Zobrazeno 1 - 10 of 27 pro vyhledávání: '"Younss Ait Mou"'
1
Towards the Mixed-Reality Platform for the Learning of Children with Autism Spectrum Disorder (ASD): A Case Study in Qatar
Autor: Kamran Khowaja, Dena Al-Thani, Asma Osman Hassan, Yasmin Abdelaal, Younss Ait Mou, Mohamad Hassan Fadi Hijab
Publikováno v: HCI in Games: Serious and Immersive Games ISBN: 9783030774134
HCI (29)
This virtual classroom has created a lot of challenges for students with limited access to the technology (computer or smartphone) or the Internet and especially for the parents of children with developmental disabilities, including children with aut
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::8000e1b3f72d3c747dd7ccf3ebd6a3c7
https://doi.org/10.1007/978-3-030-77414-1_24
Zobrazit plný text záznamu
2
Bioprinting: A review of processes, materials and applications
Autor: Bengi Yilmaz, Younss Ait Mou, Muammer Koç, Ans Al Rashid, Zafer Evis
Publikováno v: Bioprinting. 23:e00148
3D printing (3DP) processes have evolved from simple prototyping for visual inspection purposes to functional product manufacturing that offers flexibility, customization, scalability, reliability, durability, and relatively high speed. These process
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::f4346ceb0dedf477aec5f967f52e23b3
https://doi.org/10.1016/j.bprint.2021.e00148
Zobrazit plný text záznamu
3
Altered myofilament structure and function in dogs with Duchenne muscular dystrophy cardiomyopathy
Autor: Thomas C. Irving, Bijan Ghaleh, Olivier Cazorla, Younss Ait Mou, Valérie Scheuermann, Alain Lacampagne, Stéphane Blot, Pieter P. de Tombe
Publikováno v: Journal of Molecular and Cellular Cardiology
Journal of Molecular and Cellular Cardiology, Elsevier, 2018, 114, pp.345-353. ⟨10.1016/j.yjmcc.2017.12.008⟩
AimsDuchenne Muscular Dystrophy (DMD) is a disease that mainly affects young mans. It is characterized by striated muscle disorder. DMD is associated with depressed heart pump function resulting from a down regulation of the left ventricular (LV) con
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::24e6068173b15501867811eaa058c74a
https://hal.umontpellier.fr/hal-02376879/document
Zobrazit plný text záznamu
4
Myocardial Infarction-induced N-terminal Fragment of Cardiac Myosin-binding Protein C (cMyBP-C) Impairs Myofilament Function in Human Myocardium*
Autor: Namthip Witayavanitkul, Margaret V. Westfall, Sakthivel Sadayappan, Sudarsan Rajan, Ramzi J. Khairallah, David F. Wieczorek, Jason Sarkey, Ying Ge, Thomas C. Irving, Younss Ait Mou, Pieter P. de Tombe, Diederik W. D. Kuster, Suresh Govindan, Xin-Xin Chen
Publikováno v: The Journal of Biological Chemistry
Journal of Biological Chemistry, 289(13), 8818-8827. American Society for Biochemistry and Molecular Biology Inc.
Witayavanitkul, N, Mou, Y A, Kuster, D W D, Khairallah, R J, Sarkey, J, Govindan, S, Chen, X, Ge, Y, Rajan, S, Wieczorek, D F, Irving, T, Westfall, M V, de Tombe, P P & Sadayappan, S 2014, ' Myocardial Infarction-induced N-terminal Fragment of Cardiac Myosin-binding Protein C ( cMyBP-C) Impairs Myofilament Function in Human Myocardium ', Journal of Biological Chemistry, vol. 289, no. 13, pp. 8818-8827 . https://doi.org/10.1074/jbc.M113.541128
Background: Myocardial infarction (MI) leads to proteolytic cleavage of cMyBP-C (hC0C1f) and decreased contractility. Results: hC0C1f can incorporate into the human cardiac sarcomere, depressing force generation and increasing tension cost. Conclusio
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::76dd2da139ffa465fd15b5ffc5d9f269
http://europepmc.org/articles/PMC3979389
Zobrazit plný text záznamu
5
Titin strain contributes to the Frank-Starling law of the heart by structural rearrangements of both thin- and thick-filament proteins
Autor: Marion L. Greaser, Karen Hsu, Pieter P. de Tombe, Younss Ait-Mou, Gerrie P. Farman, Mohit Kumar, Thomas C. Irving
Publikováno v: Proceedings of the National Academy of Sciences of the United States of America. 113(8)
The Frank-Starling mechanism of the heart is due, in part, to modulation of myofilament Ca(2+) sensitivity by sarcomere length (SL) [length-dependent activation (LDA)]. The molecular mechanism(s) that underlie LDA are unknown. Recent evidence has imp
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e690773757a1ba4e9d22901e4e54d98f
https://pubmed.ncbi.nlm.nih.gov/26858417
Zobrazit plný text záznamu
6
Bradykinin restores left ventricular function, sarcomeric protein phosphorylation, and e/nNOS levels in dogs with Duchenne muscular dystrophy cardiomyopathy
Autor: Nicolas Blanchard-Gutton, Pablo Aguilar, Yves Unterfinger, Alain Berdeaux, Jin Bo Su, Jean Laurent Thibaud, Alain Bizé, Stéphane Blot, Luc Hittinger, Hubert Dabiré, Vassiliki Gouni, Alain Lacampagne, Jean Louis Pouchelon, Olivier Cazorla, Inès Barthélémy, Lucien Sambin, Valérie Chetboul, Bijan Ghaleh, Younss Ait Mou, Carolina Carlos Sampedrano
Publikováno v: Cardiovascular Research
Cardiovascular Research, Oxford University Press (OUP), 2012, 95 (1), pp.86-96. ⟨10.1093/cvr/cvs161⟩
Cardiovascular Research, 2012, 95 (1), pp.86-96. ⟨10.1093/cvr/cvs161⟩
AIMS: Cardiomyopathy is a lethal result of Duchenne muscular dystrophy (DMD), but its characteristics remain elusive. The golden retriever muscular dystrophy (GRMD) dogs produce DMD pathology and mirror DMD patient's symptoms, including cardiomyopath
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a11cf0844c7c732f3bc0e4d2fa6f3d59
https://doi.org/10.1093/cvr/cvs161
Zobrazit plný text záznamu
8
Exploring cardiac biophysical properties
Autor: Christian Bollensdorff, Yacoub Magdi, Olivier Cazorla, Younss Ait Mou, Pieter P. de Tombe
Publikováno v: Global Cardiology Science and Practice
Global Cardiology Science and Practice, 2015, 2015 (1), ⟨10.5339/gcsp.2015.10⟩
Global Cardiology Science & Practice
The heart is subject to multiple sources of stress. To maintain its normal function, and successfully overcome these stresses, heart muscle is equipped with fine-tuned regulatory mechanisms. Some of these mechanisms are inherent within the myocardium
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0cbca819040d7197ddedbd5ec295021c
https://hal.umontpellier.fr/hal-01762169
Zobrazit plný text záznamu
9
Abstract 18852: Myofilament Length Dependent Activation: Role of Titin
Autor: Younss Ait Mou, Marion L. Greaser, Pieter deTombe, Karen H. Hsu, Thomas C. Irving
Publikováno v: Circulation. 130
The cellular basis of the Frank-Starling mechanism is sarcomere length (SL) modulation of myofilament Ca 2+ sensitivity (LDA). The molecular mechanism(s) that underlie LDA are unknown, but recent evidence has implicated the giant protein titin as pos
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::2c64bb45cd2af18c753db0c5ad361011
https://doi.org/10.1161/circ.130.suppl_2.18852
Zobrazit plný text záznamu
10
Protein kinase C‐site phosphorylation of cardiac myosin binding protein‐C decreases cross‐bridge kinetics (1081.5)
Autor: Pieter P. de Tombe, Suresh Govindan, Younss Ait Mou, Thomas L. Lynch, Sakthivel Sadayappan
Publikováno v: The FASEB Journal. 28
Cardiac myosin binding protein-C (cMyBP-C) phosphorylation is necessary for the regulation of sarcomeric structure and myocardial contractility in vivo. Different from skeletal isoforms, cMyBP-C has M-domain phosphorylation motifs in which three seri
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::79ce71ec9a7429c169ccb04d92926ad5
https://doi.org/10.1096/fasebj.28.1_supplement.1081.5
Zobrazit plný text záznamu

Vyhledávací nástroje:

Upřesnit hledání

Omezení vyhledávání
Plný text Recenzováno Digitální knihovna AV ČR
Zdroje