Zobrazeno 1 - 10
of 78
pro vyhledávání: '"Xiu-Ling DENG"'
Autor:
Yu Zhang, Jing-Jing Li, Rui Xu, Xin-Pei Wang, Xin-Yi Zhao, Yuan Fang, Yu-Peng Chen, Shan Ma, Xiao-Hui Di, Wei Wu, Gang She, Zheng-Da Pang, Yi-Dong Wang, Xing Zhang, Wenjun Xie, Xiu-Ling Deng, Xiao-Jun Du, Yi Zhang
Publikováno v:
Redox Biology, Vol 68, Iss , Pp 102944- (2023)
Aims: Endothelial dysfunction plays a pivotal role in atherosclerosis, but the detailed mechanism remains incomplete understood. Nogo-B is an endoplasmic reticulum (ER)-localized protein mediating ER-mitochondrial morphology. We previously showed end
Externí odkaz:
https://doaj.org/article/66907925c4c1495da7a001e602e430cf
Autor:
Yi Zhang, Xiao-Zhen Ma, Xin-Yi Zhao, Jing-Jing Li, Shan Ma, Zheng-Da Pang, Jing Xu, Xiao-Jun Du, Xiu-Ling Deng, Jun-Hong Wang
Publikováno v:
Heliyon, Vol 9, Iss 4, Pp e14823- (2023)
Type 2 diabetes mellitus (T2DM) is characterized by chronic low-grade systemic inflammation. Tissue infiltration by monocyte migration contributes to the pathogenesis of vascular complications in T2DM. We studied the role of intermediate-conductance
Externí odkaz:
https://doaj.org/article/fa0d579649c7403cbc02094a04ce163a
Publikováno v:
Frontiers in Genetics, Vol 13 (2022)
Background: Mitochondrial dysfunction is implicated in the development of cardiomyopathy and heart failure. Transcription of mitochondrial DNA (mtDNA) encoded genes and subsequent protein synthesis are tightly regulated by nuclear DNA (nDNA) encoded
Externí odkaz:
https://doaj.org/article/a669fb739bc04786807a0a11eda0b73a
Publikováno v:
Frontiers in Cardiovascular Medicine, Vol 9 (2022)
BackgroundCardiac rupture (CR) and left ventricular thrombus (LVT) remain important complications of acute myocardial infarction (MI), and they are currently regarded as independent events. We explored the pathogenetic link between CR and LVT by inve
Externí odkaz:
https://doaj.org/article/e18e6dbc2bbc4417ac1fd0dda9bf3e70
Autor:
Gang She, Yu‐Jie Ren, Yan Wang, Meng‐Chen Hou, Hui‐Fang Wang, Wei Gou, Bao‐Chang Lai, Ting Lei, Xiao‐Jun Du, Xiu‐Ling Deng
Publikováno v:
Journal of the American Heart Association: Cardiovascular and Cerebrovascular Disease, Vol 8, Iss 1 (2019)
Background Cardiac fibrosis is a core pathological process associated with heart failure. The recruitment and differentiation of primitive fibroblast precursor cells of bone marrow origin play a critical role in pathological interstitial cardiac fibr
Externí odkaz:
https://doaj.org/article/9a8492e5c8314b38bd1ceb87cc83f052
Publikováno v:
PLoS ONE, Vol 12, Iss 8, p e0183708 (2017)
The present study investigated the effects of equol on cardiovascular K+ channel currents. The cardiovascular K+ channel currents were determined in HEK 293 cells stably expressing cloned differential cardiovascular K+ channels with conventional whol
Externí odkaz:
https://doaj.org/article/3e7794aced634c9ba11aa2737dd86aff
Publikováno v:
PLoS ONE, Vol 8, Iss 11, p e79952 (2013)
Cholesterol is one of the major lipid components of the plasma membrane in mammalian cells and is involved in the regulation of a number of ion channels. The present study investigates how large conductance Ca(2+)-activated K(+) (BK) channels are reg
Externí odkaz:
https://doaj.org/article/3ce7195a92084635b11a5f18798e2ca4
Autor:
Jing-Jing Li, Xin-Yi Zhao, Yan Wang, Rui Xu, Xiao-Hui Di, Yu Zhang, Hongyan Yang, Meng-Zhuan Han, Ru-Yue Bai, Lin Xie, Zheng-Da Pang, Xing Zhang, Jianbao Zhang, Xiao-Jun Du, Xiu-Ling Deng, Yi Zhang, Wenjun Xie
Publikováno v:
Arteriosclerosis, Thrombosis, and Vascular Biology. 43:726-738
Background: S1P (sphingosine-1-phosphate) has been reported to possess vasodilatory properties, but the underlying pathways are largely unknown. Methods: Isolated mouse mesenteric artery and endothelial cell models were used to determine S1P-induced
Autor:
Wei Wu, Qun Lu, Shan Ma, Jin-Chan Du, Kevin Huynh, Thy Duong, Zhang-Da Pang, Daniel Donner, Peter J. Meikle, Xiu-Ling Deng, Xiao-Jun Du
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 324:H528-H541
Takotsubo syndrome (TTS) is featured by activation of sympatho-β-adrenoceptor (βAR) system leading to acute loss of ventricular contractile performance. However, the molecular mechanism remains undefined. We demonstrated, in an isoproterenol-induce
Publikováno v:
Journal of Cardiovascular Pharmacology. 81:259-269