Zobrazeno 1 - 4
of 4
pro vyhledávání: '"William McPheat"'
Publikováno v:
Frontiers in Endocrinology, Vol 10 (2019)
When primitive vertebrates evolved from ancestral members of the animal kingdom and acquired complex locomotive and neurological toolsets, a constant supply of energy became necessary for their continued survival. To help fulfill this need, the endoc
Externí odkaz:
https://doaj.org/article/6173c589185644efb739a6cf12346c4d
Autor:
Nigeste Carter, Anca D. Dobrian, Allison Mathiesen, Michael Brown, Tyree Hamilton, William McPheat
Publikováno v:
International Journal of Molecular Sciences
International Journal of Molecular Sciences, Vol 22, Iss 4640, p 4640 (2021)
International Journal of Molecular Sciences, Vol 22, Iss 4640, p 4640 (2021)
Endothelium has a rich vesicular network that allows the exchange of macromolecules between blood and parenchymal cells. This feature of endothelial cells, along with their polarized secretory machinery, makes them the second major contributor, after
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1be99440543e58c56c1258d4388a7a84
http://europepmc.org/articles/PMC8125116
http://europepmc.org/articles/PMC8125116
Autor:
Joshua M. Turner, Vanessa L. Correll, Allison Mathiesen, Nektarios Barabutis, Eric J. Lehrer, Ryan W. Huyck, Anca D. Dobrian, Li Fang Yang, O. John Semmes, Bronson A. Haynes, William McPheat
Publikováno v:
Arterioscler Thromb Vasc Biol
Objective: Endothelial cells (EC) in obese adipose tissue (AT) are exposed to a chronic proinflammatory environment that may induce a mesenchymal-like phenotype and altered function. The objective of this study was to establish whether endothelial-to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c7a0e3fa9ee0c81eb47eb05382aa1346
https://pubmed.ncbi.nlm.nih.gov/31434495
https://pubmed.ncbi.nlm.nih.gov/31434495
Publikováno v:
Arteriosclerosis, Thrombosis, and Vascular Biology. 33
Adipose tissue (AT) inflammation is key for the development of insulin resistance (IR) and contributes to progression of atherosclerosis. Previous work from our group showed that STAT4 deficiency is protective for IR in obesity and reduces atheroscle
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::c4285f430820e9caf1e1e26faaadcf67
https://doi.org/10.1161/atvb.33.suppl_1.a58
https://doi.org/10.1161/atvb.33.suppl_1.a58