Výsledky vyhledávání - "W Z Wan, Ngah"

Targeting genes in insulin-associated signalling pathway, DNA damage, cell proliferation and cell differentiation pathways by tocotrienol-rich fraction in preventing cellular senescence of human diploid fibroblasts

Autor: L W, Durani, F, Jaafar, J K, Tan, K, Tajul Arifin, Y A, Mohd Yusof, W Z, Wan Ngah, S, Makpol

Publikováno v: La Clinica terapeutica. 166(6)

Tocotrienols have been known for their antioxidant properties besides their roles in cellular signalling, gene expression, immune response and apoptosis. This study aimed to determine the molecular mechanism of tocotrienol-rich fraction (TRF) in prev

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::329d6381a90341a79463b86927f2c668
https://pubmed.ncbi.nlm.nih.gov/26794818

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Tocotrienol-rich fraction prevents cellular aging by modulating cell proliferation signaling pathways

Autor: S C, Khor, Y A, Mohd Yusof, W Z, Wan Ngah, S, Makpol

Publikováno v: La Clinica terapeutica. 166(2)

Vitamin E has been suggested as nutritional intervention for the prevention of degenerative and age-related diseases. In this study, we aimed to elucidate the underlying mechanism of tocotrienol-rich fraction (TRF) in delaying cellular aging by targe

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::9f9a8c5fee530407da62b9db2b75ef65
https://pubmed.ncbi.nlm.nih.gov/25945449

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Comparable down-regulation of TYR, TYRP1 and TYRP2 genes and inhibition of melanogenesis by tyrostat, tocotrienol-rich fraction and tocopherol in human skin melanocytes improves skin pigmentation

Autor: S, Makpol, F A, Jam, N A, Rahim, S C, Khor, Z, Ismail, Y A M, Yusof, W Z, Wan Ngah

Publikováno v: La Clinica terapeutica. 165(1)

Antioxidant has been recognized to inhibit UV-induced melanogenesis. This study aimed to elucidate the molecular mechanism of tyrostat, tocopherol and tocotrienol-rich fraction in inhibiting melanogenesis in human skin melanocytes.Primary culture of

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::d79d50ce0077aae5ab35716c0c53d3b5
https://pubmed.ncbi.nlm.nih.gov/24589959

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Long-term tocotrienol supplementation and glutathione-dependent enzymes during hepatocarcinogenesis in the rat

Autor: A, Rahmat, W Z, Wan Ngah, A, Gapor, B A, Khalid

Publikováno v: Asia Pacific journal of clinical nutrition. 2(3)

The effects of long-term administration of tocotrienol on hepatocarcinogenesis in rats induced by diethyl nitrosamine (DEN) and 2-acetylaminofluorene (AAF) were investigated by the determination of plasma and liver gamma-glutamyl transpeptidase (GGT)

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::ccbdc9461901c609d57e0139798a40e5
https://pubmed.ncbi.nlm.nih.gov/24352144

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Long term Tai Chi exercise reduced DNA damage and increased lymphocyte apoptosis and proliferation in older adults

Autor: J A, Goon, A H, Noor Aini, M, Musalmah, M Y, Yasmin Anum, W Z, Wan Ngah

Publikováno v: The Medical journal of Malaysia. 63(4)

Effect of Tai Chi exercise on the level of DNA damage using the comet assay, lymphocyte viability and frequency of sister chromatid exchange (SCE) were determined in adults aged above 45. Tai Chi participants of 7 years (n=35), showed higher level of

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::e500a0c58e75ffb9ee8968dadf49462a
https://pubmed.ncbi.nlm.nih.gov/19385493

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Apoptosis changes and SA-beta galactosidase expression in stress-induced premature senescence (SIPS) model of human skin fibroblasts

Autor: N, Abdul Rahim, S, Makpol, K H, Chua, Y A Mohd, Yusof, G Md, Top, W Z Wan, Ngah

Publikováno v: The Medical journal of Malaysia. 63

Stress-induced premature senescence (SIPS) model is in vitro model of cellular aging. In this study, apoptosis was evaluated in SIPS model and in replicative senescent fibroblasts. We also compared the activity of senescence-associated beta-galactosi

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::8efb17bda86fe019aba2c848e667fe3a
https://pubmed.ncbi.nlm.nih.gov/19024989

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