Zobrazeno 1 - 10 of 12 pro vyhledávání: '"Violante Olivari"'
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Akademický článek
NCOA4-mediated ferritinophagy in macrophages is crucial to sustain erythropoiesis in mice
Autor: Antonella Nai, Maria Rosa Lidonnici, Giorgia Federico, Mariateresa Pettinato, Violante Olivari, Federica Carrillo, Simonetta Geninatti Crich, Giuliana Ferrari, Clara Camaschella, Laura Silvestri, Francesca Carlomagno
Publikováno v: Haematologica, Vol 106, Iss 3 (2020)
The Nuclear Receptor Coactivator 4 (NCOA4) promotes ferritin degradation and Ncoa4-ko mice in C57BL/6 background show microcytosis and mild anemia, aggravated by iron deficiency. To understand tissue specific contribution of NCOA4-mediated ferritinop
Externí odkaz: https://doaj.org/article/b4271343b7a34463b76c59669b198260
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5
Transferrin receptor 2 (Tfr2) genetic deletion makes transfusion-independent a murine model of transfusion-dependent β-thalassemia
Autor: Simona Maria Di Modica, Emanuele Tanzi, Violante Olivari, Maria Rosa Lidonnici, Mariateresa Pettinato, Alessia Pagani, Francesca Tiboni, Valeria Furiosi, Laura Silvestri, Giuliana Ferrari, Stefano Rivella, Antonella Nai
beta-thalassemia is a genetic disorder caused by mutations in the beta-globin gene, and characterized by anemia, ineffective erythropoiesis and iron overload. Patients affected by the most severe transfusion-dependent form of the disease (TDT) requir
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b7f84de63c240feecfe8aee67fc25240
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6
NCOA4-mediated ferritinophagy in macrophages is crucial to sustain erythropoiesis in mice
Autor: Maria Rosa Lidonnici, Giorgia Federico, Giuliana Ferrari, Clara Camaschella, Federica Carrillo, Laura Silvestri, Antonella Nai, Francesca Carlomagno, Violante Olivari, Mariateresa Pettinato, Simonetta Geninatti Crich
Publikováno v: Haematologica (Roma) (2020).
info:cnr-pdr/source/autori:Antonella Nai 1, Maria Rosa Lidonnici 2, Giorgia Federico 3, Mariateresa Pettinato 1, Violante Olivari 1, Federica Carrillo 4, Simonetta Geninatti Crich 5, Giuliana Ferrari 2, Clara Camaschella 1, Laura Silvestri 1, Francesca Carlomagno 6/titolo:NCOA4-mediated Ferritinophagy in Macrophages Is Crucial to Sustain Erythropoiesis in Mice/doi:/rivista:Haematologica (Roma)/anno:2020/pagina_da:/pagina_a:/intervallo_pagine:/volume
Haematologica
Nuclear receptor coactivator 4 (NCOA4) promotes ferritin degradation and Ncoa4-ko mice in a C57BL/6 background show microcytosis and mild anemia, aggravated by iron deficiency. To understand tissue-specific contributions of NCOA4-mediated ferritinoph
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fabad8656b46dc6aa3a4d10665b992f0
http://hdl.handle.net/11588/872932
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7
Correcting b-thalassemia by combined therapies that restrict iron and modulate erythropoietin activity
Autor: Hagit Domev, Despina Sitara, Nir Shapir, Garry A. Neil, Mariateresa Pettinato, Giuliana Ferrari, Emir O'Hara, Kevin A. Munoz, Antonella Nai, Maria Rosa Lidonnici, Shuling Guo, Stefano Rivella, Vania Lo Presti, Carla Casu, Simona Maria Di Modica, Violante Olivari, Sheri L. Booten, Alison Liu, Reem Miari, Mariam Aghajan, Inbal Zafir-Lavie
Publikováno v: Blood
β-Thalassemia intermedia is a disorder characterized by ineffective erythropoiesis (IE), anemia, splenomegaly, and systemic iron overload. Novel approaches are being explored based on the modulation of pathways that reduce iron absorption (ie, using
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::01f29510b23b4111ef44430637443e8f
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8
Rebalancing Iron Homeostasis and Erythropoiesis through Tfr2 Inhibition for Correction of Anemia in CKD
Autor: Alessia Pagani, Francesca Tiboni, Violante Olivari, Giuliana Ferrari, Shuling Guo, Antonella Nai, Maria Rosa Lidonnici, Mariateresa Pettinato, Mariam Aghajan, Simona Maria Di Modica, Laura Silvestri
Publikováno v: Blood. 138:940-940
Background Transferrin Receptor 2 (TFR2) is a protein expressed in the liver and in the erythroid compartment. Hepatic TFR2 activates the transcription of hepcidin, the master regulator of iron homeostasis, and its inactivation causes iron overload.
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::400ff8b96daff7f23affc1077b749ad6
https://doi.org/10.1182/blood-2021-148384
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9
Tfr2 Genetic Deletion Makes Transfusion-Independent a Murine Model of Transfusion-Dependent β-Thalassemia
Autor: Emanuele Tanzi, Maria Rosa Lidonnici, Laura Silvestri, Stefano Rivella, Alessia Pagani, Violante Olivari, Antonella Nai, Simona Maria Di Modica, Mariateresa Pettinato, Giuliana Ferrari, Francesca Tiboni
Publikováno v: Blood. 138:575-575
β-thalassemia is an autosomal recessive disorder due to mutations in the β-globin gene, that leads to defective production of hemoglobin (Hb) and red blood cells (RBC). The main features of the disease are anemia, ineffective erythropoiesis and iro
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::7af972dd26af24c2b39e0d8fbc28dcdc
https://doi.org/10.1182/blood-2021-148419
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10
Tamoxifen erythroid toxicity revealed by studying the role of nuclear receptor co-activator 4 in erythropoiesis
Autor: Francesca Carlomagno, Mariateresa Pettinato, Laura Silvestri, Giorgia Federico, Violante Olivari, Antonella Nai
Publikováno v: Haematologica (Online) 104 (2019): e383–e384. doi:10.3324/haematol.2019.224857
info:cnr-pdr/source/autori:Nai A.; Pettinato M.; Federico G.; Olivari V.; Carlomagno F.; Silvestri L./titolo:Tamoxifen erythroid toxicity revealed by studying the role of nuclear receptor co-activator 4 in erythropoiesis/doi:10.3324%2Fhaematol.2019.224857/rivista:Haematologica (Online)/anno:2019/pagina_da:e383/pagina_a:e384/intervallo_pagine:e383–e384/volume:104
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Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d225659038c945660541e4262b826387
https://doi.org/10.3324/haematol.2019.224857
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