Zobrazeno 1 - 10
of 259
pro vyhledávání: '"Y. Pawlotsky"'
Autor:
Pierre Brissot, Pascal Guggenbuhl, Gérard Chalès, Anne-Marie Jouanolle, Yves Deugnier, M Catheline, Romain Moirand, P. Le Dantec, J. Meadeb, Y. Pawlotsky
Publikováno v:
Arthritis & Rheumatism. 42:799-806
Objective To determine whether the osteoarticular changes associated with genetic hemochromatosis could be explained by metabolic parathyroid hormone (PTH) disorders. Methods The study involved 210 patients with liver iron overload syndromes. Osteoar
Autor:
Zeng, Shaoxiong1,2 (AUTHOR), Liu, Zhenzhen3 (AUTHOR), Ke, Bilun1 (AUTHOR), Zhang, Yiwang4 (AUTHOR), Wang, Qian1 (AUTHOR), Tan, Siwei1 (AUTHOR) tansw@mail.sysu.edu.cn
Publikováno v:
BMC Infectious Diseases. 6/26/2024, Vol. 24 Issue 1, p1-9. 9p.
Autor:
Chardevel F, E. Legrand, J. Meadeb, Renée Fauchet, G. Chales, Y. Pawlotsky, F. Quillivic, Aleth Perdriger, Gilbert Semana
Publikováno v:
Tissue Antigens. 39:14-18
HLA-DP polymorphism was examined in 71 rheumatoid arthritis patients and 148 controls, using dot-blot analysis with 14 synthetic oligonucleotide probes specific for the variable region of the DPB1 second exon. The DPB1 0401 allele was found to be sig
Publikováno v:
La Revue du praticien. 45(8)
Autor:
Y. Pawlotsky, J F Boisdet, Yan Rolland, Yves Deugnier, Aleth Perdriger, Gérard Chalès, Pascal Guggenbuhl
Publikováno v:
Osteoporosis international : a journal established as result of cooperation between the European Foundation for Osteoporosis and the National Osteoporosis Foundation of the USA. 16(12)
Genetic hemochromatosis (GH) is an iron overload disorder mainly due to the C282Y mutation of the HFE gene. The possibility of bone involvement was only recently recognized. The aims of this study were to assess bone mineral density (BMD) and bone re
Autor:
M N Bonnard, Y. Pawlotsky, Pascal Guggenbuhl, J Yaouanq, Gilbert Semana, Aleth Perdriger, E Quelvennec, Gérard Chalès
Publikováno v:
Rheumatology
Rheumatology, Oxford University Press (OUP), 1999, 38 (5), pp.448-52. ⟨10.1093/rheumatology/38.5.448⟩
Rheumatology, 1999, 38 (5), pp.448-52. ⟨10.1093/rheumatology/38.5.448⟩
Rheumatology, Oxford University Press (OUP), 1999, 38 (5), pp.448-52. ⟨10.1093/rheumatology/38.5.448⟩
Rheumatology, 1999, 38 (5), pp.448-52. ⟨10.1093/rheumatology/38.5.448⟩
Objective. HLA DM is a non-classical major histocompatibility complex (MHC) class II molecule that has been shown to facilitate peptide loading with classical class II molecules. Methods. In this study, we analysed the polymorphism in exon 3 of HLA D
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f025c1ca8014fdd861c7801aeba7b106
https://hal.archives-ouvertes.fr/hal-00992068
https://hal.archives-ouvertes.fr/hal-00992068
Autor:
Y, Pawlotsky, P, Le Dantec, R, Moirand, P, Guggenbuhl, A M, Jouanolle, M, Catheline, J, Meadeb, P, Brissot, Y, Deugnier, G, Chalès
Publikováno v:
Arthritis and rheumatism. 42(4)
To determine whether the osteoarticular changes associated with genetic hemochromatosis could be explained by metabolic parathyroid hormone (PTH) disorders.The study involved 210 patients with liver iron overload syndromes. Osteoarticular changes wer
Autor:
S, Jean, E, Quelvennec, M, Alizadeh, P, Guggenbuhl, B, Birebent, A, Perdriger, B, Grosbois, P Y, Pawlotsky, G, Semana
Publikováno v:
Clinical and experimental rheumatology. 16(6)
Sjögren's syndrome (SS) is a chronic autoimmune disease with a genetic component. Among the genetic factors, the role of HLA class II genes has been suggested and a positive association with DRB1*03 allele has been described. However, there is no co
Autor:
P, Guggenbuhl, S, Jean, P, Jego, B, Grosbois, G, Chalès, G, Semana, G, Lancien, E, Veillard, Y, Pawlotsky, A, Perdriger
Publikováno v:
The Journal of rheumatology. 25(5)
To examine the respective role of the DRB1*, DQB1*, and DPB1* HLA alleles in primary Sjögren's syndrome (SS) and in the clinical and autoantibody profile of primary SS.HLA-DRB1*, DQB1*, and DPB1* alleles were analyzed in 42 patients with primary SS
Publikováno v:
The Journal of rheumatology. 24(7)
To investigate the role of HLA-DRB1 genotypes and HLA-DRB1*0401-DQB1*03 haplotypes in the expression of extraarticular manifestations and rheumatoid factor (RF) in rheumatoid arthritis (RA).189 patients with RA were classified according to the presen