Zobrazeno 1 - 10
of 20
pro vyhledávání: '"Wei-yi Guo"'
Characterization of patients with IgA nephropathy with and without associated minimal change disease
Autor:
Wei-yi Guo, Li-jun Sun, Hong-rui Dong, Guo-qin Wang, Xiao-yi Xu, Wen-rong Cheng, Zhi-rui Zhao, Nan Ye, Yun Liu, Hong Cheng
Publikováno v:
Frontiers in Nephrology, Vol 3 (2023)
IntroductionImmunoglobulin A nephropathy (IgAN) presents various clinical manifestations and pathological phenotypes. Approximately 5% of patients with IgAN present with early onset nephrotic syndrome, mild mesangial lesions, and diffuse foot process
Externí odkaz:
https://doaj.org/article/33b7b22aa2aa4b9a8ad2c8e5f4ccba82
Autor:
Wei-yi Guo, Xiu-ping An, Li-jun Sun, Hong-rui Dong, Wen-rong Cheng, Nan Ye, Guo-qin Wang, Xiao-yi Xu, Zhi-rui Zhao, Hong Cheng
Publikováno v:
Frontiers in Medicine, Vol 9 (2022)
IntroductionIgA nephropathy (IgAN) encompasses a wide range of clinical and histology features. Some patients present without hematuria, with or without hypertension, still rapidly progress in renal function. Renal pathology of this part of patients
Externí odkaz:
https://doaj.org/article/2ccebff68c684ba59ccb362c2e9befbd
Publikováno v:
Kidney International Reports, Vol 6, Iss 2, Pp 404-413 (2021)
Introduction: Immunoglobulin A nephrology (IgAN), characterized by co-deposition of IgA and complement components, is an activation of complement system involved disease. Factor H–related protein 5 (FHR-5) antagonized the ability of factor H to neg
Externí odkaz:
https://doaj.org/article/56eef31909a54fe48af63d7a2909de57
Autor:
Bo-Yang Xu, Jicheng Lv, Li Zhu, Wei-yi Guo, Meng Jia, Hong Zhang, Ya-Ling Zhai, Lijun Liu, Sufang Shi, Pei Chen
Publikováno v:
Nephrology. 25:40-47
Background Immunoglobulin A (IgA) vasculitis with nephritis (IgAVN) and IgA nephropathy (IgAN) are widely considered as related diseases. Considerable evidences support the notion of involvement of complement activation in both IgAVN and IgAN. Our pr
Publikováno v:
Journal of Organometallic Chemistry. 880:341-348
Reaction of R3SnCH2CH(3,5-Me2Pz)2 (R = n-Bu, CH2Ph or Ph; Pz = pyrazol-1-yl) with W(CO)5THF in refluxing THF resulted in the oxidative addition of the inactive R3Sn–C bond to the tungsten atom to yield metal-metal bonded complexes CH2CH(3,5-Me2Pz)2
Autor:
Jicheng Lv, Bo-Yang Xu, Hong Zhang, Li Zhu, Wei-yi Guo, Sufang Shi, Min Wei, Lijun Liu, Xu-jie Zhou
Publikováno v:
Clin J Am Soc Nephrol
Background and objectives IgA nephropathy is the most common primary GN worldwide. Previous research demonstrated that collectin11, an initiator of the complement lectin pathway, was involved in both AKI and chronic tubulointerstitial fibrosis. Here,
Autor:
Lijun Liu, Nicholas R. Medjeral-Thomas, Li Zhu, Jicheng Lv, Matthew C. Pickering, Wei-yi Guo, Sufang Shi, Hannah J. Lomax-Browne, Hong Zhang
Publikováno v:
Kidney International. 94:150-158
IgA nephropathy (IgAN) is a disease associated with activation of the complement system. But the factors influencing complement activation in IgAN are not fully understood. Complement factor H (FH) is an essential negative regulator of complement C3
Publikováno v:
Journal of the American Society of Nephrology. 28:3175-3181
IgA nephropathy (IgAN) is characterized by infections followed by episodic gross hematuria. Deficiency of mannose-binding lectin (MBL) is associated with recurrent infection in many diseases, but controversy exists regarding the role of MBL in IgAN.
Publikováno v:
The American Journal of the Medical Sciences. 353:247-257
The aim of this study was to detect the spectrum of complement activation pathways in circulation and to assess their correlations with clinical and pathologic features in a large lupus nephritis cohort from China.Plasma levels of C1q, mannose-bindin
Autor:
Jicheng Lv, Wei-yi Guo, Li Zhu, Hong Zhang, Zeng-yan Li, Sufang Shi, Si-Jun Meng, Ping Hou, Qing-zhen Liu, Lijun Liu
Publikováno v:
The American journal of the medical sciences. 356(2)
Background In immunoglobulin A nephropathy (IgAN), complement activation occurs in both the systemic circulation and in situ (glomerular). A recent IgAN-genome-wide association study (GWAS) identified 1q32 as an IgAN susceptible locus that contained