Zobrazeno 1 - 10
of 13
pro vyhledávání: '"W D, Ehringer"'
Publikováno v:
European Journal of Clinical Investigation. 27:48-54
Central to the pathogenesis of atherosclerosis is an abnormally functioning endothelium and a consequent loss of vascular integrity. These abnormalities may be induced by haemodynamic factors, biochemical substances, and also by oxidatively modified
Publikováno v:
Proceedings of the National Academy of Sciences. 98:15260-15263
Several independent experiments suggest that cell walls of Bacillus subtilis are protonated during growth. When cells were grown in the presence of fluorescein-labeled dextran to saturate the cell walls, centrifuged, and suspended in PBS, fluorescenc
Autor:
W. D. Ehringer, Frederick N. Miller
Publikováno v:
Inflammation Research. 48:393-398
Objective and Design: We have developed a continuous monitoring system that will quantify lymphocyte extravasation or intercellular permeability with intracellular calcium measurements in a single preparation.¶Materials: Human microvascular endothel
Publikováno v:
Molecular and cellular biochemistry. 221(1-2)
Fructose-1,6-diphosphate (FDP) is a glycolytic intermediate which has been theorized to increase the metabolic activity of ischemic tissues. Here we examine the effects of externally applied FDP on cardiomyocyte uptake and metabolism. Adult rat cardi
Publikováno v:
Molecular and cellular biochemistry. 210(1-2)
Fructose-1,6-diphosphate (FDP) is a glycolytic intermediate which has been used an intervention in various ischemic conditions for two decades. Yet whether FDP can enter the cell is under constant debate. In this study we examined membrane permeabili
Publikováno v:
Inflammation. 24(2)
Bradykinin and alpha-thrombin both increase endothelial macromolecular permeability, however the mechanism for this effect is unclear. Human umbilical vein endothelial cell (HUVEC) permeability to human serum albumin was increased by 1 microM alpha-t
Publikováno v:
Microcirculation (New York, N.Y. : 1994). 6(4)
Filamentous actin (F-actin) plays a central role in maintaining endothelial barrier function. Thrombin and histamine, two inflammatory mediators that increase endothelial permeability, can alter F-actin production and distribution. In this study, we
Publikováno v:
Microcirculation (New York, N.Y. : 1994). 5(1)
Interleukin-2 (IL-2) induces protein leakage from the microcirculation and activates lymphocytes; yet it is unclear how it alters endothelial barrier function. Here, we report of a new continuous monitoring system that allows for the continuous measu
Publikováno v:
Inflammation. 21(3)
alpha-Thrombin (AT) and bradykinin (BK) are endogenous mediators that are released during an inflammatory response, and could have a synergistic effect on endothelial permeability. Human umbilical vein endothelial cells (HUVEC) were grown on Transwel
Publikováno v:
Journal of cellular physiology. 167(3)
alpha-Thrombin, bradykinin, and histamine are endogenous mediators that increase endothelial permeability. We examined the mechanism by which these three vasoactive mediators could alter permeability to albumin of human umbilical vein endothelial cel