Zobrazeno 1 - 10
of 125
pro vyhledávání: '"Yuchang Fu"'
Publikováno v:
Journal of Lipid Research, Vol 46, Iss 7, Pp 1369-1379 (2005)
Adiponectin is secreted from adipocytes, and low circulating levels have been epidemiologically associated with obesity, insulin resistance, type 2 diabetes, and cardiovascular disease. To investigate whether adiponectin could exert autocrine effects
Externí odkaz:
https://doaj.org/article/696401c1ca04405ba6f8bff617b3e72d
Publikováno v:
Journal of Lipid Research, Vol 44, Iss 7, Pp 1315-1321 (2003)
We have previously shown that LDL-containing immune complexes (LDL-ICs) induce up-regulation of LDL receptor (LDLR) expression in human macrophages. The present study further investigated the molecular mechanisms leading to LDLR up-regulation by LDL-
Externí odkaz:
https://doaj.org/article/464d62fc43674ffe98d04f6772485409
Autor:
Weina Gao, Yuchang Fu, Cong Yu, Shunke Wang, Yuchao Zhang, Chen Zong, Tongfu Xu, Yong Liu, Xia Li, Xiangdong Wang
Publikováno v:
PLoS ONE, Vol 9, Iss 3, p e91462 (2014)
BACKGROUND: NR4A3/NOR-1 is a member of the NR4A orphan nuclear receptor subfamily, which contains early response genes that sense and respond to a variety of stimuli in the cellular environment. The role of NR4A3 in insulin expression in pancreatic b
Externí odkaz:
https://doaj.org/article/ca7d8ce67d0c4a0ebc9f459db4a344a1
Publikováno v:
Journal of Lipid Research, Vol 41, Iss 12, Pp 2017-2023 (2000)
The adipocyte lipid-binding protein (ALBP/aP2) belongs to a multigene family of fatty acid and retinoid transport proteins. This protein is abundantly expressed in the cytoplasm and nuclear region of adipocytes and is postulated to serve as a lipid s
Externí odkaz:
https://doaj.org/article/fd2a7a23c2ca4bee8156bd6e33021f23
Autor:
Guo-Jun Zhao, Shi-Lin Tang, Yun-Cheng Lv, Xin-Ping Ouyang, Ping-Ping He, Feng Yao, Wu-Jun Chen, Qian Lu, Yan-Yan Tang, Min Zhang, Yuchang Fu, Da-Wei Zhang, Kai Yin, Chao-Ke Tang
Publikováno v:
PLoS ONE, Vol 8, Iss 9, p e74782 (2013)
ATP-binding cassette transporter A1 (ABCA1) is critical in exporting cholesterol from macrophages and plays a protective role in the development of atherosclerosis. The purpose of this study was to investigate the effects of betulinic acid (BA), a pe
Externí odkaz:
https://doaj.org/article/65783c77405b48c1aff7e5965ed1f642
Autor:
Yuchang Fu1 yfu@uab.edu, Liehong Luo1 lhluo@uab.edu, Nanlan Luo1 nluo@uab.edu, Garvey, W. Timothy1,2 garveyt@uab.edu
Publikováno v:
Nutrition & Metabolism. 2006, Vol. 3, p28-10. 10p.
Autor:
Elizabeth B. Heywood, R. Grace Walton, Helliner S. Hill, W. Timothy Garvey, Qinglin Yang, Ling Tian, Xiaolin Zhu, Yuchang Fu, Jiarong Liu, Jian Liu, Dennis Bruemmer
Publikováno v:
American Journal of Physiology-Endocrinology and Metabolism. 311:E69-E81
The NR4A orphan nuclear receptors function as early response genes to numerous stimuli. Our laboratory has previously demonstrated that overexpression of NR4A3 (NOR-1, MINOR) in 3T3-L1 adipocytes enhances insulin-stimulated glucose uptake. To assess
Autor:
Minsung Kang, Teayoun Kim, Qinglin Yang, Dennis Steverson, W. Timothy Garvey, W. John Garvey, Wei Zhang, Ling Tian, Yuchang Fu, Elizabeth Ma, Nanlan Luo, Mengrui Wu, Ravi H. Jariwala
Publikováno v:
Diabetes
In the current study, we used muscle-specific TRIB3 overexpressing (MOE) and knockout (MKO) mice to determine whether TRIB3 mediates glucose-induced insulin resistance in diabetes and whether alterations in TRIB3 expression as a function of nutrient
Publikováno v:
Metabolic Syndrome and Related Disorders. 14:7-15
Insulin resistance is central in the pathophysiology of cardiometabolic disease; however, common mechanisms that explain the parallel development of both type 2 diabetes and atherosclerosis have not been elucidated. We have previously shown that trib
Publikováno v:
Metabolic syndrome and related disorders. 16(2)
Background: Insulin resistance disrupts metabolic processes and leads to various chronic disease states such as diabetes and metabolic syndrome (MetS). However, the mechanism linking insulin resistance with cardiometabolic disease pathophysiology is