Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Yasuki Motomiya"'
Publikováno v:
Diabetology & Metabolic Syndrome, Vol 13, Iss 1, Pp 1-8 (2021)
Abstract Background 1,5-anhydroglucitol is a reduction product of 1,5-anhydrofructose. Circulating 1,5-anhydroglucitol is usually excreted by the kidneys and is reabsorbed via sodium-glucose co-transporter 4 in the renal tubules. In patients on hemod
Externí odkaz:
https://doaj.org/article/d639423bef2f4bb98b98518c15c17a7f
Autor:
Takehisa Nojima, Yasuki Motomiya
Publikováno v:
Kidney and Dialysis. 1:36-46
High flow access (HFA) is a condition in which hemodynamics is affected by a flow rate that is larger than the blood flow required for hemodialysis. HFA sometimes causes high output heart failure, venous hypertension, and dialysis access steal syndro
Autor:
Yasuki Motomiya, Takehisa Nojima
Publikováno v:
Nihon Toseki Igakkai Zasshi. 54:1-8
Publikováno v:
Diabetology & Metabolic Syndrome
Diabetology & Metabolic Syndrome, Vol 13, Iss 1, Pp 1-8 (2021)
Diabetology & Metabolic Syndrome, Vol 13, Iss 1, Pp 1-8 (2021)
Background 1,5-anhydroglucitol is a reduction product of 1,5-anhydrofructose. Circulating 1,5-anhydroglucitol is usually excreted by the kidneys and is reabsorbed via sodium-glucose co-transporter 4 in the renal tubules. In patients on hemodialysis,
Autor:
Takehisa Nojima, Yasuki Motomiya
Publikováno v:
Annals of Vascular Diseases
Objective: Flow reduction is required to preserve vascular access in cases with high flow access (HFA). We report a new flow reduction procedure, the graft inclusion technique (GIT). Methods: The GIT procedure developed by us involves the intralumina
Autor:
Tancharoen Salunya, Yoshihiro Motomiya, Yasuki Motomiya, Ikuro Maruyama, Yoko Oyama, Munekazu Yamakuchi, Tanaka Kenji
Publikováno v:
Therapeutic Apheresis and Dialysis. 21:395-404
Chronic kidney disease (CKD) has been known to be a state of excessive fibroblast growth factor-23 (FGF23) and α-Klotho deficiency. Patients undergoing hemodialysis have an increased mortality risk associated with cardiovascular disease and endothel
Autor:
Kenji, Tanaka, Tancharoen, Salunya, Yoshihiro, Motomiya, Yasuki, Motomiya, Yoko, Oyama, Munekazu, Yamakuchi, Ikuro, Maruyama
Publikováno v:
Therapeutic apheresis and dialysis : official peer-reviewed journal of the International Society for Apheresis, the Japanese Society for Apheresis, the Japanese Society for Dialysis Therapy. 21(4)
Chronic kidney disease (CKD) has been known to be a state of excessive fibroblast growth factor-23 (FGF23) and α-Klotho deficiency. Patients undergoing hemodialysis have an increased mortality risk associated with cardiovascular disease and endothel
Autor:
Mari Ishida, Izumi Amano, Fumitake Gejyo, Shigeko Hara, Tomoyuki Yamakawa, Kenji Tsuchida, Hiroyuki Morita, Motoyoshi Sato, Satoshi Kurihara, Yoshio Taguma, Makoto Hiramatsu, Ryoichi Nakazawa, Masashi Suzuki, Kunihiro Yamagata, Seiichi Obayashi, Kazutaka Kukita, Kunihiko Yoshiya, Jeongsoo Shin, Yutaka Kanno, Yasuki Motomiya, Susumu Takahashi, Ryuichi Furuya, Hiroshi Ogawa, Tateki Kitaoka, Hidetoshi Nakamura, Takeshi Nakanishi, Yoshiaki Takemoto, Ikuto Masakane, Tetsuo Ando, Toshihiko Ono
Publikováno v:
Therapeutic Apheresis and Dialysis. 17:40-47
Dialysis-related amyloidosis is a serious complication of long-term hemodialysis. Its pathogenic mechanism involves accumulation of β2-microglobulin in the blood, which then forms amyloid fibrils and is deposited in tissues, leading to inflammation
Autor:
Fumitake, Gejyo, Izumi, Amano, Tetsuo, Ando, Mari, Ishida, Seiichi, Obayashi, Hiroshi, Ogawa, Toshihiko, Ono, Yutaka, Kanno, Tateki, Kitaoka, Kazutaka, Kukita, Satoshi, Kurihara, Motoyoshi, Sato, Jeongsoo, Shin, Masashi, Suzuki, Susumu, Takahashi, Yoshio, Taguma, Yoshiaki, Takemoto, Ryoichi, Nakazawa, Takeshi, Nakanishi, Hidetoshi, Nakamura, Shigeko, Hara, Makoto, Hiramatsu, Ryuichi, Furuya, Ikuto, Masakane, Kenji, Tsuchida, Yasuki, Motomiya, Hiroyuki, Morita, Kunihiro, Yamagata, Kunihiko, Yoshiya, Tomoyuki, Yamakawa
Publikováno v:
Therapeutic apheresis and dialysis : official peer-reviewed journal of the International Society for Apheresis, the Japanese Society for Apheresis, the Japanese Society for Dialysis Therapy. 17(1)
Dialysis-related amyloidosis is a serious complication of long-term hemodialysis. Its pathogenic mechanism involves accumulation of β2-microglobulin in the blood, which then forms amyloid fibrils and is deposited in tissues, leading to inflammation