Zobrazeno 1 - 10
of 36
pro vyhledávání: '"Viveka, Vadyvaloo"'
Autor:
Xiao-Peng Guo, Hai-Qin Yan, Wenhui Yang, Zhe Yin, Viveka Vadyvaloo, Dongsheng Zhou, Yi-Cheng Sun
Publikováno v:
eLife, Vol 12 (2023)
Multiple genetic changes in the enteric pathogen Yersinia pseudotuberculosis have driven the emergence of Yesinia pestis, the arthropod-borne, etiological agent of plague. These include developing the capacity for biofilm-dependent blockage of the fl
Externí odkaz:
https://doaj.org/article/d48c7df966eb487d8c44313d0be349ba
Publikováno v:
Parasites & Vectors, Vol 13, Iss 1, Pp 1-12 (2020)
Abstract Background Yersinia pestis is the flea-transmitted etiological agent of bubonic plague. Sylvatic plague consists of complex tripartite interactions between diverse flea and wild rodent species, and pathogen strains. Transmission by flea bite
Externí odkaz:
https://doaj.org/article/196a406a2fcf41d28781aff625bb5975
Autor:
Amelia R. Silva-Rohwer, Kiara Held, Janelle Sagawa, Nicolas L. Fernandez, Christopher M. Waters, Viveka Vadyvaloo
Publikováno v:
mBio, Vol 12, Iss 4 (2021)
ABSTRACT Plague-causing Yersinia pestis is transmitted through regurgitation when it forms a biofilm-mediated blockage in the foregut of its flea vector. This biofilm is composed of an extracellular polysaccharide substance (EPS) produced when cyclic
Externí odkaz:
https://doaj.org/article/8b298241fa20412c80f6b7c1df85c1a9
Publikováno v:
Journal of Bacteriology.
Mutations enhancing c-di-GMP biosynthesis drove the evolution of Y. pestis to flea-borne transmissibility. c-di-GMP-dependent biofilm-mediated blockage of the flea foregut enables regurgitative transmission of Y. pestis by fleabite. The Y. pestis dig
Autor:
Wenhui Yang, Hai-Qin Yan, Xiao-Peng Guo, Zhe Yin, Viveka Vadyvaloo, Dongsheng Zhou, Yi-Cheng Sun
Publikováno v:
eLife. 12
Multiple genetic changes in the enteric pathogen Yersinia pseudotuberculosis have driven the emergence of Yesinia pestis, the arthropod-borne, etiological agent of plague. These include developing the capacity for biofilm-dependent blockage of the fl
Publikováno v:
Pathogens, Vol 9, Iss 12, p 1039 (2020)
Yersinia pestis, the causative agent of plague, has a complex infectious cycle that alternates between mammalian hosts (rodents and humans) and insect vectors (fleas). Consequently, it must adapt to a wide range of host environments to achieve succes
Externí odkaz:
https://doaj.org/article/7f7822aebe524932b227f02a30423a27
Autor:
Xiao-Peng Guo, Hai-Qin Yan, Wenhui Yang, Zhe Yin, Viveka Vadyvaloo, Dongsheng Zhou, Yi-Cheng Sun
Multiple genetic changes in the enteric pathogenYersinia pseudotuberculosishave driven the emergence ofYesinia pestis, the arthropod-borne, etiological agent of plague. These include developing the capacity for biofilm-dependent blockage of the flea
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e57ca9c749835fc61a4db05fb2773934
https://doi.org/10.1101/2022.10.24.512971
https://doi.org/10.1101/2022.10.24.512971
Publikováno v:
Journal of Medical Entomology. 57:1997-2001
Plague, caused by the flea-transmitted bacterial pathogen Yersinia pestis, is primarily a disease of wild rodents distributed in temperate and tropical zones worldwide. The ability of Y. pestis to develop a biofilm blockage that obstructs the flea fo
Autor:
Jennifer K. DeMarco, Matthew B. Lawrenz, Sylvie Garneau-Tsodikova, Robert D. Perry, Amanda Brady, Thomas E. Kehl-Fie, Viveka Vadyvaloo, Phoenix A Gray, Sarah L. Price
Publikováno v:
Proceedings of the National Academy of Sciences. 118
Yersinia pestis causes human plague and colonizes both a mammalian host and a flea vector during its transmission cycle. A key barrier to bacterial infection is the host's ability to actively sequester key biometals (e.g., iron, zinc, and manganese)