Zobrazeno 1 - 10
of 42
pro vyhledávání: '"YUNPING DENG"'
Publikováno v:
Frontiers in Cellular Neuroscience, Vol 17 (2023)
Externí odkaz:
https://doaj.org/article/7e8378193800436982a65973ab717f0f
Publikováno v:
Frontiers in Cellular Neuroscience, Vol 15 (2021)
Externí odkaz:
https://doaj.org/article/db9a38a939ef44f3be2b0ee85515966d
Autor:
Anton Reiner, Wei Bu, Huiling Ren, Yunping Deng, Nobel Del Mar, Natalie Hart Guley, Bob M Moore, Marcia G Honig
Publikováno v:
Frontiers in Neuroscience, Vol 10 (2016)
We have previously reported that mild TBI created by focal left-side cranial blast in mice produces widespread axonal injury, microglial activation, and a variety of functional deficits. We have also shown that these functional deficits are reduced b
Externí odkaz:
https://doaj.org/article/d81c0c54fd6343be96ac8b72d345bebd
Publikováno v:
International Archives of Allergy and Immunology. :1-11
Introduction: Chronic rhinosinusitis with nasal polyps (CRSwNPs) in China is characterized by a mixed eosinophilic-neutrophilic inflammation, linking to a more heterogeneous clinical phenotype. However, the relationship between eosinophilic and neutr
Publikováno v:
The Journal of Comparative Neurology
We used behavioral testing and morphological methods to detail the progression of basal ganglia neuron type‐specific pathology and the deficits stemming from them in male heterozygous Q175 mice, compared to age‐matched WT males. A rotarod deficit
Publikováno v:
Frontiers in Cellular Neuroscience, Vol 15 (2021)
Publikováno v:
Neurosci Lett
We have found that daily subcutaneous injection with a maximum tolerated dose of the mGluR2/3 agonist LY379268 (20 mg/kg) beginning at 4 weeks of age dramatically improves the motor, neuronal and neurochemical phenotype in R6/2 mice, a rapidly progre
Autor:
Anton Reiner, Yunping Deng
Publikováno v:
Journal of Comparative Neurology. 524:3518-3529
We have previously found that thalamostriatal axodendritic terminals are reduced as early as 1 month of age in heterozygous Q140 HD mice (Deng et al. [] Neurobiol Dis 60:89-107). Because cholinergic interneurons are a major target of thalamic axodend
Autor:
Yunping Deng, Anton Reiner
Publikováno v:
CNS neurosciencetherapeutics. 24(4)
Huntington's disease (HD) is a hereditary progressive neurodegenerative disorder caused by a CAG repeat expansion in the gene coding for the protein huntingtin, resulting in a pathogenic expansion of the polyglutamine tract in the N-terminus of this