Zobrazeno 1 - 10
of 155
pro vyhledávání: '"Wen-I. Lee"'
Publikováno v:
Scientific Reports, Vol 11, Iss 1, Pp 1-10 (2021)
Abstract Systemic juvenile idiopathic arthritis (sJIA) and cryopyrin-associated periodic syndrome (CAPS) share many common manifestations. We aim to identify an applicable method to assist disease discrimination. Inflammatory cytokines were measured
Externí odkaz:
https://doaj.org/article/846d1cacb9f549b9bdd4a5200b82f846
Publikováno v:
Journal of Microbiology, Immunology and Infection, Vol 53, Iss 6, Pp 1014-1020 (2020)
Background: Primary immunodeficiency (PID) accompanying with recurrent respiratory infections is thought to have a devastating effect on lung function. However, the associations between the airway structural abnormalities on chest computed tomography
Externí odkaz:
https://doaj.org/article/ebea20de167e4db999d9517b4c06ce0b
Publikováno v:
Taiwanese Journal of Obstetrics & Gynecology, Vol 60, Iss 4, Pp 791-793 (2021)
Objective: A monochorionic dizygotic (MCDZ) twin is rare, especially when complicated with twin–twin transfusion syndrome (TTTS) and treated by laser therapy. Case report: A pregnancy achieved from oocyte donation and intracytoplasmic sperm injecti
Externí odkaz:
https://doaj.org/article/aee3d54e6b6f479cbd6948f8a9e84e47
Autor:
Meng-Ying Hsieh, Jainn-Jim Lin, Shao-Hsuan Hsia, Jing-Long Huang, Kuo-Wei Yeh, Kuei-Wen Chang, Wen-I. Lee
Publikováno v:
Biomedical Journal, Vol 43, Iss 3, Pp 293-304 (2020)
Background: Defective human TLR3 signaling causes recurrent and refractory herpes simplex encephalitis/encephalopathy. Children with febrile infection-related epilepsy syndrome with refractory seizures may have defective TLR responses. Methods: Child
Externí odkaz:
https://doaj.org/article/cdbf42a69620400eb7a389bf4eb6211a
Autor:
Wen-I. Lee, Jing-Long Huang, Kuo-Wei Yeh, Tang-Her Jaing, Tzou-Yien Lin, Yhu-Chering Huang, Cheng-Hsun Chiu
Publikováno v:
Journal of the Formosan Medical Association, Vol 111, Iss 4, p 236 (2012)
Externí odkaz:
https://doaj.org/article/c265b02d57e740dc922d745e2794a5f7
Autor:
Bing‐Shiau Shang, Chang‐Hung Hsiao, Teng‐Fu Tsao, Yuan‐Ya Liao, Wea‐Lung Lin, Wen‐I Lee, Ko‐Huang Lue
Publikováno v:
Immunity, Inflammation and Disease, Vol 11, Iss 11, Pp n/a-n/a (2023)
Abstract Background Kimura disease (KD) is a rare chronic inflammatory disorder involving the Th2 pathway. Although medical treatment with steroids or other immunosuppressants is available, they may cause developmental issues in the pediatric populat
Externí odkaz:
https://doaj.org/article/d652898d86004475ad601bdb7d28727c
Autor:
Ting-Sheng Lin, Jyh-Hong Lee, Li-Chieh Wang, Yao-Hsu Yang, Yu-Lung Lau, Wen-I Lee, Yu-Tsan Lin, Bor-Luen Chiang, Hsin-Hui Yu
Publikováno v:
Journal of Microbiology, Immunology and Infection, Vol 56, Iss 1, Pp 130-138 (2023)
Background: Chronic granulomatous disease (CGD) is a rare primary immunodeficiency disease characterized by defective neutrophil killing of microbial pathogens and recurrent infections. We aimed to investigate the clinical, genetic features, treatmen
Externí odkaz:
https://doaj.org/article/a21817c590084631bbe0f966436f8793
Autor:
Chao-Yi, Wu, Wen-Lang, Fan, Huang-Yu, Yang, Pi-Shuang, Chu, Pei-Chun, Liao, Li-Chen, Chen, Tsung-Chieh, Yao, Kuo-Wei, Yeh, Liang-Shiou, Ou, Syh-Jae, Lin, Wen-I, Lee, Jing-Long, Huang
Publikováno v:
Journal of Allergy and Clinical Immunology. 151:1123-1131
A dysregulated immune response is a hallmark of autoimmune disorders. Evidence suggests that systemic autoimmune diseases and primary immunodeficiency disorders (PID) may be similar diseases with different clinical phenotypes.This study aimed to inve
Autor:
Wen-I. Lee, Yao-Fan Fang, Jing-Long Huang, Huey-Ling You, Meng-Ying Hsieh, Wan-Ting Huang, Chi-Jou Liang, Chen-Chen Kang, Ting-Shu Wu
Publikováno v:
Journal of Clinical Immunology. 43:717-727
Publikováno v:
Pediatric rheumatology online journal. 20(1)
Background Blau syndrome (BS) is a rare autoinflammatory disorder with NOD2 gain-of-function mutation and characterized by autoactivation of the NFκB pathway. Classically considered a disease of high penetrance, reports on NOD2 mutations underlining