Zobrazeno 1 - 10
of 85
pro vyhledávání: '"W. Heilig"'
Autor:
Sarah T Suliman, Tasnuva Rashid, Samir Khouzam, Hani M. Wadei, Martin L. Mai, Irtiza Hasan, Razvan M. Chirila, Tambi Jarmi, Hatem Amer, Pablo Moreno Franco, Charles W. Heilig
Publikováno v:
Romanian Journal of Internal Medicine, Vol 59, Iss 1, Pp 10-42 (2021)
Introduction. COVID-19 presents a special challenge to the kidney transplant population. Methods. A systematic review of articles that examined COVID-19 in kidney transplant recipients was performed. Patients’ demographics, clinical, laboratory and
Autor:
Virin Rajiv Neil Ramoutar, Raafat Makary, Leighton R. James, Malleswari Ravi, Charles W. Heilig
Publikováno v:
Case Reports in Nephrology
Case Reports in Nephrology, Vol 2020 (2020)
Case Reports in Nephrology, Vol 2020 (2020)
Prior to the advent of combined antiretroviral therapy (cART), human immunodeficiency virus-associated nephropathy (HIVAN) was inevitably associated with rapidly progressive renal failure and dialysis dependence. HIV-1 seropositive patients often met
Publikováno v:
Case Reports in Nephrology and Dialysis
We report the case of a 22-year-old African American female who presented to another facility for routine follow-up in the 34th week of pregnancy with lower extremity swelling and nephrotic-range proteinuria. Although she was normotensive, it was ini
Autor:
Julia Zimmermann, Tobias Würschum, H. Gütler, W. Heilig, A. Gütler, H. Beck, Carl Friedrich Horst Longin, Stephan C. Bischoff
Publikováno v:
Journal of Cereal Science. 95:103021
FODMAPs are fermentable oligo-, di, and monosaccharides and polyols, which can have adverse effects on human health. Especially in individuals with gastrointestinal disorders such as irritable bowel syndrome, ingestion of FODMAPs may trigger or aggra
Autor:
Leighton R. James, Afu Abdul, Hasan Riaz, N. Stanley Nahman, Jamal Salameh, Charles W. Heilig, Dilip K. Deb
Publikováno v:
American Journal of Nephrology. 38:39-49
Diabetic glomerulosclerosis is characterized by accumulation of extracellular matrix proteins, mesangial expansion, and tubulointerstitial fibrosis. Hyperglycemia accelerates development of the disease, a direct result of increased intracellular gluc
Autor:
Isaac Marin-Valencia, Christopher M. Sinton, Juan M. Pascual, Joao A.G. Duarte, Teodoro Bottiglieri, Levi B. Good, Qian Ma, Charles W. Heilig
Publikováno v:
Neurobiology of Disease, Vol 48, Iss 1, Pp 92-101 (2012)
Brain glucose supplies most of the carbon required for acetyl-coenzyme A (acetyl-CoA) generation (an important step for myelin synthesis) and for neurotransmitter production via further metabolism of acetyl-CoA in the tricarboxylic acid (TCA) cycle.
Publikováno v:
American Journal of Physiology-Renal Physiology. 301:F588-F596
Enhanced GLUT1 expression in mesangial cells plays an important role in the development of diabetic nephropathy by stimulating signaling through several pathways resulting in increased glomerular matrix accumulation. Similarly, enhanced mammalian tar
Autor:
Carmela Monteiro, Charles W. Heilig, Kathleen Othelia Heilig, Thomas L. Saunders, Dilip K. Deb, Anthony Chang, Youli Wang, Frank C. Brosius, Andrew W. Minto
Publikováno v:
American Journal of Physiology-Renal Physiology. 299:F99-F111
Previous work identified an important role for hyperglycemia in diabetic nephropathy (The Diabetes Control and Complications Trial Research Group. N Engl J Med 329: 977–986, 1993; UK Prospective Diabetes Study Group. Lancet 352: 837–853, 1998), a
Autor:
Anthony Chang, Ying Wang, Dimitrina D Pravtcheva, David A. Dean, Dilip K. Deb, Richard C Geiger, Shenglin Chen, Kathleen Othelia Heilig, Youli Wang, Minghui Xiang, Charles W. Heilig, Andrew W. Minto, Martin Schlimme
Publikováno v:
Laboratory Investigation. 90:83-97
Reduced nephron numbers may predispose to renal failure. We hypothesized that glucose transporters (GLUTs) may contribute to progression of the renal disease, as GLUTs have been implicated in diabetic glomerulosclerosis and hypertensive renal disease
Autor:
Albrecht Pfäfflin, Charles W. Heilig, Hans-Ulrich Häring, Erwin Schleicher, Cora Weigert, Katrin Brodbeck
Publikováno v:
Cellular Physiology and Biochemistry. 18:199-210
Previous results indicate that enhanced glucose transporter (GLUT)1 expression mediates the deleterious effects of metabolic and hemodynamic perturbations leading to diabetic kidney disease. First screening for altered gene expression in GLUT1 overex