Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Xinghao S. Shuai"'
Autor:
Maham Rais, Jonathan W. Lovelace, Xinghao S. Shuai, Walker Woodard, Steven Bishay, Leo Estrada, Ashwin R. Sharma, Austin Nguy, Anna Kulinich, Patricia S. Pirbhoy, Arnold R. Palacios, David L. Nelson, Khaleel A. Razak, Iryna M. Ethell
Publikováno v:
Neurobiology of Disease, Vol 162, Iss , Pp 105577- (2022)
Background: Fragile X syndrome (FXS) is a leading genetic cause of autism and intellectual disability with cortical hyperexcitability and sensory hypersensitivity attributed to loss and hypofunction of inhibitory parvalbumin-expressing (PV) cells. Ou
Externí odkaz:
https://doaj.org/article/59d4773c4e2a47b0bd3f51d0b57d3dd3
Autor:
Maham Rais, Anna O. Kulinich, Victoria Wagner, Walker Woodard, Xinghao S. Shuai, Samantha N. Sutley, Jamiela Kokash, Timo P. Piepponen, Maija Castren, Khaleel A. Razak, Iryna M. Ethell
Fragile X syndrome (FXS) is a leading genetic cause of autism-like symptoms associated with sensory hypersensitivity and cortical hyperexcitability. Recent observations in humans and Fmr1 knockout (KO) animal models of FXS suggest abnormal GABAergic
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d4365dedfbbc531ad973e33e208945ac
https://doi.org/10.1101/2022.02.08.479618
https://doi.org/10.1101/2022.02.08.479618
Autor:
Maham Rais, Jonathan W. Lovelace, Xinghao S. Shuai, Walker Woodard, Steven Bishay, Leo Estrada, Ashwin R. Sharma, Austin Nguy, Anna Kulinich, Patricia S. Pirbhoy, Arnold R. Palacios, David L. Nelson, Khaleel A. Razak, Iryna M. Ethell
Publikováno v:
Neurobiology of Disease, Vol 162, Iss, Pp 105577-(2022)
Background: Fragile X syndrome (FXS) is a leading genetic cause of autism and intellectual disability with cortical hyperexcitability and sensory hypersensitivity attributed to loss and hypofunction of inhibitory parvalbumin-expressing (PV) cells. Ou
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fa3d2f2e791b992c246f6384bc95158b
https://pubmed.ncbi.nlm.nih.gov/34871737
https://pubmed.ncbi.nlm.nih.gov/34871737