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pro vyhledávání: '"Yassine Bentefour"'
Autor:
Yassine Bentefour, Julie Bakker
Publikováno v:
Nature Communications, Vol 15, Iss 1, Pp 1-19 (2024)
Abstract Puberty is a crucial phase for the development of female sexual behavior. Growing evidence suggests that stress during this period may interfere with the development of sexual behavior. However, the neural circuits involved in this alteratio
Externí odkaz:
https://doaj.org/article/4c134bd7ddb74cd0a6d4b2b63b7e51e3
Publikováno v:
Neuroscience
Neuroscience, 2019, 418, pp.166-176. ⟨10.1016/j.neuroscience.2019.08.018⟩
Neuroscience, 2019, 418, pp.166-176. ⟨10.1016/j.neuroscience.2019.08.018⟩
International audience; Post-traumatic stress disorder (PTSD) is triggered by exposure to traumatic events, but not everyone who experiences trauma develops this disorder. Like humans, PTSD-like symptoms develop in some laboratory rodents (susceptibl
Autor:
Julie Bakker, Yassine Bentefour
Publikováno v:
Neuropharmacology. 198
It was recently shown that kisspeptin neurons in the anteroventral periventricular area (AVPV) orchestrate female sexual behavior, including lordosis behavior and mate preference. A potential target of AVPV kisspeptin signaling could be neurons expre
Publikováno v:
Brain Stimulation, Vol 11, Iss 4, Pp 913-920 (2018)
Background We have previously demonstrated, in mice, that antidepressant treatment can prevent relapse of PTSD-like behaviors (avoidance, hyperarousal, and anxiety) through increased activation in the infralimbic cortex (IL) of the medial prefrontal
Publikováno v:
Psychopharmacology. 232:2303-2312
After exposure to a severe traumatic event, avoidance, fear sensitization, and increased anxiety are among features that can persist over time in people developing posttraumatic stress disorder (PTSD). Basic research on treatment interfering with the
Publikováno v:
European neuropsychopharmacology : the journal of the European College of Neuropsychopharmacology. 26(2)
Clinical studies have shown that post-traumatic stress disorder (PTSD) remission, induced by selective serotonin reuptake inhibitor (SSRI) treatment, is associated with increased prefrontal activation during post-treatment symptom provocation. Other