Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Yayoi Shikata"'
Autor:
Toshihiko Shiroishi, Jun Motoyama, Tammy Ellis, Masaharu Ogawa, Mitsuhiro Hashimoto, Daisuke Matsumaru, Brandon J. Wainwright, Yayoi Shikata, Toshiaki Okada
Publikováno v:
Developmental Biology. 349:147-159
Sonic hedgehog (Shh) signaling regulates cell differentiation and proliferation during brain development. However, the role of Shh in neurogenesis during late gestation (embryonic day 13.5–18.5) remains unclear. Herein, we used a genetic approach a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1c521ee642c1a42449e84d53e3a9456e
https://doi.org/10.1016/j.ydbio.2010.10.014
https://doi.org/10.1016/j.ydbio.2010.10.014
Publikováno v:
Birth Defects Research Part A: Clinical and Molecular Teratology. 82:224-231
BACKGROUND: In humans, fetal ethanol exposure can cause holoprosencephaly (HPE), one of the most common birth defects that is characterized by brain, facial, and oral abnormalities. However, the pathogenesis of HPE is not clear. In the present study,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::21bb12b2da55ac95929fa9843a19258d
https://doi.org/10.1002/bdra.20447
https://doi.org/10.1002/bdra.20447
Autor:
Seiji Shioda, Jun Motoyama, Yayoi Shikata, Kazushi Aoto, Hajime Imai, Tomoyuki Tokunaga, Gen Yamada, Daisuke Matsumaru
Publikováno v:
Developmental biology. 327(1)
In humans, holoprosencephaly (HPE) is a common birth defect characterized by the absence of midline cells from brain, facial, and oral structures. To understand the pathoetiology of HPE, we investigated the involvement of mammalian prechordal plate (
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::71c47b5bbc5f00e8ddfedcb0fcd9072b
https://pubmed.ncbi.nlm.nih.gov/19103193
https://pubmed.ncbi.nlm.nih.gov/19103193
Autor:
Yayoi Shikata, Ljiljana Milenkovic, Matthew P. Scott, Chi-chung Hui, Jun Motoyama, Mizuho Iwama
Publikováno v:
Developmental Biology. (1):150-161
Sonic hedgehog (Shh) directs the development of ventral cell fates, including floor plate and V3 interneurons, in the mouse neural tube. Here, we show that the transcription factors Gli2 and Gli3, mediators of Shh signaling, are required for the deve
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bde056e7bd5253fb2826f8ea2030a922