Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Ya-Zhen Mo"'
Publikováno v:
Toxicon. 55:888-896
It has been recognized that phospholipase A2 (PLA2) is a crucial factor of snake venom induced inflammation. Recently, promutoxin, a novel member of minor subgroup of snake venom PLA2 (R49 PLA2) has been characterized in our laboratory, but its roles
Autor:
Hua Xie, Xiao-Long Wei, Ji-Fu Wei, Wan-Yu Wang, Shao-Heng He, Yu-Liang Xiong, Yi-Ling Fu, Li-Ya Qiao, Ya-Zhen Mo, Han-Qiu Chen
Publikováno v:
The International Journal of Biochemistry & Cell Biology. 38:510-520
Poisonous snakebite wound is a popular disease worldwide. However, the pathogenesis remains unclear. In the present study, a novel metalloproteinase atrahagin in Chinese cobra (Naja atra) snake venom was purified, using heparin–sepharose followed b
Publikováno v:
BioMed Research International, Vol 2013 (2013)
BioMed Research International
BioMed Research International
Local inflammation is a prominent characteristic of snakebite wound, and snake-venom phospholipase A2s (PLA2s) are some of the main component that contribute to accumulation of inflammatory cells. However, the action of an R49 PLA2s, promutoxin fromP
Publikováno v:
BioMed Research International; 2013, Vol. 2013, p1-5, 5p
Publikováno v:
BMC Immunology; 2009, Vol. 10, Special section p1-14, 14p, 5 Charts, 6 Graphs
Autor:
Ji-Fu Wei1,2,3, Xiao-Long Wei2, Ya-Zhen Mo2, Haiwei Yang1, Shaoheng He1,2 shoahenghe@hotmail.com
Publikováno v:
BioMed Research International. 2013, Vol. 2013, p1-5. 5p.
Autor:
Ji-Fu Wei1,2 weijifu@hotmail.com, Xiao-Long Wei2 g_xlwei@stu.edu.cn, Ya-Zhen Mo2 g_yzmo@stu.edu.cn, Shao-Heng He1,2 shoahenghe@hotmail.com
Publikováno v:
BMC Immunology. 2009, Vol. 10, Special section p1-14. 14p. 5 Charts, 6 Graphs.
Publikováno v:
BMC Immunology
BMC Immunology, Vol 10, Iss 1, p 21 (2009)
BMC Immunology, Vol 10, Iss 1, p 21 (2009)
Background It has been recognized that phospholipase A2 (PLA2) is a crucial component of snake venom, which contributes greatly to snake venom induced inflammation in man. However, the mechanisms through which N49 PLA2 provoke inflammation remain unc