Zobrazeno 1 - 10
of 25
pro vyhledávání: '"Wei-Hsuan Tung"'
Autor:
Wei-Hsuan Tung, 董維軒
93
Enterovirus 71 (EV71) is a widespread virus that causes severe and fatal diseases in patients, including circulation failure. The blood vessel will be damaged first, and produced necrosis and inflammation, this phenomenon is called vasculiti
Enterovirus 71 (EV71) is a widespread virus that causes severe and fatal diseases in patients, including circulation failure. The blood vessel will be damaged first, and produced necrosis and inflammation, this phenomenon is called vasculiti
Externí odkaz:
http://ndltd.ncl.edu.tw/handle/20203161802085743122
Publikováno v:
Journal of Cellular Physiology. 226:3316-3329
Oxidative stress became emerged as a key player in the development and progression of many pathological conditions including virus-induced encephalitis. Heme oxygenase-1 (HO-1) plays a crucial role in defending the body against oxidant-induced injury
Autor:
I-Ta Lee, Hsin-Wen Tsai, Yuh-Lien Chen, Wei-June Chen, Chuen-Mao Yang, Wei-Hsuan Tung, Hsi-Lung Hsieh
Publikováno v:
British Journal of Pharmacology. 161:1566-1583
BACKGROUND AND PURPOSE Japanese encephalitis virus (JEV) is a member of the family Flaviviridae and JEV infection is a major cause of acute encephalopathy in children, which destroys cells in the CNS, including astrocytes and neurons. However, the de
Autor:
Chuen-Mao Yang, Wei Hsuan Tung, Chiang-Wen Lee, I-Ta Lee, Shyi-Wu Wang, Jwu Ching Shu, Chih-Chung Lin
Publikováno v:
The American Journal of Pathology. 176:1671-1684
Lipoteichoic acid (LTA) plays a role in the pathogenesis of severe inflammatory responses induced by Gram-positive bacterial infection. Cytosolic phospholipase A(2) (cPLA(2)), cyclooxygenase-2 (COX-2), prostaglandin E(2) (PGE(2)), and interleukin (IL
Publikováno v:
Journal of Cellular Physiology. 224:376-386
Enterovirus 71 (EV71) induces the expression of cyclooxgenase (COX)-2 served as a major neurotoxic factor in CNS injury. However, the mechanisms underlying EV71-initiated intracellular signaling pathways leading to COX-2 expression remain unknown. Th
Autor:
Cheng-Ying Wu, Wei-Hsuan Tung, Hsi-Lung Hsieh, Chuen-Mao Yang, Hui-Hsin Wang, Tze-Shyuan Wang, Chih-Chung Lin
Publikováno v:
Arteriosclerosis, Thrombosis, and Vascular Biology. 29:1594-1601
Objection— Thrombin upregulates expression of several proteins in vascular smooth muscle cells (VSMCs) which may contribute to atherosclerosis. Here, we investigated the mechanisms underlying thrombin-induced EGF receptor (EGFR) expression and its
Publikováno v:
Neurotoxicity Research. 17:344-359
Bacterial infections have been shown to be involved in several inflammatory diseases such as brain inflammation. A major factor for these findings is due to the secretion of pro-inflammatory mediators by host cells triggered by the components release
Publikováno v:
Cellular Signalling. 19:2127-2137
Enterovirus 71 (EV71) is a widespread virus that causes severe and fatal diseases in patients, including circulation failure. The mechanisms underlying EV71-initiated intracellular signaling pathways to influence host cell functions remain unknown. I
Publikováno v:
Journal of cellular physiology. 227(4)
Phenotype transition of vascular smooth muscle cells (VSMCs) is important in vascular diseases, such as atherosclerosis and restenosis. Once released, ATP may promote activation of VSMCs by stimulating cyclooxygenase-2 (COX-2), cytosolic phospholipas
Publikováno v:
Journal of Cellular Biochemistry
Enterovirus 71 (EV71) has been shown to induce cyclooxygenase‐2 (COX‐2) expression in human neuroblastoma SK‐N‐SH cells through the action of MAPKs, NF‐κB, and AP‐1. On the other hand, the transcription factor CREB has also been implicat