Zobrazeno 1 - 10
of 103
pro vyhledávání: '"William H. Hoffman"'
Publikováno v:
Archives of Endocrinology and Metabolism, Vol 68 (2024)
SUMMARY We present the case of a young adult who had lethargy and significant weight loss for the three weeks before his death. The history of the present illness suggested a prodrome of several weeks, with progressive weakness indicating an advancin
Externí odkaz:
https://doaj.org/article/90119f51af9b4111aa74edb4f490841d
Publikováno v:
JCRPE, Vol 12, Iss 2, Pp 160-167 (2020)
Objective:To determine the time relationships of soluble receptor for glycation end-products (sRAGE), [a decoy of the advanced glycation end-products (AGE)-RAGE axis] and D-lactate, (a metabolite of methylglyoxal) in the inflammatory response to diab
Externí odkaz:
https://doaj.org/article/0c396704d6f44976b229e5d47c06eb84
Autor:
Tomoya Ikeda, Takaki Ishikawa, Naoto Tani, William H. Hoffman, James E. Blum, Carol M. Artlett
Publikováno v:
JCRPE, Vol 12, Iss 2, Pp 160-167 (2020)
Journal of Clinical Research in Pediatric Endocrinology
Journal of Clinical Research in Pediatric Endocrinology
Objective To determine the time relationships of soluble receptor for glycation end-products (sRAGE), [a decoy of the advanced glycation end-products (AGE)-RAGE axis] and D-lactate, (a metabolite of methylglyoxal) in the inflammatory response to diab
Publikováno v:
PLoS ONE, Vol 16, Iss 7, p e0254116 (2021)
PLoS ONE
PLoS ONE
Diabetic ketoacidosis (DKA) is a serious complication of complete insulin deficiency and insulin resistance in Type 1 diabetes (T1D). This results in the body producing high levels of serum ketones in an attempt to compensate for the insulin deficien
Autor:
Dallas Boodhoo, Alexandru Tatomir, Violeta Rus, Cornelia Cudrici, Horea Rus, William H. Hoffman
Publikováno v:
Exp Mol Pathol
There is increasing awareness that in addition to the metabolic crisis of diabetic ketoacidosis (DKA) caused by severe insulin deficiency, the immune inflammatory response is likely an active multicomponent participant in both the acute and chronic i
Autor:
Gregory P. Passmore, William H. Hoffman, Ali S. Calikoglu, Ashley B. Jessup, Mary Beth Grimley, Echo Meyer, Aysenil Belger
Publikováno v:
Journal of Clinical Research in Pediatric Endocrinology
Objective To evaluate the effects of diabetic ketoacidosis (DKA) on neurocognitive functions in children and adolescents presenting with new-onset type 1 diabetes. Methods Newly diagnosed patients were divided into two groups: those with DKA and thos
Autor:
Dallas Boodhoo, Horea Rus, Dries Mulder, Mary G. F. Gilliland, Carol M. Artlett, D. H. T. Tjan, William H. Hoffman, Alvaro Martin, Luis A Ortiz, Alexandru Tatomir
Publikováno v:
Experimental and molecular pathology. 102(3)
Due to the limited data on diabetic ketoacidosis and brain edema (DKA/BE) in children/adolescents and the lack of recent data on adults with type 1 diabetes (T1D), we addressed the question of whether neuroinflammation was present in the fatal DKA of
Autor:
Daniela Cihakova, Monica V. Talor, Monal Sharma, Gregory G. Passmore, Thalachallour Mohanakumar, Noel R. Rose, William H. Hoffman
Publikováno v:
Autoimmunity. 49(3)
Diabetic cardiomyopathy (DC) is an independent phenotype of diabetic cardiovascular disease. The understanding of the pathogenesis of DC in young patients with type 1 diabetes (T1D) is limited. The cardiac insults of diabetic ketoacidosis (DKA) and p
Publikováno v:
Experimental and Molecular Pathology. 93:273-280
Semi-quantitative neuroradiologic studies, quantitative neuron density studies and immunocytochemistry markers of oxidative stress and neuroinflammation indicate neuronal injury and deficits in young patients with chronic poorly controlled type 1 dia
Autor:
Ian R. Mackay, Ingrid M. Outschoorn, Noel R. Rose, William H. Hoffman, Merrill J. Rowley, Monica V. Talor, C. Lynne Burek
Publikováno v:
Autoimmunity. 44:195-200
Using a simple screening technique called regression of offspring on mid-parent (ROMP) to examine the role of IgG subclasses in affected and unaffected siblings of children and adolescents with autoimmune thyroid disease and their parents, both total