Zobrazeno 1 - 9
of 9
pro vyhledávání: '"W R, Reenstra"'
Autor:
J. A. Buras, W. R. Reenstra
Publikováno v:
Neurological Research. 29:127-131
Ischemia/reperfusion injury plays a central role in the development of tissue injury during multiple central nervous system diseases including acute stroke. Neutrophil adhesion to the endothelium indicates a major component of ischemia/reperfusion pa
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b3601ab26a25873d225d37b1d7b901d8
https://doi.org/10.1179/016164107x174147
https://doi.org/10.1179/016164107x174147
Publikováno v:
The American journal of pathology. 159(3)
Oxidative stress increases endothelial mannose-binding lectin (MBL) binding and activates the lectin complement pathway (LCP). However, the molecular mechanism of MBL binding to the endothelium after oxidative stress is unknown. Intermediate filament
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::40bcbd459e63d2467c59a7432ac15975
https://pubmed.ncbi.nlm.nih.gov/11549596
https://pubmed.ncbi.nlm.nih.gov/11549596
Erk and PI-3 kinase are necessary for collagen binding and actin reorganization in corneal epithelia
Publikováno v:
Investigative ophthalmologyvisual science. 41(11)
It was recently shown that phosphatidylinositol-(PI)3 kinase is upregulated in wounded rabbit corneal epithelia. Extracellular signal-regulated kinase (erk)-1 and -2 proteins and PI-3 kinase were activated in embryonic corneal epithelia after 1-hour
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::9d6592ae70bd98c3f6affe8d44f9326c
https://pubmed.ncbi.nlm.nih.gov/11006227
https://pubmed.ncbi.nlm.nih.gov/11006227
Autor:
C D, Collard, A, Väkevä, M A, Morrissey, A, Agah, S A, Rollins, W R, Reenstra, J A, Buras, S, Meri, G L, Stahl
Publikováno v:
The American journal of pathology. 156(5)
The complement system plays an important role in mediating tissue injury after oxidative stress. The role of mannose-binding lectin (MBL) and the lectin complement pathway (LCP) in mediating complement activation after endothelial oxidative stress wa
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::44fa7e3d78dbf0ec8dd88aae0e36939f
https://pubmed.ncbi.nlm.nih.gov/10793066
https://pubmed.ncbi.nlm.nih.gov/10793066
Publikováno v:
The Anatomical record. 254(3)
Previous studies demonstrated that corneal epithelial cells isolated without basal lamina respond to extracellular matrix (ECM) in an actin dependent manner; the basal cell surface flattens and the actin cortical mat reorganizes. We hypothesize that
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::3f73aed7cbf55a0300365ef624e92c22
https://pubmed.ncbi.nlm.nih.gov/10096666
https://pubmed.ncbi.nlm.nih.gov/10096666
Publikováno v:
The Anatomical record. 248(1)
Exogenous TNF alpha, IL-1 beta, M-CSF, and GM-CSF all stimulate growth of macrophages arising in explanted fetal rat lungs. The present study examines the intrinsic availability of these factors in intact and organ-cultured lungs and utilizes express
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::c7ba553dd4932cf52d48045b8be9426d
https://pubmed.ncbi.nlm.nih.gov/9143672
https://pubmed.ncbi.nlm.nih.gov/9143672
Autor:
W. R. Reenstra
Publikováno v:
Academic Emergency Medicine. 11:517-518
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::4193947f2c1e9f086a9ead06ae4d5968
https://doi.org/10.1197/j.aem.2004.02.062
https://doi.org/10.1197/j.aem.2004.02.062
Autor:
W. R. Reenstra
Publikováno v:
Academic Emergency Medicine. 12:8-8
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::3e9755b44002f33b4a63a5fc43264613
https://doi.org/10.1197/j.aem.2005.03.006
https://doi.org/10.1197/j.aem.2005.03.006
Autor:
W. R. Reenstra
Publikováno v:
Academic Emergency Medicine. 11:440-440
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::6dd7a007f437cc9feb2cfd947e24a680
https://doi.org/10.1197/j.aem.2004.02.023
https://doi.org/10.1197/j.aem.2004.02.023