Zobrazeno 1 - 10
of 15
pro vyhledávání: '"Yaser Cheema"'
Publikováno v:
Revista Portuguesa de Cardiologia, Vol 36, Iss 9, Pp 671.e1-671.e4 (2017)
The best outcome for coronary intervention in coronary artery bypass graft patients requires knowledge of prior coronary anatomy. This information is not always available as many cases present acutely, especially in ST-elevation myocardial infarction
Externí odkaz:
https://doaj.org/article/9ccf432f91264b4096814e695b17901b
Publikováno v:
Current Problems in Cardiology. 41:195-203
The adoption of radial catheterization has been relatively slow in the United States. This study was conducted to assess the perceived comfort level of cardiology fellows with radial catheterizations and to predict the practice patterns in the United
Autor:
Arie Szatkowski, Diana Barker, Amit Malhotra, Gilbert J. Zoghbi, Yaser Cheema, Brian Dragutsky, Dharmesh Patel, Darrell Sneed, Justin May, Huibo Shao
Publikováno v:
Journal of the American College of Cardiology. 73:141
Publikováno v:
Revista Portuguesa de Cardiologia, Vol 36, Iss 9, Pp 671.e1-671.e4 (2017)
Revista Portuguesa de Cardiologia (English Edition), Vol 36, Iss 9, Pp 671.e1-671.e4 (2017)
Revista Portuguesa de Cardiologia (English Edition), Vol 36, Iss 9, Pp 671.e1-671.e4 (2017)
The best outcome for coronary intervention in coronary artery bypass graft patients requires knowledge of prior coronary anatomy. This information is not always available as many cases present acutely, especially in ST-elevation myocardial infarction
Autor:
M. Usman Khan, Ivan C. Gerling, Kelly D. Green, Tieqiang Zhao, Karl T. Weber, Robert A. Ahokas, Syamal K. Bhattacharya, Wenyuan Zhao, Yaser Cheema
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 303:H486-H495
The congestive heart failure (CHF) syndrome with soft tissue wasting, or cachexia, has its pathophysiologic origins rooted in neurohormonal activation. Mechanical cardiocirculatory assistance reveals the potential for reverse remodeling and recovery
Publikováno v:
Progress in Cardiovascular Diseases. 55:77-86
A synchronized dyshomeostasis of extra- and intracellular Ca(2+), expressed as plasma ionized hypocalcemia and excessive intracellular Ca(2+) accumulation, respectively, represents a common pathophysiologic scenario that accompanies several diverse d
Autor:
Yaser Cheema, Karl T. Weber, Robert A. Ahokas, Yao Sun, Ivan C. Gerling, Syamal K. Bhattacharya, Jonathan N Sherrod, Tieqiang Zhao, Wenyuan Zhao
Publikováno v:
Journal of Cardiovascular Pharmacology. 58:80-86
Foci of fibrosis, footprints of cardiomyocyte necrosis, are scattered throughout the failing myocardium and are a major component to its pathologic remodeling. Understanding pathogenic mechanisms contributing to hormone-mediated necrosis is therefore
Autor:
Peter VanBuren, Stephen P. Bell, Zengyi Chen, Ting Yi, Yaser Cheema, Martin M. LeWinter, Sarah M. Tremble, Bradley M. Palmer, Meenakumari Subramanian
Publikováno v:
Cardiovascular Diabetology, Vol 11, Iss 1, p 135 (2012)
Cardiovascular Diabetology
Cardiovascular Diabetology
It has been reported previously that diabetic cardiomyopathy can be inhibited or reverted with chronic zinc supplementation. In the current study, we hypothesized that total cardiac calcium and zinc content is altered in early onset diabetes mellitus
Autor:
Robert A. Ahokas, Karl T. Weber, Ivan C. Gerling, Yaser Cheema, M. Usman Khan, Syamal K. Bhattacharya, Yao Sun, Atta U. Shahbaz
The survival of cardiomyocytes must be ensured as the myocardium adjusts to a myriad of competing physiological and pathophysiological demands. A significant loss of these contractile cells, together with their replacement by stiff fibrillar collagen
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6a424374afbddd0a3c6c471aec185260
https://europepmc.org/articles/PMC3386379/
https://europepmc.org/articles/PMC3386379/
Publikováno v:
European heart journal. 32(15)
An ongoing loss of cardiomyocytes to apoptotic and necrotic cell death pathways contributes to the progressive nature of heart failure. The pathophysiological origins of necrotic cell loss relate to the neurohormonal activation that accompanies acute