Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Yi‐Ing Chen"'
Autor:
Yu‐Ting Chang, Hsuan‐Yu Peng, Chun‐Mei Hu, Sui‐Chih Tien, Yi‐Ing Chen, Yung‐Ming Jeng, Ming‐Chu Chang
Publikováno v:
Molecular Oncology, Vol 17, Iss 8, Pp 1628-1647 (2023)
Cancer‐associated fibroblasts (CAFs), a major component of the tumor microenvironment (TME) in pancreatic ductal adenocarcinoma (PDAC), play an important role in tumorigenesis, metastasis, and chemoresistance. Tumor‐derived small extracellular ve
Externí odkaz:
https://doaj.org/article/da4eb509dd15471e9d95bcf9706e728d
Oncogenic KRAS, Mucin 4, and Activin A‐Mediated Fibroblast Activation Cooperate for PanIN Initiation
Autor:
Chun‐Mei Hu, Chien‐Chang Huang, Min‐Fen Hsu, Hung‐Jen Chien, Pei‐Jung Wu, Yi‐Ing Chen, Yung‐Ming Jeng, Shiue‐Cheng Tang, Mei‐Hsin Chung, Chia‐Ning Shen, Ming‐Chu Chang, Yu‐Ting Chang, Yu‐Wen Tien, Wen‐Hwa Lee
Publikováno v:
Advanced Science, Vol 10, Iss 36, Pp n/a-n/a (2023)
Abstract Over 90% of patients with pancreatic ductal adenocarcinoma (PDAC) have oncogenic KRAS mutations. Nevertheless, mutated KRAS alone is insufficient to initiate pancreatic intraepithelial neoplasia (PanIN), the precursor of PDAC. The identities
Externí odkaz:
https://doaj.org/article/4a0e87c67012457eb7ccce5aa913ffd6
Autor:
Yi-Ing Chen, Chin-Chun Chang, Min-Fen Hsu, Yung-Ming Jeng, Yu-Wen Tien, Ming-Chu Chang, Yu-Ting Chang, Chun-Mei Hu, Wen-Hwa Lee
Publikováno v:
Nature Communications, Vol 13, Iss 1, Pp 1-20 (2022)
Direct contact between tumour cells and fibroblasts influences tumour cell behaviour. Here the authors show that pancreatic cancer cells and fibroblasts directly interact via homophilic ATP1A1 binding, which induces fibroblasts to secrete activin A t
Externí odkaz:
https://doaj.org/article/0ef09420616e4351be36b283dc768cbd
Autor:
Yi-Ing Chen, 陳以瑛
100
Glaucoma, a group of diseases characterized by progressive optic nerve degeneration, is the leading cause of irreversible blindness worldwide. The initiation of the disease and its progression involve an ischaemic-like insult to the ganglion
Glaucoma, a group of diseases characterized by progressive optic nerve degeneration, is the leading cause of irreversible blindness worldwide. The initiation of the disease and its progression involve an ischaemic-like insult to the ganglion
Externí odkaz:
http://ndltd.ncl.edu.tw/handle/75009875709414131619
Autor:
Yi-Ing Chen, Chin-Chun Chang, Min-Fen Hsu, Yung-Ming Jeng, Yu-Wen Tien, Ming-Chu Chang, Yu-Ting Chang, Chun-Mei Hu, Wen-Hwa Lee
Publikováno v:
Nature communications. 13(1)
Tumor cells with diverse phenotypes and biological behaviors are influenced by stromal cells through secretory factors or direct cell-cell contact. Pancreatic ductal adenocarcinoma (PDAC) is characterized by extensive desmoplasia with fibroblasts as
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f834527a704accdd47db84bb3754d9ef
https://pubmed.ncbi.nlm.nih.gov/35618735
https://pubmed.ncbi.nlm.nih.gov/35618735
Autor:
Wen-Hwa Lee, Mei-Yu Chen, Lung-Hung Tsai, Yu-Wen Tien, Ming-Chu Chang, Chun-Mei Hu, Chia-Ning Shen, Yi-Ing Chen, Eva Y.-H. P. Lee, Heng-Hsiung Wu, Chun-Kai Huang, Yung-Ming Jeng, Yu-Ting Chang, Chen-Chen Lee, Pang-Hung Hsu
Publikováno v:
Science translational medicine. 13(583)
The members of the interleukin-17 (IL-17) cytokine family and their receptors were identified decades ago. Unlike IL-17 receptor A (IL-17RA), which heterodimerizes with IL-17RB, IL-17RC, and IL-17RD and mediates proinflammatory gene expression, IL-17
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4b6febc3a4e88757642bbab483c100d9
https://pubmed.ncbi.nlm.nih.gov/36475907
https://pubmed.ncbi.nlm.nih.gov/36475907
Publikováno v:
American journal of translational research. 8(4)
NPGPx (GPx7) is a member of the glutathione peroxidase (GPx) family without any GPx activity. GPx7 displays a unique function which serves as a stress sensor/transmitter to transfer the signal to its interacting proteins by shuttling disulfide bonds
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::fefcb33cc1f2956e542767b4df66eac8
https://pubmed.ncbi.nlm.nih.gov/27186289
https://pubmed.ncbi.nlm.nih.gov/27186289