Zobrazeno 1 - 10
of 239
pro vyhledávání: '"Wall, M.J."'
Autor:
Privitera, Lucia, Hogg, Ellen L., Lopes, M., Domingos, L.B., Gaestel, M., Muller, Jurgen, Wall, M.J., Corrêa, Sonia A.L.
Yes
A key aim of Alzheimer disease research is to develop efficient therapies to prevent and/or delay the irreversible progression of cognitive impairments. Early deficits in long-term potentiation (LTP) are associated with the accumulation of a
A key aim of Alzheimer disease research is to develop efficient therapies to prevent and/or delay the irreversible progression of cognitive impairments. Early deficits in long-term potentiation (LTP) are associated with the accumulation of a
Externí odkaz:
http://hdl.handle.net/10454/19174
Yes
The ability to either erase or update the memories of a previously learned spatial task is an essential process that is required to modify behaviour in a changing environment. Current evidence suggests that the neural representation of such
The ability to either erase or update the memories of a previously learned spatial task is an essential process that is required to modify behaviour in a changing environment. Current evidence suggests that the neural representation of such
Externí odkaz:
http://hdl.handle.net/10454/17110
Autor:
Wall, M.J., Corrêa, Sonia A.L.
Yes
The activity-regulated cytoskeleton associated protein (Arc/Arg3.1) plays a key role in determining synaptic strength through facilitation of AMPA receptor (AMPAR) endocytosis. Although there is considerable data on the mechanism by which Ar
The activity-regulated cytoskeleton associated protein (Arc/Arg3.1) plays a key role in determining synaptic strength through facilitation of AMPA receptor (AMPAR) endocytosis. Although there is considerable data on the mechanism by which Ar
Externí odkaz:
http://hdl.handle.net/10454/13240
Autor:
Wall, M.J., Collins, D.R., Chery, S.L., Allen, Z.D., Pastuzyn, E.D., George, A.J., Nikolova, V.D., Moy, S.S., Philpot, B.D., Shepherd, J.D., Muller, Jurgen, Ehlers, M.D., Mabb, A.M., Corrêa, Sonia A.L.
Yes
Neuronal activity regulates the transcription and translation of the immediate-early gene Arc/Arg3.1, a key mediator of synaptic plasticity. Proteasomedependent degradation of Arc tightly limits its temporal expression, yet the significance
Neuronal activity regulates the transcription and translation of the immediate-early gene Arc/Arg3.1, a key mediator of synaptic plasticity. Proteasomedependent degradation of Arc tightly limits its temporal expression, yet the significance
Externí odkaz:
http://hdl.handle.net/10454/16201
Autor:
DaSilva, L.L., Wall, M.J., de Almeida, Luciana P., Wauters, S.C., Januario, Y.C., Muller, Jurgen, Corrêa, Sonia A.L.
Yes
The activity-regulated cytoskeleton-associated (Arc) protein control synaptic strength by facilitating AMPA receptor (AMPAR) endocytosis. Here we demonstrate that Arc targets AMPAR to be internalized through a direct interaction with the cla
The activity-regulated cytoskeleton-associated (Arc) protein control synaptic strength by facilitating AMPA receptor (AMPAR) endocytosis. Here we demonstrate that Arc targets AMPAR to be internalized through a direct interaction with the cla
Externí odkaz:
http://hdl.handle.net/10454/8321
Autor:
Mabb, A.M., Je, H.S., Wall, M.J., Robinson, C.G., Larsen, R.S., Qiang, Y., Corrêa, Sonia A.L., Ehlers, M.D.
Activity-dependent gene transcription and protein synthesis underlie many forms of learning-related synaptic plasticity. At excitatory glutamatergic synapses, the immediate early gene product Arc/ Arg3.1 couples synaptic activity to postsynaptic endo
Externí odkaz:
http://hdl.handle.net/10454/8080
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