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of 10
pro vyhledávání: '"Vishakha S Bhave"'
Autor:
Shashikiran Donthamsetty, Vishakha S Bhave, Wendy M Mars, William C Bowen, Anne Orr, Meagan M Haynes, Chuanyue Wu, George K Michalopoulos
Publikováno v:
PLoS ONE, Vol 8, Iss 9, p e74625 (2013)
Particularly interesting new cysteine-histidine-rich protein (PINCH) protein is part of the ternary complex known as the IPP (integrin linked kinase (ILK)-PINCH-Parvin-α) complex. PINCH itself binds to ILK and to another protein known as Rsu-1 (Ras
Externí odkaz:
https://doaj.org/article/69ecc133f5954a81b574b6c64850e1aa
Autor:
Shashikiran Donthamsetty, Vishakha S. Bhave, John R. Latendresse, Harihara M. Mehendale, Michael L. Cunningham
Publikováno v:
Toxicology and Applied Pharmacology. 251:173-180
We have previously reported that among the other death proteins, hepatic secretory phospholipase A{sub 2} (sPLA{sub 2}) is a leading mediator of progression of liver injury initiated by CCl{sub 4} in rats. The aim of our present study was to test the
Autor:
Vishakha S. Bhave, Harihara M. Mehendale, John R. Latendresse, Shashikiran Donthamsetty, Mayurranjan S. Mitra
Publikováno v:
Toxicology and Applied Pharmacology. 230:327-337
The objective was to investigate if the hepatotoxic sensitivity in nonalcoholic steatohepatitic mice to acetaminophen (APAP) is due to downregulation of nuclear receptor PPARalpha via lower cell division and tissue repair. Male Swiss Webster mice fed
Autor:
Vishakha S. Bhave, Harihara M. Mehendale, Shashikiran Donthamsetty, Levan Muskhelishvili, John R. Latendresse
Publikováno v:
Toxicology and Applied Pharmacology. 228:225-238
Previous studies have shown that injury initiated by toxicants progresses even after most of the toxicant is eliminated from the body. One mechanism of progression of injury is the extracellular appearance of hydrolytic enzymes following leakage or u
Autor:
Vishakha S, Bhave, Shashikiran, Donthamsetty, John R, Latendresse, Michael L, Cunningham, Harihara M, Mehendale
Publikováno v:
Toxicology and applied pharmacology. 251(3)
We have previously reported that among the other death proteins, hepatic secretory phospholipase A₂ (sPLA₂) is a leading mediator of progression of liver injury initiated by CCl₄ in rats. The aim of our present study was to test the hypothesis
Publikováno v:
Toxicology and applied pharmacology. 228(2)
Our previous study [Bhave, V. S., Donthamsetty, S., Latendresse, J. R., Muskhelishvili, L., and Mehendale, H. M. 2008-this issue. Secretory phospholipase A(2) mediates progression of acute liver injury in the absence of sufficient COX-2. Toxicol Appl
Autor:
Vishakha S. Bhave, Harihara M. Mehendale, John R. Latendresse, Shashikiran Donthamsetty, Mayurranjan S. Mitra
Publikováno v:
Hepatology (Baltimore, Md.). 45(2)
This study tested whether hepatic steatosis sensitizes liver to toxicant-induced injury and investigated the potential mechanisms of hepatotoxic sensitivity. Male Sprague-Dawley rats were fed a methionine- and choline-deficient diet for 31 days to in
Autor:
vishakha S bhave, harihara mehendale, John R. Latendresse, shashikiran donthamsetty, mayur mitra
Publikováno v:
The FASEB Journal. 20