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Autor:
Dubois PC, Trynka G, Francke L, Hunt KA, Rpmanos J, Curtotti A, Zhernakova A, Heap GA, Adàny R, Aromaa A, Bardella MT, Van Den Berg LH, Bockett NA, De La Concha EG, DEma B, Fehrmann RS, Fernandez Aquero M, Fiatal S, Grandone E, Green PM, Groen HJ, Gwilliam R, Houwn RH, Hunt SE, Kaukinen K, Kelleher D, Korponay Szabo I, Kurppa K, Mac Mathuna P, Maki M, Mazzilli MC, McCann OT, Mearin ML, Mein CA, Mirza MM, Mistry V, Mora B, Morley KI, Mulder CJ, Murray JA, Nunez C, Oosterom E, Ophoff RA, Polanco I, Peltonen L, Platteel M, Rybak A, Salomaa V, Schweizer JJ, Sperandeo MP, Tack GJ, Turner G, Veldink JH, Verbeek WH, Weersma RK, Wolters WM, Urcelay E, Cukrowska B, Neuhausen SL, McManus R, Barisani D, Deloukas P, Barrett JC, Saavalainen P, Wijmenga C, Van Heel DA, GRECO, LUIGI
We performed a second-generation genome-wide association study of 4,533 individuals with celiac disease (cases) and 10,750 control subjects. We genotyped 113 selected SNPs with P(GWAS) < 10(-4) and 18 SNPs from 14 known loci in a further 4,918 cases
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od______3730::a9f83733ed613dd5b9f22f2c58da1f02
http://hdl.handle.net/11588/365958
http://hdl.handle.net/11588/365958
