anesthetizing infant rats for 6 h with a combination of anesthetic drugs (midazolam, nitrous oxide, isoflurane) caused widespread apoptotic neurodegeneration in the developing brain, followed by lifelong cognitive defic
Ethanol-induced neuroapoptosis in the developing rodent cerebellum and related brain stem structures
Publikováno v:
Developmental Brain Research. 155:1-13
For three decades since the fetal alcohol syndrome (FAS) was first described, researchers have been keenly interested in understanding the mechanism(s) by which ethanol damages or disrupts development of the human fetal brain. It has been reported re
Autor:
John W. Olney, Junjeong Choi, Kevin A. Roth, Tatyana Tenkova, David M. Holtzman, Chainllie Young, Yue-Qin Qin, Joann Labruyere, Barbara J. Klocke
Publikováno v:
Cell Death & Differentiation. 10:1148-1155
A single episode of ethanol intoxication triggers widespread apoptotic neurodegeneration in the infant rat or mouse brain. The cell death process occurs over a 6-16 h period following ethanol administration, is accompanied by a robust display of casp
Autor:
Chrysanthy Ikonomidou, Yue-Qin Qin, Masahiko Ishimaru, Krikor Dikranian, Joann Labruyere, John W. Olney, Tatyana Tenkova
Publikováno v:
Neurobiology of Disease, Vol 8, Iss 3, Pp 359-379 (2001)
Apoptosis is a word originally introduced by Kerr, Wyllie, and colleagues for a cell death process they defined in terms of its ultrastructural appearance in nonneuronal cells from various tissues. There are very few studies providing detailed ultras
Publikováno v:
Pediatric Research. 39:1020-1027
Recent evidence implicates the endogenous excitatory neurotransmitters, glutamate (Glu) and aspartate, in the pathophysiology of traumatic injury in the adult CNS, but it is not known whether similar excitotoxic mechanisms mediate traumatic injury in
Publikováno v:
Journal of Neuropathology and Experimental Neurology. 55:211-224
A superoxide dismutase 1 (SOD-1) genetic defect has been identified in familial amyotrophic lateral sclerosis (ALS) and motor neuron degeneration has been described in SOD-1 transgenic mice. Because an excitotoxic mechanism has been implicated in ALS
Autor:
John W. Olney, Philip V. Bayly, Erin E. Black, Chainllie Young, Joann Labruyere, Krikor Dikranian, Yue-Qin Qin
Closed head injury to the developing rat brain causes an acute excitotoxic lesion and axonal disruption at the impact site followed by a delayed pattern of apoptotic damage at various distant sites. Using an electromagnetic impact device to deliver a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5c6a4380572189814c0830ce25960e69
https://europepmc.org/articles/PMC2376971/
https://europepmc.org/articles/PMC2376971/