Zobrazeno 1 - 10
of 33
pro vyhledávání: '"Yina Dong"'
Publikováno v:
Zhongguo quanke yixue, Vol 25, Iss 08, Pp 952-956 (2022)
BackgroundKidney is a major extramedullary organ involved in leukemia, but clinicians have insufficient understanding of it due to rare case reports.ObjectiveTo analyze the clinicopathological manifestations of kidney injury in leukemia.MethodsFive p
Externí odkaz:
https://doaj.org/article/a5c631265512471bb56a7973f9ecbcc9
Publikováno v:
PLoS ONE, Vol 10, Iss 11, p e0142340 (2015)
The perforant pathway projection from layer II of the entorhinal cortex to the hippocampal dentate gyrus is especially important for long-term memory formation, and is preferentially vulnerable to developing a degenerative tauopathy early in Alzheime
Externí odkaz:
https://doaj.org/article/75845554aaa84c40862df3dc54c1a210
Autor:
Jennifer Farmer, Robert B. Wilson, Kimberly Schadt, Yina Dong, Sarah Gelbard, Debbie L. Foerster, M. G. Cotticelli, Alicia Brocht, Lauren Seyer, Nathaniel R. Greeley, David A. Lynch, Cassandra J Strawser
Publikováno v:
Acta Neurologica Scandinavica. 132:7-15
Objectives/Aims This is an open-label trial of the safety of interferon gamma-1b (IFN-γ) and its effect on frataxin levels and neurologic measures in 12 children with Friedreich ataxia. Materials and Methods Interferon gamma-1b was administered via
Publikováno v:
Journal of Neuropathology and Experimental Neurology
Supplemental Digital Content is available in the text.
The perforant pathway projection from the entorhinal cortex (EC) to the hippocampal dentate gyrus is critically important for long-term memory and develops tau and amyloid pathologies and pr
The perforant pathway projection from the entorhinal cortex (EC) to the hippocampal dentate gyrus is critically important for long-term memory and develops tau and amyloid pathologies and pr
Autor:
David A. Lynch, Yina Dong, Grace Yoon, Theresa A. Zesiewicz, Christoper M. Gomez, S. H. Subramony, Karlla W. Brigatti, Nathaniel R. Greeley, Jennifer Farmer, Robert B. Wilson, Chad Hoyle, Susan Perlman, Eric C. Deutsch, Michael Lazaropoulos, George Wilmot, Lauren Seyer, Elisia M. Clark, Katherine D. Mathews, Alicia Brocht, Carlton W. Christie
Publikováno v:
Annals of Clinical and Translational Neurology
Objective Friedreich ataxia (FRDA) is an autosomal recessive ataxia resulting from mutations in the frataxin gene (FXN). Such mutations, usually expanded guanine–adenine–adenine (GAA) repeats, give rise to decreased levels of frataxin protein in
Publikováno v:
PLoS ONE, Vol 10, Iss 11, p e0142340 (2015)
PLoS ONE
PLoS ONE
The perforant pathway projection from layer II of the entorhinal cortex to the hippocampal dentate gyrus is especially important for long-term memory formation, and is preferentially vulnerable to developing a degenerative tauopathy early in Alzheime
Publikováno v:
Journal of Pharmacology and Experimental Therapeutics. 302:1023-1030
N-Methyl-D-aspartate (NMDA) receptors are calcium-permeable glutamate receptors that play putative roles in learning, memory, and excitotoxicity. NMDA receptor-mediated calcium entry can activate the calcium-dependent protease calpain, leading to sub
Publikováno v:
Molecular Brain Research. 77:29-36
The hypothalamic suprachiasmatic nucleus (SCN) functions as a circadian pacemaker regulating a variety of physiological and behavioral rhythms in mammals. Retinal illumination evokes expression of several immediate-early genes, including junB, in the
Publikováno v:
Biology of the NMDA Receptor ISBN: 9781420044140
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f551dc72c561f4964342a733a0f3026b
https://doi.org/10.1201/9781420044157.ch7
https://doi.org/10.1201/9781420044157.ch7
Autor:
Robert W. Neumar, Rodney P. Guttmann, Kelly L. Simpkins, Yina Dong, Set Sokol, Zhaoming Chen, David A. Lynch
Publikováno v:
The Journal of neuroscience : the official journal of the Society for Neuroscience. 23(36)
Although activation of calcium-activated neutral protease (calpain) by the NMDA receptor has been suggested to play critical roles in synaptic modulation and neurologic disease, the nature of its substrates has not been completely defined. In this st