Zobrazeno 1 - 10
of 26
pro vyhledávání: '"Yoshiko Nakagomi"'
Autor:
Mie Mochizuki, Yoshie Shimura, Kisho Kobayashi, Kanji Sugita, Kenji Ohyama, Rika Kobayashi, Yoshiko Nakagomi
Publikováno v:
Biochemical and Biophysical Research Communications. 389:478-483
Stanniocalcin-1 (STC-1) produced by ovaries endocrinologically targets to mammary glands and is secreted into milk during lactation. The decline of mother's serum level by STC-1 antiserum administration reduced the milk fat content and the pups' body
Autor:
Yoshiko Nakagomi, Hiroyuki Koshiyama, Masaaki Inaba, Kenichi Satomura, Keiichi Ozono, Akimitsu Miyauchi, Eiichi Kinoshita, Yastto Imanishi, Nobuhiko Shimizu, Yoshiki Nishizawa, Harald Jüppner, Takehisa Yamamoto
Publikováno v:
Journal of Bone and Mineral Metabolism. 23:231-237
McCune-Albright syndrome (MAS) is sometimes complicated by hypophosphatemia and abnormally low levels of 1,25(OH)(2)D in the presence of hypophosphatemia. Recently, fibroblast growth factor 23 (FGF-23) was reported as a phosphaturic and a causal fact
Autor:
Kisho Kobayashi, Shin Amemiya, Yoshiko Nakagomi, Toshihide Ishihara, Yoshie Shimura, Akira Kasuga, Mie Mochizuki, Koji Kobayashi, Shinpei Nanazawa, Shinya Tamai, Kazumichi Onigata
Publikováno v:
Diabetes Care. 26:843-847
OBJECTIVE—To clarify the role of the T-lymphocyte–associated-4 (CTLA-4) polymorphism in the susceptibility to child-onset type 1 diabetes with regard to its clinical characteristics and complications with autoimmune thyroid disease (AITD) in the
Autor:
Yoshiko Nakagomi, Keiko Arai, Shin Amemiya, Mitsue Iketani, Yoshie Shimura, Kenji Ohyama, Tamotsu Shibasaki
Publikováno v:
Human Genetics. 112:91-97
Pseudohypoaldosteronism (PHA) is characterized by urinary salt-wasting in infancy resulting from a congenital resistance to aldosterone involving the genes for the mineralocorticoid receptor (MR) and the amiloride-sensitive sodium channel (ENaC). We
Autor:
Masanori Ohta, Hiromichi Ishikawa, Noritaka Iwatani, Kazumasa Sato, Kenji Ohyama, Yoshie Shimura, Shinpei Nakazawa, Hiroyo Mabe, Yoshiko Nakagomi, Tomoaki Sano, Rika Ohno
Publikováno v:
Endocrine Journal. 46:555-562
To clarify the effect of GH on the development of seminiferous tubules in premature male rats, we investigated whether GH accelerates spermatogenesis under the condition of gonadotropin deprivation. Male Wistar rats aged three weeks were divided into
Autor:
Heigoro Shirai, Kenji Ohyama, Tadashi Inagami, Yoshiko Nakagomi, Jonathan S. Blank, Mitsuhiro Kikyo, John H. Exton, Tomoaki Sano, Shinpei Nakazawa, Y. Yamano
Publikováno v:
Journal of Biological Chemistry. 272:23631-23636
To delineate domains essential for Gq protein coupling in the C-terminal region (C-tail) of rat angiotensin II (Ang II) receptor type 1A (AT1A), we modified the putative cytosolic regions of the receptor by truncation or alanine substitution and dete
Autor:
Takao Hamakubo, Isao Morishima, Deng-Fu Guo, Kenji Ohyama, Yoshiko Nakagomi, Mitsuhiro Kikyo, Norifumi Nakamura, Yoshiaki Yamano, Tadashi Inagami, Tomoaki Sano, Yoshihisa Inoue
Publikováno v:
Journal of Biological Chemistry. 270:14024-14030
The molecular interaction involved in the ligand binding of the rat angiotensin II receptor (AT1A) was studied by site-directed mutagenesis and receptor model building. The three-dimensional structure of AT1A was constructed on the basis of a multipl
Autor:
Kenji Ohyama, Isao Morishima, Takao Hamakubo, Tomoaki Sano, Yoshiko Nakagomi, Yoshiaki Yamano, Tadashi Inagami
Publikováno v:
Regulatory Peptides. 57:141-147
Angiotensin II receptor type IA (AT1A) has a cysteine (Cys) residue in each of four extracellular domains, and these Cys residues are believed to form two disulfide bridges. However, the question as to which pairs of Cys residues form disulfide bridg
Autor:
Yoshiko Nakagomi, Kazumasa Sato, Masanori Ohta, Kenji Ohyama, Tomoaki Sano, Shinpei Nakazawa, Yoshie Shimura, Toshitsugu Yamori
Publikováno v:
Endocrine Journal. 42:817-820
Present study was planned to clarify the effects of GH and insulin-like growth factor I (IGF-I) on testosterone secretion using premature male rats. Forty rats were divided four groups. GH, IGF-I, both of them or normal saline solution as control wer
Autor:
Yoshimi Suzuki, Koji Muroya, Koji Tsubouchi, Hisashi Yokoi, Tetsuo Miyake, Nobuyuki Kikuchi, Tsutomu Ogata, Maki Fukami, Fumiko Kato, Reiko Horikawa, Yoshiko Nakagomi, Manami Iso
Publikováno v:
Endocrine journal. 58(2)
GATA3 mutations cause HDR (hypoparathyroidism, sensorineural deafness, and renal dysplasia) syndrome and, consistent with the presence of the second DiGeorge syndrome locus (DGS2) proximal to GATA3, distal 10p deletions often leads to HDR and DiGeorg