Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Yilei Jing"'
Autor:
Yixiao Pan, Shiming Gong, Xinye Zhu, Chuqing Xue, Yilei Jing, Yinghua Sun, Yongbing Qian, Jianjun Zhang, Qiang Xia
Publikováno v:
Frontiers in Public Health, Vol 10 (2022)
ObjectivesThe hospitalization and mortality rate from COVID-19 appears to be higher in liver transplant recipients when compared with general populations. Vaccination is an effective strategy to reduce the risk during the COVID-19 pandemic. We aimed
Externí odkaz:
https://doaj.org/article/498db8f000614041bd33fdb327e26d6d
Autor:
Mengmeng Dou, Xueliang Zhou, Lifeng Li, Mingliang Zhang, Wenbin Wang, Mengru Wang, Yilei Jing, Rui Ma, Jie Zhao, Lin Zhu
Publikováno v:
Frontiers in Immunology, Vol 12 (2021)
The etiology of multiple sclerosis (MS) is not clear, and the treatment of MS presents a great challenge. This study aimed to investigate the pathogenesis and potential therapeutic targets of MS and to define target genes of matrine, a quinolizidine
Externí odkaz:
https://doaj.org/article/94e4a43c7b2942f1ab9c3b93a3e14aa7
Autor:
Rui Ma, Yaojuan Chu, Mengmeng Dou, Yilei Jing, Xiaoyu Zhao, Xinyu Li, Silu Li, Yuan Zhang, Limei Wang, Lin Zhu
Publikováno v:
Journal of neuroimmunology. 367
Oligodendrocyte (OL) death and remyelination failure lead to progressive neurological deficits in multiple sclerosis (MS) and its animal model, experimental autoimmune encephalomyelitis (EAE). Matrine (MAT), a quinolizidine alkaloid component derived
Autor:
Ying Lv, Xiaoyu Zhao, Mingliang Zhang, Yaojuan Chu, Lin Zhu, Wendi Ma, Mengru Wang, Yilei Jing, Rui Ma
Publikováno v:
Journal of neuroimmunology. 352
The inflammatory mediator high-mobility group box 1 (HMGB1)-induced signaling pathway has been shown to play an important role in the pathogenesis of multiple sclerosis (MS) and its animal model, experimental autoimmune encephalomyelitis (EAE). Matri
Publikováno v:
Journal of Chemical Neuroanatomy. 117:102004
Type 1 astrocytes (A1), which are highly proinflammatory and neurotoxic, are prevalent in multiple sclerosis (MS). In addition, in MS and its animal model, experimental autoimmune encephalomyelitis (EAE), immune cells must cross the blood-brain barri