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pro vyhledávání: '"Vivian Y Lim"'
Autor:
Jennifer B. Kahn, Lee Melvin Peralta, Laurie H. Rubel, Vivian Y. Lim, Shiyan Jiang, Beth Herbel-Eisenmann
Publikováno v:
Educational Technology & Society, Vol 25, Iss 4, Pp 80-92 (2022)
In this paper, we introduce Notice, Wonder, Feel, Act, and Reimagine (NWFAR) to promote social justice in data science (DS) education. NWFAR draws on intersectional feminist DS to scaffold critical perspectives towards systems of power and oppression
Externí odkaz:
https://doaj.org/article/b8ce23504d3547d6b584f6f313e17325
Autor:
Vivian Y. Lim, Lee Melvin M. Peralta, Laurie H. Rubel, Shiyan Jiang, Jennifer B. Kahn, Beth Herbel-Eisenmann
Publikováno v:
ZDM – Mathematics Education. 55:109-118
The mathematical medium of data visualization and other data representations (DV) has served as a primary means of communicating about the COVID-19 crisis. DVs about the pandemic are highly visible across news journalism and include an increasingly i
Autor:
Runfeng Miao, Vivian Y. Lim, Neeharika Kothapalli, Yifan Ma, Julia Fossati, Sandra Zehentmeier, Ruifeng Sun, João P. Pereira
Publikováno v:
Frontiers in Immunology, Vol 11 (2020)
Studies over the last couple of decades have shown that hematopoietic stem cells (HSCs) are critically dependent on cytokines such as Stem Cell Factor and other signals provided by bone marrow niches comprising of mesenchymal stem and progenitor cell
Externí odkaz:
https://doaj.org/article/c380d9d71bc3427d9f2354e20212fbd4
Autor:
Sandra Zehentmeier, Vivian Y. Lim, Yifan Ma, Julia Fossati, Takeshi Ito, Yawen Jiang, Alexei V. Tumanov, Ho-Joon Lee, Lukas Dillinger, Jihyun Kim, Krisztian Csomos, Jolan E. Walter, Jungmin Choi, João P. Pereira
Publikováno v:
Sci Immunol
Gain-of-function (GOF) mutations in CXCR4 cause WHIM (warts, hypogammaglobulinemia, infections, and myelokathexis) syndrome, characterized by infections, leukocyte retention in bone marrow (BM), and blood leukopenias. B lymphopenia is evident at earl
Autor:
Vivian Y Lim, Xing Feng, Runfeng Miao, Sandra Zehentmeier, Nathan Ewing-Crystal, Moonyoung Lee, Alexei V Tumanov, Ji Eun Oh, Akiko Iwasaki, Andrew Wang, Jungmin Choi, João P Pereira
Publikováno v:
Life Science Alliance. 6:e202301924
Systemic inflammation halts lymphopoiesis and prioritizes myeloid cell production. How blood cell production switches from homeostasis to emergency myelopoiesis is incompletely understood. Here, we show that lymphotoxin-β receptor (LTβR) signaling
Autor:
Julia Fossati, João Pereira, Ruifeng Sun, Sandra Zehentmeier, Vivian Y. Lim, Yifan Ma, Runfeng Miao, Neeharika Kothapalli
Publikováno v:
Frontiers in Immunology, Vol 11 (2020)
Frontiers in Immunology
Frontiers in Immunology
Studies over the last couple of decades have shown that hematopoietic stem cells (HSCs) are critically dependent on cytokines such as Stem Cell Factor and other signals provided by bone marrow niches comprising of mesenchymal stem and progenitor cell
Autor:
Vivian Y. Lim, Koichi Ikuta, João Pereira, Dietmar Herndler-Brandstetter, Erin Nevius, Ellen R. Richie, Richard A. Flavell, Hans Reimer Rodewald, Tatsuki Sugiyama, Takashi Nagasawa, Shizue Tani-ichi, Takahiro Hara, Ana Cordeiro Gomes, Susan M. Schlenner
Publikováno v:
Immunity. 45:1219-1231
Hematopoietic stem cells (HSCs) self-renew in bone marrow niches formed by mesenchymal progenitors and endothelial cells expressing the chemokine CXCL12, but whether a separate niche instructs multipotent progenitor (MPP) differentiation remains uncl
Publikováno v:
Advances in immunology. 134
B lymphocytes develop from hematopoietic stem cells (HSCs) in specialized bone marrow niches composed of rare mesenchymal-lineage stem/progenitor cells (MSPCs) and sinusoidal endothelial cells. These niches are defined by function and location: MSPCs
B lymphocytes develop from hematopoietic stem cells (HSCs) in specialized bone marrow niches composed of rare mesenchymal lineage stem/progenitor cells (MSPCs) and sinusoidal endothelial cells. These niches are defined by function and location: MSPCs
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::03ede225c4e0883541db740a5541e276
https://doi.org/10.1016/bs.ai.2017.02.001
https://doi.org/10.1016/bs.ai.2017.02.001
Publikováno v:
PLoS ONE
PLoS ONE, Vol 10, Iss 1, p e0113824 (2015)
PLoS ONE, Vol 10, Iss 1, p e0113824 (2015)
V(D)J recombination creates antibody light chain diversity by joining a Vκ gene segment with one of four Jκ segments. Two Jκ germline-transcript (GT) promoters control Vκ-Jκ joining, but the mechanisms that govern Jκ choice are unclear. Here, w