Zobrazeno 1 - 10
of 14
pro vyhledávání: '"Yu-Ye Wen"'
Autor:
Jian Chen, Shih-Lan Hsu, Chun-Hui Su, Changju Qu, Mong Hong Lee, Chiehlin Teng, Ken Parreno, Sai-Ching Yeung, Liem Phan, Yuwen Xue, Yun-Chi Hsieh, Yu-Ye Wen, Huamin Wang, Ping Chieh Chou, Christopher Gully
Publikováno v:
Cancer Hallmarks. 1:3-17
Autor:
Shaofan Weng, Hyun Ho Choi, Douglas Webb, Ismael Samudio, Nibal Rizk, John D. Hazle, Enrique Fuentes-Mattei, Mouhammed Amir Habra, Lajos Pusztai, Sai Ching J. Yeung, Stephen Skerl, Wei Tse Yang, Marc S. Ramirez, Jaehyuk Lee, Yaling Huang, Huamin Wang, Xin Lin, Ping Chieh Chou, Yu Ye Wen, James A. Bankson, Chun Hui Su, Dianna D. Cody, Charles V. Kingsley, Kenneth Parreno, Marzenna Blonska, Yun Wu, Guermarie Velazquez-Torres, Andrew Elliott, Jian Chen, Colin Carlock, Jorge Delacerda, Christine Y. Shiang, Hua Wang, Mong Hong Lee, Thuy M. Vu, Brian C. Grabiner, Lei Li, Christopher Gully, Xuefeng Xia, Yiping Shao, Liem Phan, Ji-Hyun Shin, Zhongguo Zhou, Yun Chih Hsieh, Aijun Zhang, Chieh Tseng, Yongxing Wang, Edward Wang
Publikováno v:
Nature communications
Summary Extensive reprogramming of cellular energy metabolism is a hallmark of cancer. Despite its importance, the molecular mechanism controlling this tumour metabolic shift remains not fully understood. Here we show that 14-3-3σ regulates cancer m
Autor:
Keith Fournier, Yu Ye Wen, Heng Yin Yang, Mong Hong Lee, Ruiying Zhao, Yu Li Lin, Christine Laronga, Yun Qiu, Jose I. Diaz, Huiling Yang
Publikováno v:
Cancer Research. 66:3096-3105
14-3-3 σ is induced by tumor suppressor protein p53 in response to DNA damage. p53 can directly transactivate the expression of 14-3-3 σ to cause a G2 cell cycle arrest when cell DNA is damaged. The expression of 14-3-3 σ protein is down-regulated
Publikováno v:
Molecular and Cellular Biology. 23:7096-7107
The 14-3-3 sigma (sigma) protein, a negative regulator of the cell cycle, is a human mammary epithelium-specific marker that is downregulated in transformed mammary carcinoma cells. It has also been identified as a p53-inducible gene product involved
Autor:
Agda Karina Eterovic, Li Huang, George A. Calin, Gal Markel, Denise Brooks, Yu Ye Wen, A. Gordon Robertson, Menashe Bar-Eli, Aaron K. Mobley, Isaiah J. Fidler, Patrick Hwu, Anil K. Sood, Cristina Ivan, Mayra E. Vasquez, Ka Ming Nip, Guermarie Velazquez-Torres, Jeffrey E. Gershenwald, Kelsey Zhu, Einav Shoshan, Ahmad Salameh, Maribel Mosqueda, Sun Jin Kim, Steven J.M. Jones, Inanc Birol, Ho Jeong Lee, Russell R. Braeuer, Takafumi Kamiya
Publikováno v:
Nature cell biology. 17(3)
Although recent studies have shown that adenosine-to-inosine (A-to-I) RNA editing occurs in microRNAs (miRNAs), its effects on tumour growth and metastasis are not well understood. We present evidence of CREB-mediated low expression of ADAR1 in metas
Publikováno v:
Virology. 275:258-262
Carboxyfullerene (C60) is known as a photosensitizer for virus inactivation. Its regioisomer with C3 symmetry, named the C3 isomer, could also inactivate the dengue-2 virus without light when the dose of C3 isomer was increased to 40 μM, indicating
Autor:
Christopher R. Vakoc, Gerd A. Blobel, Richard A. Gibbs, Yu-Ye Wen, Anil K. Rustgi, Cameron N. Johnstone
Publikováno v:
Cancer genetics and cytogenetics. 189(2)
Large-scale sequencing of cancer genomes holds great promise in identifying gene mutations that drive human cancer progression. A study was reported recently in which the coding region of 13,023 genes was systematically sequenced within a panel color
Autor:
Christine Laronga, H. B. Wu, Yu Ye Wen, Yun Zhang, Heng Yin Yang, Mong Hong Lee, Keith Fournier, Jose I. Diaz, Rui-Xun Zhao, Heng-Yin Yang
Publikováno v:
Oncogene. 25(33)
The 14-3-3sigma (sigma) protein is a human cancer marker downregulated in various tumors, but its function has not been fully established. 14-3-3sigma is a negative regulator of cell cycle when overexpressed, but it is not clear whether 14-3-3sigma r
Autor:
Phillip Chen, Erica Fang, Kent Osborne, Ganka Douglas, Yanchun Li, Yu-Ye Wen, Condie Edwin Carmack, Marilyn M. Li
Publikováno v:
Cancer Genetics. 205:422
Publikováno v:
Journal of Clinical Oncology. 30:10598-10598
10598 Background: The emergence of next-generation sequencing (NGS) technologies has significantly accelerated the identification of cancer-causing mutations and the development of personalized cancer care. However, the clinical application of these