Zobrazeno 1 - 10
of 18
pro vyhledávání: '"Yu-Wang Chang"'
Publikováno v:
Biomedicines, Vol 11, Iss 11, p 2891 (2023)
The Cav3.2 T-type calcium channel is implicated in various pathological conditions, including cardiac hypertrophy, epilepsy, autism, and chronic pain. Phosphorylation of Cav3.2 by multiple kinases plays a pivotal role in regulating its calcium channe
Externí odkaz:
https://doaj.org/article/666847f33b254a12976cc7d0b8911ebd
Autor:
Hsiao-hui Yeh, Yao-Ming Chang, Yu-Wang Chang, Mei-Yeh Jade Lu, Yi-Hua Chen, Chia-Che Lee, Chien-Chang Chen
Publikováno v:
Journal of Molecular and Cellular Cardiology Plus, Vol 1, Iss , Pp 100011- (2022)
Background: Cardiac pressure overload induces cardiac hypertrophy and eventually leads to heart failure. One distinct feature of pathological cardiac hypertrophy is fetal-gene re-expression, but not every cardiomyocyte exhibits fetal gene re-expressi
Externí odkaz:
https://doaj.org/article/42466b180bdb42bba77f30e3c1962871
Autor:
Volker Herzig, Yong-Cyuan Chen, Yanni K.-Y. Chin, Zoltan Dekan, Yu-Wang Chang, Hui-Ming Yu, Paul F. Alewood, Chien-Chang Chen, Glenn F. King
Publikováno v:
Biomedicines, Vol 10, Iss 5, p 1066 (2022)
Inhibition of T-type calcium channels (CaV3) prevents development of diseases related to cardiovascular and nerve systems. Further, knockout animal studies have revealed that some diseases are mediated by specific subtypes of CaV3. However, subtype-s
Externí odkaz:
https://doaj.org/article/19efb67a550341f79157382ee8451a40
Autor:
Yao-Ming Chang, Li Ling, Ya-Ting Chang, Yu-Wang Chang, Wen-Hsiung Li, Arthur Chun-Chieh Shih, Chien-Chang Chen
Publikováno v:
Scientific Reports, Vol 7, Iss 1, Pp 1-13 (2017)
Abstract Pathological cardiac hypertrophy, a dynamic remodeling process, is a major risk factor for heart failure. Although a number of key regulators and related genes have been identified, how the transcription factors (TFs) dynamically regulate th
Externí odkaz:
https://doaj.org/article/e6c3c3b0dd254a86b4559886f5ceded0
Autor:
Yu-Wang Chang, 張裕旺
102
Pressure-overload stress to the heart causes pathological cardiac hypertrophy, which increases the risk of cardiac morbidity and mortality. However, the detailed signaling pathways induced by pressure overload remain unclear. Here we used ph
Pressure-overload stress to the heart causes pathological cardiac hypertrophy, which increases the risk of cardiac morbidity and mortality. However, the detailed signaling pathways induced by pressure overload remain unclear. Here we used ph
Externí odkaz:
http://ndltd.ncl.edu.tw/handle/93200031200689183623
Publikováno v:
The FEBS journalReferences. 289(7)
Loss of the mitochondrial fission enzyme dynamin-related protein 1 (Drp1) in cardiomyocytes results in energy shortage and heart failure. We aim to understand the intracellular signal pathway and extracellular factors regulating Drp1 phosphorylation
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7e5a2cdd51c10060e470092bb557d047
https://pubmed.ncbi.nlm.nih.gov/34739186
https://pubmed.ncbi.nlm.nih.gov/34739186
Autor:
Yu-Wang Chang, Chien-Chang Chen, Li Ling, Ya-Ting Chang, Wen-Hsiung Li, Yao-Ming Chang, Arthur Chun-Chieh Shih
Publikováno v:
Scientific Reports
Scientific Reports, Vol 7, Iss 1, Pp 1-13 (2017)
Scientific Reports, Vol 7, Iss 1, Pp 1-13 (2017)
Pathological cardiac hypertrophy, a dynamic remodeling process, is a major risk factor for heart failure. Although a number of key regulators and related genes have been identified, how the transcription factors (TFs) dynamically regulate the associa
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e35373af5d5bbbb34c0914d09a2a07cd
https://doi.org/10.1038/s41598-017-07981-4
https://doi.org/10.1038/s41598-017-07981-4
Publikováno v:
Molecular & Cellular Proteomics. 12:3094-3107
Pressure-overload stress to the heart causes pathological cardiac hypertrophy, which increases the risk of cardiac morbidity and mortality. However, the detailed signaling pathways induced by pressure overload remain unclear. Here we used phosphoprot
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1beb097d3361f5a884a881c7b05e5814
https://doi.org/10.1074/mcp.m113.027649
https://doi.org/10.1074/mcp.m113.027649
Autor:
Chi-Huey Wong, An Chen, Su-Chin Huang, Feng-Yee Chang, Yu-Ju Chen, Jenn-Han Chen, Yu-Wang Chang, Ko-Yi Chien, Tzong-Shi Chiueh, Chen-Wen Yao
Publikováno v:
Proceedings of the National Academy of Sciences
We have investigated the plasma proteome by using 2D gel electrophoresis and MS from patients with severe acute respiratory syndrome (SARS). A complete proteomic analysis was performed on four patients with SARS in different time courses, and a total
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a16b915badbe2a0b4d3621e5edaae518
https://doi.org/10.1073/pnas.0407992101
https://doi.org/10.1073/pnas.0407992101