Zobrazeno 1 - 10
of 42
pro vyhledávání: '"YUET-CHING TAY"'
Publikováno v:
Nephrology. 4:57-64
SUMMARY: The effect of mild acute tubular injury on the progression of tubulointerstitial fibrosis was studied in pair-fed uninephrectomized male Wistar rats with established adriamycin nephrosis (n= 34). Rats were stratified into three groups accord
Autor:
David Harris, Gopala K. Rangan, Yiping Wang, Shisan Bao, Yuet-Ching Tay, Lukas Kairaitis, Deepika Mahajan, Tim Spicer
Publikováno v:
Nephrology. 10:470-477
SUMMARY: Background: Because macrophages are considered to be possible effectors of disease in Adriamycin (ADR) nephrosis, we hypothesized that depletion of macrophages might protect against the initiation of renal injury. In the present study, a mon
Publikováno v:
Kidney International. 68:391-398
Can murine diabetic nephropathy be separated from superimposed acute renal failure? Background Streptozotocin (STZ) is commonly used to induce diabetes in experimental animal models, but not without accompanying cytotoxic effects. This study was unde
Autor:
Yuet-Ching Tay, Huiling Wu, David Harris, Yiping Wang, Guoping Zheng, Chun Zhang, Stephen I. Alexander
Publikováno v:
Kidney International. 67:2178-2186
DNA vaccination with naked DNA encoding MCP-1 and RANTES protects against renal injury in adriamycin nephropathy.BackgroundWe have previously shown that monocyte chemoattractant protein-1 (MCP-1) and regulated upon activation, normal T-cell expressed
Publikováno v:
Renal Failure. 27:609-614
In a previous study we found that an episode of acute subclinical nephrotoxicity with gentamicin (G) (but not that induced by another proximal tubular cell nephrotoxin: ferric nitrilotriacetate, FeNTA), paradoxically reduced the progression of renal
Autor:
Yang Wang, Ximin Feng, Shounan Yi, David Harris, Yiping Wang, Yuet-Ching Tay, Lukas Kairaitis
Publikováno v:
Kidney International. 61:1303-1311
Transfection of tubule cells with Fas ligand causes leukocyte apoptosis.BackgroundSince the Fas/Fas Ligand (FasL) interaction is recognized as a major pathway of apoptosis in immune cells, we hypothesized that selective expression of FasL by tubular
Autor:
Yiping Wang, Gopala K. Rangan, Ximin Feng, Shounan Yi, Shisan Bao, Yuet-Ching Tay, Yang Wang, Lukas Kairaitis, David Harris
Publikováno v:
Kidney International. 59:975-984
Depletion of CD4+ T cells aggravates glomerular and interstitial injury in murine adriamycin nephropathy.BackgroundCD4+ T cells play an important role in various types of immunologic renal disease, including lupus nephritis, IgA nephropathy, and cres
Publikováno v:
Renal Failure. 23:773-780
The persistence of NF-kappaB independent inflammatory signals in the cortical tubulointerstitium may explain the incomplete suppression of interstitial monocyte accumulation by the antioxidant NF-kappaB inhibitor, pyrrolidine dithiocarbamate (PDTC),
Publikováno v:
Kidney International. 58(4):1797-1804
Progressive adriamycin nephropathy in mice: Sequence of histologic and immunohistochemical events. Background As an experimental analogue of human focal glomerular sclerosis (FGS), adriamycin (ADR)-induced nephropathy is well-characterized in rats. P
Publikováno v:
Kidney International. 57:2011-2022
Lipopolysaccharide-induced MCP-1 gene expression in rat tubular epithelial cells is nuclear factor-kB dependent. Background Endotoxin is an important factor in the development of acute renal failure related to infection and in acceleration of chronic