Zobrazeno 1 - 10
of 39
pro vyhledávání: '"W A, Groenewegen"'
Publikováno v:
Journal of Lipid Research, Vol 35, Iss 6, Pp 1012-1025 (1994)
We have identified a new truncated apolipoprotein B (apoB) that provides insights into the interaction of apoB with apo[a] and with lipids. Both total and LDL-cholesterol were below the 5th percentile in the proband; Lp[a] was 28 mg/dl. Four other af
Externí odkaz:
https://doaj.org/article/e9445b3cdcf444109b103e8b6bf7915c
Publikováno v:
Nederlands tijdschrift voor geneeskunde. 162
As of May 2018, the use of personal data in medical research is regulated under the General Data Protection Regulation (GDPR). While, as before, in principle patients' consent for the use of their personal data is still required, exemptions for medic
The Effect of Shadows on Performing Stereo visual Pointing Tasks: Is Shadow-Free Open Surgery Ideal?
Autor:
Elisabeth D. Kaptein, Marco W. M. Groenewegen, Jenny Dankelman, Arjan J. Knulst, Jesse van Dongen
Publikováno v:
LEUKOS. 8:111-122
Surgical overhead luminaires and head-mounted luminaires are important tools for surgeons. The optical design of these luminaires focuses on providing shadow free light. Shadows, however, are reported as important cues for depth perception in mono- a
Autor:
Toon A.B. van Veen, Nicolaas de Jonge, Harold V.M. van Rijen, Marti F.A. Bierhuizen, André C. Linnenbank, Markus A. Engelen, Jacques M.T. de Bakker, Mohamed Boulaksil, J. H. Kirkels, Marc A. Vos, Mera Stein, Stephan K.G. Winckels, András Varró, John A. Jansen, Matthijs F.M. van Oosterhout, W. Antoinette Groenewegen
Publikováno v:
European journal of heart failure, 12(9), 913-921. Wiley-Blackwell
Aims Sudden arrhythmogenic cardiac death is a major cause of mortality in patients with congestive heart failure (CHF). To investigate determinants of the increased arrhythmogenic susceptibility, we studied cardiac remodelling and arrhythmogenicity i
Publikováno v:
Volume 5A: 39th Mechanisms and Robotics Conference.
Advanced robotic hand prostheses are praised for their impressive robust and fine grasping capabilities generated from intricate systems. Nevertheless, a high demand remains for grasping mechanisms that are mechanically simple, lightweight, and cheap
Autor:
Bobby P. C. Koeleman, Habo J. Jongsma, Andreas Busjahn, Peter W. de Leeuw, Jan M. Ruijter, Bart Kok, Maria Schipper, Connie R. Bezzina, Wilko Spiering, Mehran Firouzi, W. Antoinette Groenewegen
Publikováno v:
Journal of hypertension, 24(2), 325-330. Lippincott Williams and Wilkins
OBJECTIVE: Gap junctions, formed by connexins (Cx), are important in the regulation of vascular tone. Previously, we reported two closely linked polymorphisms (-44G --> A and +71A --> G) within regulatory regions of the gene for Cx40, a major connexi
Autor:
Takashi Yokota, Koji Sasaki, Hisashi Yokoshiki, Naomasa Makita, W. Antoinette Groenewegen, Hiroyuki Tsutsui, Tomoaki Murakami
Publikováno v:
Heart Rhythm. 2(10):1128-1134
Congenital atrial standstill has been linked to SCN5A. Incomplete penetrance observed in atrial standstill has been attributed in part to the digenic inheritance of polymorphisms in the atrial-specific gap junction connexin 40 (Cx40) in conjunction w
Autor:
Connie R. Bezzina, W. Antoinette Groenewegen, Arthur A.M. Wilde, Theo M. Hoorntje, Martin B. Rook, J. Peter van Tintelen, Habo J. Jongsma, Marcel M.A.M. Mannens
Publikováno v:
Cardiovascular research, 57(4), 1072-1078. Oxford University Press
Cardiovascular Research, 57(4), 1072-1078. Oxford University Press
Cardiovascular Research, 57(4), 1072-1078. Oxford University Press
The Long QT3 syndrome is associated with mutations in the cardiac sodium channel gene SCN5A. Objective: The aim of the present study was the identification and functional characterization of a mutation in a family with the long QT3 syndrome. Methods:
Autor:
Marti F.A. Bierhuizen, Habo J. Jongsma, Saskia Vliex, Shirley C.M. van Amersfoorth, W. Antoinette Groenewegen
Publikováno v:
Cardiovascular Research. 46:511-522
Objectives: The gap junction protein connexin40 (Cx40) is differentially expressed during embryonic development and in adult tissues, for which the molecular basis is unknown. In order to elucidate the molecular mechanisms controlling Cx40 expression