Zobrazeno 1 - 10
of 58
pro vyhledávání: '"William E. Segar"'
Autor:
William E, Segar
Publikováno v:
Pediatric research. 91(1)
Publikováno v:
The Journal of Pediatrics. 145:584-587
Physicians giving children fluid therapy today seldom encounter overt dehydration or signs of shock that call for rapid and aggressive extracellular fluid (ECF) expansion. Physicians in the era when severe diarrheal dehydration was common first resto
Autor:
Russell W. Chesney, Katia Giugno, Clotilde Druck Garcia, Bernard S. Kaplan, Roberto Correa Chem, William E. Segar
Publikováno v:
Pediatric Nephrology. 14:985-989
Three patients with hemolytic uremic syndrome (HUS) developed peripheral gangrene. Bilateral carotid artery thromboses occurred in one of these patients after recovery from HUS. One patient had a long history of juvenile rheumatoid arthritis. In the
Publikováno v:
Pediatrics. 111(2)
Autor:
William E. Segar
Publikováno v:
Pediatric Nephrology. 5:99-101
Publikováno v:
The Journal of Pediatrics. 147:274-275
Publikováno v:
Pediatrics. 111:424-425
The article by Moritz and Ayus in this month’s issue of Pediatrics 1 noted errors in fluid therapy management causing hyponatremia, brain damage, and death in previously well children who had been hospitalized with an acute illness or for elective
Autor:
Søren Nielsen, Trevor Duke, Laurence Finberg, Malcolm A. Holliday, Russell W. Chesney, Kathryn Maitland, Tamás Dóczi, Mike English, Aaron L. Friedman, Zs Vajda, Endre Sulyok, William E. Segar, Charles R. Newton
Publikováno v:
The Lancet. 363:241
Autor:
Aaron L. Friedman, William E. Segar
Publikováno v:
The Journal of Pediatrics. 94:521-526
Summary Known physiologic mechanisms explain the elevatedblood ADH levels observed in most patients with the syndrome of inappropriate ADH. Therefore the word “inappropriate” is a misnomer. It implies that the mechanisms that regulate ADH release
Autor:
Enid F. Gilbert, Aaron L. Friedman, Rocco Latorraca, Russell W. Chesney, William E. Segar, Kennedy W. Gilchrist
Publikováno v:
American Journal of Nephrology. 3:248-252
Secondary oxalosis is a complication of renal failure. Since oxalate is excreted by the kidney, the severity of oxalate deposition has been directly related to the duration of renal failure. We report a patient with acute renal failure on hemodialysi