Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Yinze Han"'
Publikováno v:
MedComm – Oncology, Vol 2, Iss 4, Pp n/a-n/a (2023)
Abstract Owing to the limitations of conventional cancer therapies, including chemotherapy, radiotherapy, and surgery, gene therapy has become a prominent strategy for cancer treatment over the past few decades. Gene therapy is a medical approach for
Externí odkaz:
https://doaj.org/article/02d409e44cc546eb9d3958e2266eab78
Publikováno v:
Communications Biology, Vol 6, Iss 1, Pp 1-12 (2023)
Reporting the crystal structure of the N-terminal domain of the viral nucleocapsid protein in complex with the ubiquitin-like domain 1 of Nsp3 in SARS-CoV-2
Externí odkaz:
https://doaj.org/article/068b8bd3e28544359edba609f154cff0
Publikováno v:
Biochemistry and Biophysics Reports, Vol 32, Iss , Pp 101359- (2022)
In HNSCC, few studies have focused on the relationship between wild-type TP53 and mutant TP53-related immunity and prognosis. Our objective was to explore how TP53 mutation regulates the immunophenotype of HNSCC and thus affects the prognosis of HNSC
Externí odkaz:
https://doaj.org/article/2d7fd82c2aeb484092e298f385640170
Autor:
Yinze Han, Hailong Gao, Jinzhi Xu, Jian Luo, Bing Han, Jialing Bao, Guoqing Pan, Tian Li, Zeyang Zhou
Publikováno v:
Frontiers in Microbiology, Vol 11 (2020)
Microsporidia are obligate intracellular and eukaryotic pathogens that can infect immunocompromised and immunocompetent mammals, including humans. Both innate and adaptive immune systems play important roles against microsporidian infection. The inna
Externí odkaz:
https://doaj.org/article/db3eaaa01a804296b4369474b459e8e3
Publikováno v:
Virologica Sinica (16740769); Apr2024, Vol. 39 Issue 2, p331-334, 4p
Autor:
Heinrich Leonhardt, Rolf Hilgenfeld, Robert Buschauer, Jian Lei, Roland Beckmann, Yinze Han, Volker Thiel, Yue Ma-Lauer, Albrecht von Brunn, Matthias Thoms, Joerg Jores, Wen Deng
Publikováno v:
The EMBO Journal
The EMBO journal 40(11), e102277 (1-19) (2021). doi:10.15252/embj.2019102277
Lei, Jian; Ma-Lauer, Yue; Han, Yinze; Thoms, Matthias; Buschauer, Robert; Jores, Joerg; Thiel, Volker; Beckmann, Roland; Deng, Wen; Leonhardt, Heinrich; Hilgenfeld, Rolf; von Brunn, Albrecht (2021). The SARS-unique domain (SUD) of SARS-CoV and SARS-CoV-2 interacts with human Paip1 to enhance viral RNA translation. The EMBO journal, 40(11), e102277. EMBO Press 10.15252/embj.2019102277
The EMBO journal 40(11), e102277 (1-19) (2021). doi:10.15252/embj.2019102277
Lei, Jian; Ma-Lauer, Yue; Han, Yinze; Thoms, Matthias; Buschauer, Robert; Jores, Joerg; Thiel, Volker; Beckmann, Roland; Deng, Wen; Leonhardt, Heinrich; Hilgenfeld, Rolf; von Brunn, Albrecht (2021). The SARS-unique domain (SUD) of SARS-CoV and SARS-CoV-2 interacts with human Paip1 to enhance viral RNA translation. The EMBO journal, 40(11), e102277. EMBO Press 10.15252/embj.2019102277
The EMBO journal 40(11), e102277 (1-19) (2021). doi:10.15252/embj.2019102277
The ongoing outbreak of severe acute respiratory syndrome (SARS) coronavirus 2 (SARS-CoV-2) demonstrates the continuous threat of emerging coronaviruses (CoVs) to publi
The ongoing outbreak of severe acute respiratory syndrome (SARS) coronavirus 2 (SARS-CoV-2) demonstrates the continuous threat of emerging coronaviruses (CoVs) to publi
Autor:
Tian Li, Hailong Gao, Zeyang Zhou, Jian Luo, Jiajing Chen, Yinze Han, Chunxia Wang, Bing Han, Boyan Pei, Jin-Zhi Xu, Bin Yu
Background: Microsporidia, a group of obligate intracellular parasites that can infect humans and nearly all animals, have lost the pathways for de novo amino acid, lipid and nucleotide synthesis and instead evolved strategies to manipulate host meta
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::a103c12b7737c11ef8cc5943bf0a63d6
https://doi.org/10.21203/rs.3.rs-319485/v1
https://doi.org/10.21203/rs.3.rs-319485/v1
Autor:
Chen Xu, Jian Luo, Hailong Gao, Tian Li, Guoqing Pan, Jin-Zhi Xu, Xianzhi Meng, Qiang He, Zeyang Zhou, Yinze Han
Publikováno v:
Journal of invertebrate pathology. 186
Microsporidia are a group of obligate intracellular parasites which lack mitochondria and have highly reduced genomes. Therefore, they are unable to produce ATP via the tricarboxylic acid (TCA) cycle and oxidative phosphorylation. Instead, they have