Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Youngeun Yun"'
Autor:
Seung-Yoon Park, Youngeun Yun, Jung-Suk Lim, Mi-Jin Kim, Sang-Yeob Kim, Jung-Eun Kim, In-San Kim
Publikováno v:
Nature Communications, Vol 7, Iss 1, Pp 1-15 (2016)
Phosphatidylserine and its receptors are associated with cell-cell fusion. Here, the authors show the phosphatidylserine receptor stabilin-2 is expressed by muscle cells and plays a vital role in myoblast fusion and post-injury muscle regeneration in
Externí odkaz:
https://doaj.org/article/046a6a0777334fb3beeab11133907710
Autor:
Jung Suk Lim, Mi-Jin Kim, In San Kim, Seung Yoon Park, Jung-Eun Kim, Youngeun Yun, Sang Yeob Kim
Publikováno v:
Nature Communications
Nature Communications, Vol 7, Iss 1, Pp 1-15 (2016)
NATURE COMMUNICATIONS(7)
Nature Communications, Vol 7, Iss 1, Pp 1-15 (2016)
NATURE COMMUNICATIONS(7)
Myoblast fusion is essential for the formation of skeletal muscle myofibres. Studies have shown that phosphatidylserine is necessary for myoblast fusion, but the underlying mechanism is not known. Here we show that the phosphatidylserine receptor sta
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::aa10ce9fe0d326614efdb48fdee5739c
https://doi.org/10.1038/ncomms10871
https://doi.org/10.1038/ncomms10871
Publikováno v:
Biochemical and biophysical research communications. 450(4)
MEGF10 is known to function as a myogenic regulator of satellite cells in skeletal muscle. Mutations in MEGF10 gene cause a congenital myopathy called early onset myopathy, areflexia, respiratory distress and dysphagia (EMARDD). Despite its biologica
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::48cd2bb20e18c7e9b1a1b8282be8e711
https://pubmed.ncbi.nlm.nih.gov/25044114
https://pubmed.ncbi.nlm.nih.gov/25044114
Autor:
Seung Yoon Park, Youngeun Yun, Thoudam Debraj Singh, Jae-sung Bae, Yong Chul Bae, Rang Woon Park, In San Kim
Publikováno v:
FEBS letters. 584(18)
MEGF10 is predominantly expressed in the brain and known to function as a phagocytic receptor. Here, we provide evidence that MEGF10 is involved in the uptake of amyloid-β peptide (Aβ42) in the brain. Overexpression of MEGF10 dramatically increased
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c991b3f51bd42fabe778e9a87b348047
https://pubmed.ncbi.nlm.nih.gov/20828568
https://pubmed.ncbi.nlm.nih.gov/20828568