Zobrazeno 1 - 10
of 11
pro vyhledávání: '"William H Eschenbacher"'
Autor:
William H Eschenbacher, Moshi Song, Yun Chen, Poonam Bhandari, Peter Zhao, Casey C Jowdy, John T Engelhard, Gerald W Dorn
Publikováno v:
PLoS ONE, Vol 7, Iss 9, p e44296 (2012)
Mitochondrial fusion is essential to organelle homeostasis and organ health. Inexplicably, loss of function mutations of mitofusin 2 (Mfn2) specifically affect neurological tissue, causing Charcot Marie Tooth syndrome (CMT) and atypical optic atrophy
Externí odkaz:
https://doaj.org/article/71f7223d383946cebf9db25c193667f9
Autor:
William H. Eschenbacher
Publikováno v:
Annals of Allergy, Asthma & Immunology. 130:2-3
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::445622faa2675f8ac64f48f7a4e4e17f
https://doi.org/10.1016/j.anai.2022.09.030
https://doi.org/10.1016/j.anai.2022.09.030
Autor:
William H, Eschenbacher
Publikováno v:
Annals of allergy, asthmaimmunology : official publication of the American College of Allergy, Asthma,Immunology. 130(1)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::bda182d461fd18ee54c3acf12c646da0
https://pubmed.ncbi.nlm.nih.gov/36596611
https://pubmed.ncbi.nlm.nih.gov/36596611
Publikováno v:
Medical educationReference. 53(11)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c7f765a6be682e1362e1665c086a42f0
https://pubmed.ncbi.nlm.nih.gov/31650608
https://pubmed.ncbi.nlm.nih.gov/31650608
Publikováno v:
Circulation Research. 111:521-531
Rationale: MicroRNA-499 and other members of the myomiR family regulate myosin isoforms in pressure-overload hypertrophy. miR-499 expression varies in human disease, but results of mouse cardiac miR-499 overexpression are inconsistent, either protect
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7bbba02c64bb6ae6fa32c4f0c4617423
https://doi.org/10.1161/circresaha.112.265736
https://doi.org/10.1161/circresaha.112.265736
Publikováno v:
Circulation Research. 110:958-967
Rationale: MyomiRs miR-499, miR-208a and miR-208b direct cardiac myosin gene expression. Sequence complementarity between miRs and their mRNA targets determines miR effects, but the functional consequences of human myomiR sequence variants are unknow
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::93c976ebc8894f0ae4ec45ece91486ef
https://doi.org/10.1161/circresaha.111.260752
https://doi.org/10.1161/circresaha.111.260752
Autor:
Scot J. Matkovich, Wei Wang, Jeanne M. Nerbonne, Gianluigi Condorelli, Gerald W. Dorn, Lisa E. Dorn, Abhinav Diwan, Yizheng Tu, William H. Eschenbacher
Publikováno v:
Circulation Research. 106:166-175
Rationale : MicroRNA (miR)-133a regulates cardiac and skeletal muscle differentiation and plays an important role in cardiac development. Because miR-133a levels decrease during reactive cardiac hypertrophy, some have considered that restoring miR-13
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::08f9f1bcb04e140284395efb3002b7f8
https://doi.org/10.1161/circresaha.109.202176
https://doi.org/10.1161/circresaha.109.202176
Autor:
Scot J. Matkovich, John T. Engelhard, Casey C. Jowdy, Gerald W. Dorn, William H. Eschenbacher, Stephen J. Warner, Min-Young Kang, Charles F. Clark
Publikováno v:
Circulation research. 108(1)
Rationale: Mitochondria interact via actions of outer and inner membrane fusion proteins. The role of mitochondrial fusion in functioning of the heart, where mitochondria comprise ≈30% of cardiomyocyte volume and their intermyofilament spatial arra
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3a3d0bb8cbaa6f8229554056b169c0b3
https://pubmed.ncbi.nlm.nih.gov/21148429
https://pubmed.ncbi.nlm.nih.gov/21148429
Publikováno v:
Circulation research. 108(1)
Rationale: MicroRNAs (miRs) are expanding our understanding of cardiac disease and have the potential to transform cardiovascular therapeutics. One miR can target hundreds of individual mRNAs, but existing methodologies are not sufficient to accurate
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5227fde55567c301ca55c55dd4f7c1ad
https://pubmed.ncbi.nlm.nih.gov/21212388
https://pubmed.ncbi.nlm.nih.gov/21212388
Autor:
Gerald W. Dorn, Abhinav Diwan, Mark A. Watson, Kenneth B. Margulies, Keith A. Youker, Derek J. Van Booven, William H. Eschenbacher, Guillermo Torre-Amione, Lisa E. Dorn, Scot J. Matkovich
Publikováno v:
Circulation. 119(9)
Background— Much has been learned about transcriptional control of cardiac gene expression in clinical and experimental congestive heart failure (CHF), but less is known about dynamic regulation of microRNAs (miRs) in CHF and during CHF treatment.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0a041711796bc74e347bf48fe56ffbdb
https://pubmed.ncbi.nlm.nih.gov/19237659
https://pubmed.ncbi.nlm.nih.gov/19237659