Zobrazeno 1 - 10
of 28
pro vyhledávání: '"YI‑DENG JIANG"'
Publikováno v:
Neural Regeneration Research, Vol 13, Iss 2, Pp 272-279 (2018)
Hyperhomocysteinemia has been shown to be associated with neurodegenerative diseases; however, lesions or histological changes and mechanisms underlying homocysteine-induced injury in olfactory bulb neurons remain unclear. In this study, hyperhomocys
Externí odkaz:
https://doaj.org/article/3a43d08c5f734c0ebed633c03d81ae04
Autor:
Ling‑Bo Xu, An‑Ning Yang, Jue Tian, Yun Jiao, Yin‑Ju Hao, Hua Xu, Hui‑Ping Zhang, Cai‑Yan Mao, Xiao‑Ling Yang, Sheng‑Chao Ma, Yan‑Hua Wang, Ming‑Hao Zhang, Yi‑Deng Jiang, Shao‑Ju Jin
Publikováno v:
Molecular Medicine Reports. 16:9181-9188
Atherosclerosis (AS) is a progressive disease of multifactorial origin, which occurs in response to endothelial injury. Increased homocysteine (Hcy) is considered a major cause of endothelial dysfunction, oxidative stress and DNA methylation; however
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2206562301d5c4951456229b5f3820e2
https://doi.org/10.3892/mmr.2017.7753
https://doi.org/10.3892/mmr.2017.7753
Autor:
Yan‑Hua Wang, Yang‑Yang He, Ming‑Hao Zhang, Sheng‑Chao Ma, Hui Zhang, Hua Xu, Cheng Yang, Fan‑Qi Kong, Yi‑Deng Jiang, Zheng Yu, Xiao‑Ling Yang, Hui‑Ping Zhang, Ju Tian, An‑Ning Yang
Publikováno v:
Molecular Medicine Reports
The aim of the present study was to identify an effective method for detecting early‑phase atherosclerosis (AS), as well as to provide useful DNA methylation profiles to serve as biomarkers for the detection of AS. A total of 300 individuals (150 A
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::59feb92c3fca859773457dbe2b79c056
https://doi.org/10.3892/mmr.2016.5120
https://doi.org/10.3892/mmr.2016.5120
Autor:
Yan‑Hua Wang, Xiao‑Ming Yang, Hua Xu, Yi‑Deng Jiang, Hui‑Ping Zhang, Sheng‑Chao Ma, Xue‑Bo Han, Xiao‑Ling Yang, Jue Tian, Cheng‑Jian Cao, An‑Ning Yang, Ming‑Hao Zhang
Publikováno v:
Molecular Medicine Reports. 13:483-490
Accumulating evidence has suggested that homocysteine (Hcy) is an independent risk factor for atherosclerosis (AS). Hcy can promote vascular smooth muscle cell (VSMC) proliferation, which is pivotal in the pathogenesis and progression of AS. The aim
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ca2a28379c1d157a8da9e556567cf662
https://doi.org/10.3892/mmr.2015.4558
https://doi.org/10.3892/mmr.2015.4558
Autor:
Yan‑Hua Wang, Sheng‑Chao Ma, Hui‑Ping Zhang, Hui Zhang, Yun Jiao, Yi‑Deng Jiang, Xiao‑Ling Yang, An‑Ning Yang
Publikováno v:
Molecular medicine reports. 17(4)
Vascular smooth muscle cell (VSMC) proliferation is a primary pathological event in the development of atherosclerosis (AS), and the presence of homocysteine (Hcy) acts as an independent risk factor for AS. However, the underlying mechanisms remain t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::aef17a6a23da4578704db105a24d5d4b
https://pubmed.ncbi.nlm.nih.gov/29393420
https://pubmed.ncbi.nlm.nih.gov/29393420
Autor:
Yi‑Deng Jiang, Sheng‑Chao Ma, Ning Ding, Xiao‑Ling Yang, Yan‑Hua Wang, Hui‑Ping Zhang, An‑Ning Yang, Guan‑Jun Lu, Hui Zhang
Publikováno v:
International journal of molecular medicine.
Ahead of Print article withdrawn by publisher.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3b7f940cc34717ee829575785a50ef2c
https://pubmed.ncbi.nlm.nih.gov/30431063
https://pubmed.ncbi.nlm.nih.gov/30431063
Autor:
Jue Tian, Sheng‑Chao Ma, An‑Ning Yang, Xiao‑Ming Yang, Yang‑Yang He, Jian‑Cheng Cao, Shao‑Ju Jin, Yan‑Hua Wang, Yun Jiao, Guan‑Jun Lu, Yi‑Deng Jiang, Hui Zhang, Hui‑Ping Zhang, Xiao‑Ling Yang, Ming‑Hao Zhang, Yue‑Xia Jia
Publikováno v:
Molecular medicine reports. 16(5)
Vascular smooth muscle cell (VSMC) proliferation is a primary pathological event in atherosclerosis (AS), and homocysteine (Hcy) is an independent risk factor for AS. However, the underlying mechanisms are still lagging. Studies have used the combina
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f5262cffdf617c2656e13541956e7006
https://pubmed.ncbi.nlm.nih.gov/28944836
https://pubmed.ncbi.nlm.nih.gov/28944836
Publikováno v:
Neural Regeneration Research, Vol 13, Iss 2, Pp 272-279 (2018)
Neural Regeneration Research
Neural Regeneration Research
Hyperhomocysteinemia has been shown to be associated with neurodegenerative diseases; however, lesions or histological changes and mechanisms underlying homocysteine-induced injury in olfactory bulb neurons remain unclear. In this study, hyperhomocys
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::32f9612d35ae9fc13b03cfc96c20f871
https://doi.org/10.4103/1673-5374.220779
https://doi.org/10.4103/1673-5374.220779
Autor:
Li, Zhao, Yun, Jiao, An-Ning, Yang, Cheng-Jian, Cao, Fan-Qi, Kong, Xian-Mei, Liu, Xiao-Ling, Yang, Yi-Deng, Jiang
Publikováno v:
Sheng li xue bao : [Acta physiologica Sinica]. 67(2)
The aim of the present study is to explore the role of miR-124 and its promoter region DNA methylation in homocysteine (Hcy)-induced atherosclerosis. ApoE(-/-) mice were fed with hypermethionine diet for 16 weeks to duplicate hyperhomocysteinemia mod
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::11a6e4c7e5e492ddbf817d53656a8b28
https://pubmed.ncbi.nlm.nih.gov/25896052
https://pubmed.ncbi.nlm.nih.gov/25896052
Autor:
Xiu-Yu, Wang, Ming-Hao, Zhang, Mei-Ling, Yang, Yi-Deng, Jiang, Gui-Zhong, Li, Xiao-Ling, Yang, Hua, Xu, Jun, Cao
Publikováno v:
Zhongguo Zhong yao za zhi = Zhongguo zhongyao zazhi = China journal of Chinese materia medica. 38(16)
To explore the effect of oxymatrine (OMT) on JAK2/STAT3 signaling in renal tissues of rats with septic shock.The cecal ligation and puncture (CLP) was adopted to establish the rat septic shock model. Fifty-six male SD rats were randomly divided into
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::20e8842ca62decdefc6c48bc2ad35945
https://pubmed.ncbi.nlm.nih.gov/24228589
https://pubmed.ncbi.nlm.nih.gov/24228589