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Inhaled Nitric Oxide Modifies Left Ventricular Diastolic Stress in the Presence of Vasoactive Agents in Heart Failure

Autor: Naoyuki Hasebe, Hironobu Matsuhashi, Shunsuke Natori, Kenjiro Kikuchi, Tadashi Ihara, Yin-Tie Jin, Akira Ido, Tomoyuki Matsusaka

Publikováno v: American Journal of Respiratory and Critical Care Medicine. 167:895-901

Nitric oxide (NO) inhalation therapy has been widely used in several diseases with pulmonary hypertension. However, application of NO inhalation therapy remains controversial in heart failure. Cardiovascular effects of inhaled NO (iNO) were evaluated

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0e159d8fec051620b79991c7686a1025
https://doi.org/10.1164/rccm.200201-057oc

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Effects of Angiotensin Converting Enzyme Inhibitor and Angiotensin II Type 1 Receptor Blocker on Cardiac Dysfunction Induced by Isoproterenol in Dogs

Autor: Yin-Tie Jin, Shinsuke Kido, Kenjiro Kikuchi, Takafumi Ohta, Naoyuki Hasebe, Motohiko Sato, Shiro Tsuji, Shunsuke Natori

Publikováno v: Journal of Cardiovascular Pharmacology. 38:S63-S67

We examined whether angiotensin converting enzyme (ACE) inhibitors and angiotensin II type 1 receptor blockers (ARB) prevent isoproterenol (ISO)-induced left ventricular (LV) dysfunction in dogs. The effects of a large dose of ISO, 1 microg/kg/min, 3

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::00949c08aa72838f397678d5b5f0a16f
https://doi.org/10.1097/00005344-200110001-00014

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Effect of Inhaled Nitric Oxide on Cardiovascular Response to Catecholamine in Heart Failure

Autor: Masahiko Morihira, Tomoyuki Matsusaka, Hideki Nakamura, Kenjiro Kikuchi, Shunsuke Natori, Takafumi Ohta, Hironobu Matsuhashi, Akira Ido, Yin-Tie Jin, Naoyuki Hasebe, Shiro Tsuji

Publikováno v: Journal of Cardiovascular Pharmacology. 36:S55-S60

We examined the dose responses to continuous infusion of isoproterenol (ISO) and norepinephrine (NE) in normal (control) and procainamide-induced heart failure dogs with or without inhalation of 70 ppm nitric oxide (NO). Inhaled NO affected neither l

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d67e88aaf0c2650cc05d6d3fc4840b93
https://doi.org/10.1097/00005344-200000006-00012

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Magnesium attenuates isoproterenol-induced acute cardiac dysfunction and beta-adrenergic desensitization

Autor: Kenjiro Kikuchi, Tomoyuki Matsusaka, Takafumi Ohta, Naoyuki Hasebe, Shiro Tsuji, Yin-Tie Jin, Shunsuke Natori

Publikováno v: American journal of physiology. Heart and circulatory physiology. 292(3)

Sympathetic nervous activation is a crucial compensatory mechanism in heart failure. However, excess catecholamine may induce cardiac dysfunction and β-adrenergic desensitization. Although magnesium is known to be a cardioprotective agent, its benef

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5b191771a6f931601d711a2a45d30813
https://pubmed.ncbi.nlm.nih.gov/17114241

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Unequal effects of renin-angiotensin system inhibitors in acute cardiac dysfunction induced by isoproterenol

Autor: Takafumi Ohta, Shiro Tsuji, Yin-Tie Jin, Kazuma Izawa, Syunsuke Natori, Shinsuke Kido, Naoyuki Hasebe, Motohiko Sato, Kenjiro Kikuchi

Publikováno v: American journal of physiology. Heart and circulatory physiology. 287(6)

Several clinical trials have demonstrated that angiotensin-converting enzyme inhibitor (ACEI) and angiotensin II type 1 receptor blocker (ARB) are equally effective in the treatment of chronic heart failure. However, this has not been confirmed for a

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cb63d262889960fb93e47791934015e7
https://pubmed.ncbi.nlm.nih.gov/15297251

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Magnesium reduces myocardial infarct size via enhancement of adenosine mechanism in rabbits

Autor: Yin Tie Jin, Naoyuki Hasebe, Jun-ichi Kawabe, Kenjiro Kikuchi, Tomoyuki Matsusaka

Publikováno v: Cardiovascular research. 54(3)

Objectives: Clinical impact of magnesium (Mg) therapy remains controversial in acute myocardial infarction. We investigated the infarct size limiting effects of Mg and its mechanism in rabbits. Methods: Anesthetized rabbits underwent 30 min coronary

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3ecfe976f6efd4da7cb4162d9d6be220
https://pubmed.ncbi.nlm.nih.gov/12031702

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