Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Yann, Tonin"'
Autor:
Yann Tonin, Nina Entelis
Publikováno v:
médecine/sciences. 30:1101-1109
Defects in mitochondrial genome can cause a wide range of clinical disorders, mainly neuromuscular diseases. Various strategies have been proposed to address these pathologies; unfortunately no efficient treatment is currently available. In some case
Autor:
Ilya, Dovydenko, Anne-Marie, Heckel, Yann, Tonin, Ali, Gowher, Alya, Venyaminova, Ivan, Tarassov, Nina, Entelis
Publikováno v:
Methods in molecular biology (Clifton, N.J.). 1265
Mitochondrial import of small noncoding RNA is found in a large variety of species. In mammalian cells, this pathway can be used for therapeutic purpose, to restore the mitochondrial functions affected by pathogenic mutations. Recently, we developed
Autor:
Ilya S. Dovydenko, Ali Gowher, Ivan Tarassov, Yann Tonin, Alya G. Venyaminova, Nina Entelis, Anne-Marie Heckel
Publikováno v:
Methods in Molecular Biology ISBN: 9781493922871
Mitochondrial import of small noncoding RNA is found in a large variety of species. In mammalian cells, this pathway can be used for therapeutic purpose, to restore the mitochondrial functions affected by pathogenic mutations. Recently, we developed
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::6efca3b85425385032aa57cb267bd5cf
https://doi.org/10.1007/978-1-4939-2288-8_16
https://doi.org/10.1007/978-1-4939-2288-8_16
Autor:
Yann, Tonin, Nina, Entelis
Publikováno v:
Medecine sciences : M/S. 30(12)
Defects in mitochondrial genome can cause a wide range of clinical disorders, mainly neuromuscular diseases. Various strategies have been proposed to address these pathologies; unfortunately no efficient treatment is currently available. In some case
Autor:
Anne-Marie Heckel, Ilya S. Dovydenko, Agnès Rötig, Yann Tonin, Ivan Tarassov, Mikhail Y. Vysokikh, Arnold Munnich, Alya G. Venyaminova, Mariya I. Meschaninova, Nina Entelis
Defects in mitochondrial genome can cause a wide range of clinical disorders, mainly neuromuscular diseases. Presently, no efficient therapeutic treatment has been developed against this class of pathologies. Because most of deleterious mitochondrial
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::063235b5b4b745299c5d5f08f64be635
https://europepmc.org/articles/PMC4036341/
https://europepmc.org/articles/PMC4036341/
Autor:
Mariya I. Meschaninova, Ivan Tarassov, Anne-Marie Heckel, Nina Entelis, Dmitrii V. Pyshnyi, Yann Tonin, Caroline Comte, Alya G. Venyaminova, Ilya S. Dovydenko
Publikováno v:
Biochimie. 100
Defects in mitochondrial genome can cause a wide range of clinical disorders, mainly neuromuscular diseases. Most of the deleterious mitochondrial mutations are heteroplasmic, meaning that wild type and mutated forms of mtDNA coexist in the same cell
Autor:
Nina Entelis, Yann Tonin, Alexandre Smirnov, Ivan Tarassov, Karine Auré, Anne Lombès, Abdeldjalil Boucheham, Caroline Comte, Robert P. Martin, Anne-Marie Heckel-Mager
Publikováno v:
Nucleic Acids Research
Nucleic Acids Research, 2013, 41 (1), pp.418-433. ⟨10.1093/nar/gks965⟩
Nucleic Acids Research, Oxford University Press, 2013, 41 (1), pp.418-433. ⟨10.1093/nar/gks965⟩
Nucleic Acids Research, 2013, 41 (1), pp.418-433. ⟨10.1093/nar/gks965⟩
Nucleic Acids Research, Oxford University Press, 2013, 41 (1), pp.418-433. ⟨10.1093/nar/gks965⟩
International audience; Mitochondrial mutations, an important cause of incurable human neuromuscular diseases, are mostly heteroplasmic: mutated mitochondrial DNA is present in cells simultaneously with wild-type genomes, the pathogenic threshold bei
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7e50a0b2e208d144f39663197b4504dc
https://hal.sorbonne-universite.fr/hal-01587401/document
https://hal.sorbonne-universite.fr/hal-01587401/document
Publikováno v:
Translation in Mitochondria and Other Organelles ISBN: 9783642394256
Mitochondrial translation depends on the macromolecular components imported from the cytosol, which include translation factors, ribosomal proteins, aminoacyl-tRNA synthetases, and a variable number of small noncoding RNAs. The lasts are essentially
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::03acf6ccac94da2f715650595f9ec679
https://doi.org/10.1007/978-3-642-39426-3_4
https://doi.org/10.1007/978-3-642-39426-3_4