Zobrazeno 1 - 10
of 243
pro vyhledávání: '"Vreni Schneider"'
Autor:
Eleonora Patsenker, Philip Sachse, Andrea Chicca, María Salomé Gachet, Vreni Schneider, Johan Mattsson, Christian Lanz, Mathias Worni, Andrea de Gottardi, Mariam Semmo, Jochen Hampe, Clemens Schafmayer, Rudolf Brenneisen, Jürg Gertsch, Felix Stickel, Nasser Semmo
Publikováno v:
International Journal of Molecular Sciences, Vol 16, Iss 4, Pp 7057-7076 (2015)
The endocannabinoid (EC) system is implicated in many chronic liver diseases, including hepatitis C viral (HCV) infection. Cannabis consumption is associated with fibrosis progression in patients with chronic hepatitis C (CHC), however, the role of E
Externí odkaz:
https://doaj.org/article/ef6088cf5268434d8814fd7d69b3c5d0
Autor:
Johan Mattsson, Felix Stickel, Jochen Hampe, Eleonora Patsenker, Rudolf Brenneisen, Christian Lanz, Mathias Worni, Mariam Semmo, Philip Sachse, Vreni Schneider, Nasser Semmo, Andrea Chicca, Jürg Gertsch, María Salomé Gachet, Andrea De Gottardi, Clemens Schafmayer
Publikováno v:
International Journal of Molecular Sciences
Volume 16
Issue 4
Pages 7057-7076
Patsenker, Eleanora; Sachse, Philip; Chicca, Andrea; Gachet Otanez, Maria Salomé; Schneider, Vreni; Mattsson, Johan; Lanz, Christian; Worni, Mathias; De Gottardi, Andrea; Semmo, Mariam; Hampe, Jochen; Schafmayer, Clemens; Brenneisen, Rudolf Max; Gertsch, Jürg; Stickel, Felix; Semmo, Nasser (2015). Elevated levels of endocannabinoids in chronic hepatitis C may modulate cellular immune response and hepatic stellate cell activation. International journal of molecular sciences, 16(4), pp. 7057-7076. Molecular Diversity Preservation International MDPI 10.3390/ijms16047057
International Journal of Molecular Sciences, Vol 16, Iss 4, Pp 7057-7076 (2015)
Volume 16
Issue 4
Pages 7057-7076
Patsenker, Eleanora; Sachse, Philip; Chicca, Andrea; Gachet Otanez, Maria Salomé; Schneider, Vreni; Mattsson, Johan; Lanz, Christian; Worni, Mathias; De Gottardi, Andrea; Semmo, Mariam; Hampe, Jochen; Schafmayer, Clemens; Brenneisen, Rudolf Max; Gertsch, Jürg; Stickel, Felix; Semmo, Nasser (2015). Elevated levels of endocannabinoids in chronic hepatitis C may modulate cellular immune response and hepatic stellate cell activation. International journal of molecular sciences, 16(4), pp. 7057-7076. Molecular Diversity Preservation International MDPI 10.3390/ijms16047057
International Journal of Molecular Sciences, Vol 16, Iss 4, Pp 7057-7076 (2015)
The endocannabinoid (EC) system is implicated in many chronic liver diseases, including hepatitis C viral (HCV) infection. Cannabis consumption is associated with fibrosis progression in patients with chronic hepatitis C (CHC), however, the role of E
Autor:
Matthias Stoll, Helmut K. Seitz, Gunda Millonig, Felix Stickel, Vreni Schneider, Matthias Ocker, Abbas Agaimy, Eleonora Patsenker, Sebastian Mueller, Rudolf Brenneisen, Till Wissniowski
Publikováno v:
Molecular Medicine. 17:1285-1294
The cannabinoid system (CS) is implicated in the regulation of hepatic fibrosis, steatosis and inflammation, with cannabinoid receptors 1 and 2 (CB1 and CB2) being involved in regulation of pro- and antifibrogenic effects. Daily cannabis smoking is a
Autor:
Gerald Niedobitek, Simon Goodman, Vreni Schneider, Felix Stickel, Yury Popov, Hans Sägesser, Eleonora Patsenker, Detlef Schuppan, M. Ledermann
Publikováno v:
Hepatology. 50:1501-1511
The vitronectin receptor integrin alphavbeta3 promotes angiogenesis by mediating migration and proliferation of endothelial cells, but also drives fibrogenic activation of hepatic stellate cells (HSCs) in vitro. Expecting antifibrotic synergism, we s
Publikováno v:
Los Angeles Times -- Southern California Edition; 04/20/95, Vol. 114 Issue 138, pC6
Publikováno v:
Journal of Hepatology. 45:786-796
Background/Aims Mammalian target of rapamycin (mTOR) signalling is central in the activation of hepatic stellate cells (HSCs), the key source of extracellular matrix (ECM) in fibrotic liver. We tested the therapeutic potential of the mTOR inhibitor r
Oral imatinib treatment reduces early fibrogenesis but does not prevent progression in the long term
Autor:
Nicolas Widmer, Markus Neef, Bertrand Rochat, M. Ledermann, Juerg Reichen, Vreni Schneider, H. Saegesser, Laurent A. Decosterd
Publikováno v:
Journal of Hepatology. 44:167-175
Background/Aims: Transactivated hepatic stellate cells (HSCs) represent the key source of extra cellular matrix (ECM) in fibrotic liver. Imatinib, a potent inhibitor of the PDGF receptor tyrosine kinase, reduces HSC proliferation and fibrogenesis whe