Zobrazeno 1 - 10
of 161
pro vyhledávání: '"Yao-Liang Tang"'
Autor:
Junlan Zhou, Min Cheng, Yu-Hua Liao, Yu Hu, Min Wu, Qing Wang, Bo Qin, Hong Wang, Yan Zhu, Xiu-Mei Gao, David Goukassian, Ting C Zhao, Yao-Liang Tang, Raj Kishore, Gangjian Qin
Publikováno v:
PLoS ONE, Vol 8, Iss 5, p e63126 (2013)
Circulating endothelial progenitor cells (circEPCs) of bone marrow (BM) origin contribute to postnatal neovascularization and represent a potential therapeutic target for ischemic disease. Statins are beneficial for ischemia disease and have been imp
Externí odkaz:
https://doaj.org/article/89f4a63573804b55b61154f8d08f687e
Autor:
Ahmed S. Bayoumi, Amer Sayed, Zuzana Broskova, Jian-Peng Teoh, James Wilson, Huabo Su, Yao-Liang Tang, Il-man Kim
Publikováno v:
International Journal of Molecular Sciences, Vol 17, Iss 3, p 356 (2016)
Protein-coding genes account for only a small part of the human genome; in fact, the vast majority of transcripts are comprised of non-coding RNAs (ncRNAs) including long ncRNAs (lncRNAs) and small ncRNAs, microRNAs (miRs). Accumulating evidence indi
Externí odkaz:
https://doaj.org/article/dca63505dc504bc0941b02912c02534f
Autor:
Emily Parker, Bharati Mendhe, Ling Ruan, Brendan Marshall, Wenbo Zhi, Yutao Liu, Sadanand Fulzele, Yao Liang Tang, Meghan McGee-Lawrence, Tae Jin Lee, Ashok Sharma, Maribeth Johnson, Jie Chen, Mark W. Hamrick
Publikováno v:
Physiological Genomics. 54:296-304
Fibro-adipogenic progenitor cells (FAPs) are a population of stem cells in skeletal muscle that play multiple roles in muscle repair and regeneration through their complex secretome; however, it is not well understood how the FAP secretome is altered
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::44aa8695c09972b800aab65ad6ceb9d1
https://doi.org/10.1152/physiolgenomics.00177.2021
https://doi.org/10.1152/physiolgenomics.00177.2021
Publikováno v:
Computational and Structural Biotechnology Journal, Vol 18, Iss, Pp 765-773 (2020)
Duchenne muscular dystrophy (DMD) is a progressive myopathic disease caused by mutations in the gene encoding dystrophin protein that eventually leads to the exhaustion of myogenic progenitor cells (MPC). Autologous induced pluripotent stem cells (iP
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7e6973b1b447dc26c2ff0238e14a55cc
http://www.sciencedirect.com/science/article/pii/S2001037019304143
http://www.sciencedirect.com/science/article/pii/S2001037019304143
Publikováno v:
Frontiers in Pharmacology, Vol 12 (2021)
Frontiers in Pharmacology
Frontiers in Pharmacology
Elderly patients are more susceptible to ischemic injury. N6-methyladenosine (m6A) modification is the most abundant reversible epitranscriptomic modification in mammalian RNA and plays a vital role in many biological processes. However, it is unclea
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2d5ecc59981ce9ed797098d4427f3fa7
https://doi.org/10.3389/fphar.2021.654316
https://doi.org/10.3389/fphar.2021.654316
Autor:
Zixin Chen, Il-man Kim, Yue Jin, Yan Shen, Tong Luo, Yao Liang Tang, Neal L. Weintraub, Xuan Su
Publikováno v:
J Mol Cell Cardiol
Myocardial ischemia/reperfusion (MI/R) causes loss of cardiomyocytes via oxidative stress-induced cardiomyocyte apoptosis. miR322, orthologous to human miR-424, was identified as an ischemia-induced angiogenic miRNA, but its cellular source and funct
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ab4c41c934baeeb7fb1f7acbfdf7bf76
https://doi.org/10.1016/j.yjmcc.2019.05.020
https://doi.org/10.1016/j.yjmcc.2019.05.020
Autor:
David Kim, Mourad Ogbi, Yao Liang Tang, Ha Won Kim, Neal L. Weintraub, Abdalrahman Zarzour, David W. Stepp, Tyler W. Benson, Xin Yun Lu, Weiqin Chen, Samah Ahmadieh, Brandee Goo, Renee Hilton, Charlotte Greenway, Vijay Patel, David Y. Hui, Nicole K.H. Yiew
Publikováno v:
J Biol Chem
Enhancer of zeste homolog 2 (EZH2), an epigenetic regulator that plays a key role in cell differentiation and oncogenesis, was reported to promote adipogenic differentiation in vitro by catalyzing trimethylation of histone 3 lysine 27. However, inhib
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2ad4635e9139e48568d74db506a48ce6
https://doi.org/10.1074/jbc.ra118.006871
https://doi.org/10.1074/jbc.ra118.006871
Autor:
Walter J. Koch, Namakkal Soorapan Rajasekaran, Timothy J. Kamp, Brenda M. Ogle, William T. Pu, Peipei Ping, Keitaro Domae, Song Li, Wuqiang Zhu, Huang-Tian Yang, Daniel J. Garry, Yabing Chen, Jianyi Zhang, John P. Cooke, Philippe Menasché, Roberto Bolli, Joseph C. Wu, Lei Ye, Shijun Hu, Sumanth D. Prabhu, Jack M. Rogers, Ying Ge, Prasanna Krishnamurthy, Michael Davis, Yibin Wang, Yao Liang Tang, Gordana Vunjak-Novakovic, Joel L. Berry, Nenad Bursac, Ke Cheng, Ronglih Liao, E. Dale Abel, Gangjian Qin
Publikováno v:
Circulation research, vol 124, iss 1
On March 1 and 2, 2018, the National Institutes of Health 2018 Progenitor Cell Translational Consortium (PCTC) and Cardiovascular Bioengineering Symposium (CVBE) was held at the University of Alabama at Birmingham. Convergence of life sciences and en
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b682faa0acaea3abb7cea441002632ad
https://doi.org/10.1161/circresaha.118.314216
https://doi.org/10.1161/circresaha.118.314216
Autor:
Hong Hu, Mark Maienschein-Cline, Muhammad Ashraf, Yan Wang, Ailia Ali, Yao Liang Tang, Wanling Xuan
Publikováno v:
Shock. 50:627-639
Cardiac progenitor cells (CPCs) being multipotent offer a promising source for cardiac repair due to their ability to proliferate and multiply into cardiac lineage cells. Here, we explored a novel strategy for human CPCs generation from human induced
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::893d2806d5348fb7d8e1efde96678087
https://doi.org/10.1097/shk.0000000000001133
https://doi.org/10.1097/shk.0000000000001133
Autor:
Tatsuya Aonuma, Il-Man Kim, Satoshi Kawaguchi, R. Mark Payne, Suthat Liangpunsakul, Marisa N Sepúlveda, Nipuni P Barupala, Monte S. Willis, Bruno Moukette, Christopher Corr, Yao Liang Tang
Publikováno v:
JCI Insight
MicroRNA-150 (miR-150) is downregulated in patients with multiple cardiovascular diseases and in diverse mouse models of heart failure (HF). miR-150 is significantly associated with HF severity and outcome in humans. We previously reported that miR-1
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5415c3466c4bd499b90ef117856a681e
https://pubmed.ncbi.nlm.nih.gov/34403363
https://pubmed.ncbi.nlm.nih.gov/34403363