Zobrazeno 1 - 10
of 38
pro vyhledávání: '"Yong Ming Dang"'
Autor:
Bo You, Yu Long Zhang, Gao Xing Luo, Yong Ming Dang, Bei Jiang, Guang Tao Huang, Xin Zhu Liu, Zi Chen Yang, Yu Chen, Jing Chen, Zhi Qiang Yuan, Su Peng Yin, Yi Zhi Peng
Publikováno v:
Critical Care, Vol 22, Iss 1, Pp 1-12 (2018)
Abstract Background In the early stage of severe burn, patients often exhibit a high level of inflammatory mediators in blood and are likely to develop sepsis. High-volume haemofiltration (HVHF) can eliminate these inflammatory mediators. We hypothes
Externí odkaz:
https://doaj.org/article/8febc8ab49824cdaba62f87a5e9c68dc
Autor:
Su Peng Yin, Yi Zhi Peng, Yong Ming Dang, Yu Chen, Gao Xing Luo, Jing Chen, Zi Chen Yang, Xin Zhu Liu, Bei Jiang, Yulong Zhang, Guang Tao Huang, Bo You, Zhi Qiang Yuan
Publikováno v:
Critical Care, Vol 22, Iss 1, Pp 1-12 (2018)
Critical Care
Critical Care
Background In the early stage of severe burn, patients often exhibit a high level of inflammatory mediators in blood and are likely to develop sepsis. High-volume haemofiltration (HVHF) can eliminate these inflammatory mediators. We hypothesised that
Publikováno v:
Cellular Physiology and Biochemistry, Vol 49, Iss 2, Pp 717-727 (2018)
Background/Aims: The phosphatidylinositol-3-kinase -AKT (PI3K-AKT) is an important intracellular signal pathway in regulating cell proliferation, differentiation and apoptosis. In previous studies, we’ve demonstrated that PI3K–AKT pathway protect
Publikováno v:
Microscopy and Microanalysis. 21:1582-1590
Microtubule analysis is of significant value for a better understanding of normal and pathological cellular processes. Although immunofluorescence microscopic techniques have proven useful in the study of microtubules, comparative results commonly re
Publikováno v:
Journal of Trauma: Injury, Infection & Critical Care. 71:663-672
Background Ischemic/hypoxic myocardial damage and functional impairment of the myocardium occurs immediately after major burns. This experimental study investigated whether the prompt cardiac dysfunction initiates hepatic, renal, and intestinal injur
Publikováno v:
Clinical and Experimental Pharmacology and Physiology. 37:598-604
1. After a severe burn, a marked decrease in myocardial blood flow results in ischaemic and hypoxic injury, which subsequently leads to apoptosis or necrosis. Phosphatidylinositol 3-kinase (PI3-K)/Akt is an important intracellular signal transduction
Autor:
Qiong Zhang, Zhi-Gang Chu, Guang-ping Liang, Yuesheng Huang, Yong-ming Dang, Jian Han, Jiongyu Hu, Hong Yan
Publikováno v:
Cellular and Molecular Life Sciences. 67:321-333
In both cardiomyocytes and HeLa cells, hypoxia (1% O(2)) quickly leads to microtubule disruption, but little is known about how microtubule dynamics change during the early stages of hypoxia. We demonstrate that microtubule associated protein 4 (MAP4
Publikováno v:
Microscopy and microanalysis : the official journal of Microscopy Society of America, Microbeam Analysis Society, Microscopical Society of Canada. 21(2)
Microtubules of cardiac myocytes depolymerize after a hypoxic insult or treatment with colchicine. However, little attention has been paid to quantifying changes in microtubule distribution when using fluorescent images. We converted fluorescence ima
Autor:
Rong Xiao, Xiu-wu Bian, Ze-yuan Lei, Jiongyu Hu, Yuesheng Huang, Bing-qian Zhang, Guang Wang, Jiaping Zhang, Dongxia Zhang, Jing Ruan, Yong-ming Dang
Publikováno v:
Biotechnology and applied biochemistry. 59(4)
The aim of this study is to investigate the effects of enalapril, an angiotensin-converting enzyme inhibitor, on multiple organ damage after scald injury. Healthy adult rats (half male and half female; 8-12 weeks old) were randomly assigned to the fo
Autor:
Xia Dai, Miao Teng, Yuesheng Huang, Jiaping Zhang, Ya-dong Fang, Dongxia Zhang, Yong-ming Dang, Jiongyu Hu, Xue Xu, Yi-ming Zhang, Qiong Zhang
Publikováno v:
PLoS ONE, Vol 6, Iss 12, p e28052 (2011)
PLoS ONE
PLoS ONE
Mitochondrial membrane permeability has received considerable attention recently because of its key role in apoptosis and necrosis induced by physiological events such as hypoxia. The manner in which mitochondria interact with other molecules to regu