Zobrazeno 1 - 10
of 34
pro vyhledávání: '"Yinjie Yang"'
Autor:
Tadaaki Inano, Marito Araki, Soji Morishita, Misa Imai, Yoshihiko Kihara, Maho Okuda, Yinjie Yang, Masafumi Ito, Satoshi Osaga, Hiroyuki Mano, Yoko Edahiro, Tomonori Ochiai, Kyohei Misawa, Yasutaka Fukuda, Jun Ando, Norio Komatsu
Publikováno v:
Scientific Reports, Vol 12, Iss 1, Pp 1-7 (2022)
Externí odkaz:
https://doaj.org/article/098c249009b9457ba2829d304a4b75a2
Autor:
Tadaaki Inano, Marito Araki, Soji Morishita, Misa Imai, Yoshihiko Kihara, Maho Okuda, Yinjie Yang, Masafumi Ito, Satoshi Osaga, Hiroyuki Mano, Yoko Edahiro, Tomonori Ochiai, Kyohei Misawa, Yasutaka Fukuda, Jun Ando, Norio Komatsu
Publikováno v:
Scientific Reports, Vol 11, Iss 1, Pp 1-11 (2021)
Abstract A subset of essential thrombocythemia (ET) cases are negative for disease-defining mutations on JAK2, MPL, and CALR and defined as triple negative (TN). The lack of recurrent mutations in TN-ET patients makes its pathogenesis ambiguous. Here
Externí odkaz:
https://doaj.org/article/a94cc6fdb69b4023a4cc0476b24bda89
Autor:
Qilin Cao, Yinjie Yang
Publikováno v:
2022 7th International Conference on Cloud Computing and Big Data Analytics (ICCCBDA).
Publikováno v:
SSRN Electronic Journal.
Autor:
Misa Imai, Masafumi Ito, Kyohei Misawa, Maho Okuda, Yoko Edahiro, Tomonori Ochiai, Yoshihiko Kihara, Norio Komatsu, Jun Ando, Yinjie Yang, Soji Morishita, Satoshi Osaga, Yasutaka Fukuda, Tadaaki Inano, Marito Araki, Hiroyuki Mano
Publikováno v:
Scientific Reports
Scientific Reports, Vol 11, Iss 1, Pp 1-11 (2021)
Scientific Reports, Vol 11, Iss 1, Pp 1-11 (2021)
A subset of essential thrombocythemia (ET) cases are negative for disease-defining mutations on JAK2, MPL, and CALR and defined as triple negative (TN). The lack of recurrent mutations in TN-ET patients makes its pathogenesis ambiguous. Here, we scre
Autor:
Federico, De Marchi, Maho, Okuda, Soji, Morishita, Misa, Imai, Terumi, Baba, Mei, Horino, Yosuke, Mori, Chiho, Furuya, Satoko, Ogata, Yinjie, Yang, Jun, Ando, Miki, Ando, Marito, Araki, Norio, Komatsu
Publikováno v:
Leukemia Research. 119:106883
Cyclic AMP-response element-binding protein 3-like 1 (CREB3L1) is a gene involved in the unfolded protein response (UPR). Recently, we demonstrated that CREB3L1 is specifically overexpressed in the platelets of patients with Philadelphia chromosome-n
Publikováno v:
The Science of the total environment. 780
The Yangtze River, the third largest river and supporting nearly one-third of Chinese population, has been severely polluted in recent decades. Among the numerous pollutants, organic micropollutants, as one kind of important emerging contaminants, ar
Autor:
Yosuke Mori, Misa Imai, Satoko Ogata, Mei Horino, Yo Mabuchi, Teppei Taguchi, Norio Komatsu, Soji Morishita, Yoshihiko Kihara, Syunpei Yoshikawa, Takehiro Suzuki, Nami Masubuchi, Yinjie Yang, Marito Araki, Chihiro Akazawa, Naoshi Domae, Yasutaka Fukuda, Akimichi Ohsaka, Motoyuki Shimonaka
Publikováno v:
Blood. 136:9-10
Mutant calreticulin (CALR) has been shown to play a causal role in the development of essential thrombocythemia (ET) and primary myelofibrosis (PMF) via activation of the thrombopoietin receptor MPL. The oncogenic property of mutant CALR originates f
Autor:
Misa Imai, Yinjie Yang, Masafumi Ito, Yoko Edahiro, Tomonori Ochiai, Tadaaki Inano, Satoshi Osaga, Norio Komatsu, Yasutaka Fukuda, Marito Araki, Soji Morishita, Akimichi Ohsaka, Kyohei Misawa, Yoshihiko Kihara, Maho Okuda
Publikováno v:
Blood. 136:27-28
Somatic mutations in JAK2, MPL, and CALR are found in approximately 80% of patients with essential thrombocythemia (ET), whereas the remaining patients are negative for disease-defining mutations and are defined as triple-negative (TN). Studies have
Autor:
Nami Masubuchi, Misa Imai, Marito Araki, Mai Nudejima, Yumi Hironaka, Yinjie Yang, Hiraku Takei, Akimichi Ohsaka, Yoko Edahiro, Yoshihiko Kihara, Norio Komatsu, Yoshihisa Mizukami, Erina Hayashi, Masato Koike
Publikováno v:
Leukemia. 34(2)
Studies have shown that mutant calreticulin (CALR) constitutively activates the thrombopoietin (TPO) receptor MPL and thus plays a causal role in the development of myeloproliferative neoplasms (MPNs). To further elucidate the molecular mechanism by