Zobrazeno 1 - 10
of 25
pro vyhledávání: '"Yanfang Rui"'
Publikováno v:
eNeuro
Oligodendrocytes (OLs) insulate axonal fibers for fast conduction of nerve impulses by wrapping axons of the CNS with compact myelin membranes. Differentiating OLs undergo drastic chances in cell morphology. Bipolar oligodendroglial precursor cells (
Autor:
Yanfang Rui1,2, Zheng, James Q.1,2,3 james.zheng@emory.edu
Publikováno v:
Molecular Brain. 8/17/2016, Vol. 9, p1-12. 12p.
Autor:
Yanfang Rui, Velia M. Fowler, Kuai Yu, Omotola F. Omotade, H. Criss Hartzell, Wenliang Lei, James Q. Zheng
Publikováno v:
The Journal of neuroscience : the official journal of the Society for Neuroscience. 38(48)
Neurons of the CNS elaborate highly branched dendritic arbors that host numerous dendritic spines, which serve as the postsynaptic platform for most excitatory synapses. The actin cytoskeleton plays an important role in dendrite development and spine
Publikováno v:
eNeuro; 2020, Vol. 7 Issue 4, p1-16, 16p
Autor:
Yanfang Rui1, Jiaping Gu1, Kuai Yu1, Hartzell, H. Criss1, Zheng, James Q.1 james.zheng@emory.edu
Publikováno v:
Molecular Brain. 2010, Vol. 3, p10-22. 13p.
Autor:
James Q. Zheng, Kenneth R. Myers, Wenliang Lei, Denis Tsygankov, Yanfang Rui, Siarhei Hladyshau
Publikováno v:
The Journal of Cell Biology
Katalin Schlett previews the study by Lei et al., which reveals that dendritic spines contain local pools of G-actin that are dynamically regulated in response to synaptic activity by profilin and PIP3.
Synaptic activity reshapes the morphology
Synaptic activity reshapes the morphology
Autor:
Andrew N. Bankston, Yue Feng, Megan E. Allen, Yanfang Rui, Wenqi Li, Guanglu Liu, Li Ku, James Q. Zheng
Publikováno v:
The Journal of neuroscience : the official journal of the Society for Neuroscience. 36(44)
Two distinct protein cofactors, p35 and p39, independently activate Cyclin-dependent kinase 5 (Cdk5), which plays diverse roles in normal brain function and the pathogenesis of many neurological diseases. The initial discovery that loss of p35 impair
Autor:
Yanfang Rui, Zheng, James
Supplemental figures S1-S7 and methods. (PDF 902 kb)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9dc2f7a997bfcc2f9f4a5f8c5b2dbdbd
Autor:
Kuai Yu, Hartzell, H. Criss, Omotade, Omotola F., Yanfang Rui, Wenliang Lei, Zheng, James Q., Fowler, Velia M.
Publikováno v:
Journal of Neuroscience; 11/28/2018, Vol. 38 Issue 48, p10271-10285, 15p
Publikováno v:
Molecular Brain
Molecular Brain, Vol 3, Iss 1, p 10 (2010)
Molecular Brain, Vol 3, Iss 1, p 10 (2010)
Background Synaptic defects represent a major mechanism underlying altered brain functions of patients suffering Alzheimer's disease (AD) [1–3]. An increasing body of work indicates that the oligomeric forms of β-amyloid (Aβ) molecules exert prof