Zobrazeno 1 - 10
of 25
pro vyhledávání: '"Viola Marschall"'
Autor:
David M. Richards, Viola Marschall, Katharina Billian-Frey, Karl Heinonen, Christian Merz, Mauricio Redondo Müller, Julian P. Sefrin, Matthias Schröder, Jaromir Sykora, Harald Fricke, Oliver Hill, Christian Gieffers, Meinolf Thiemann
Publikováno v:
Journal for ImmunoTherapy of Cancer, Vol 7, Iss 1, Pp 1-13 (2019)
Abstract Background Glucocorticoid-induced TNFR-related protein (TNFRSF18, GITR, CD357), expressed by T cells, and its ligand (TNFSF18, GITRL), expressed by myeloid populations, provide co-stimulatory signals that boost T cell activity. Due to the im
Externí odkaz:
https://doaj.org/article/04921d3723f8400dab5dcc210d3292d0
Autor:
Meinolf Thiemann, David M. Richards, Karl Heinonen, Michael Kluge, Viola Marschall, Christian Merz, Mauricio Redondo Müller, Tim Schnyder, Julian P. Sefrin, Jaromir Sykora, Harald Fricke, Christian Gieffers, Oliver Hill
Publikováno v:
Frontiers in Oncology, Vol 8 (2018)
Tumor necrosis factor receptor superfamily member 7 (TNFRSF7, CD27), expressed primarily by T cells, and its ligand CD27L (TNFSF7, CD70) provide co-stimulatory signals that boost T cell activation, differentiation, and survival. Agonistic stimulation
Externí odkaz:
https://doaj.org/article/025eb8d7f2eb4ce1b1593f12beb4631b
Publikováno v:
Neoplasia: An International Journal for Oncology Research, Vol 17, Iss 6, Pp 481-489 (2015)
Second mitochondria-derived activator of caspase (Smac) mimetics are considered as promising anticancer therapeutics that are currently under investigation in early clinical trials. They induce apoptosis by antagonizing inhibitor of apoptosis protein
Externí odkaz:
https://doaj.org/article/adf9a956f129483ba1af5d37586ae4e2
Publikováno v:
International Journal of Blood Transfusion and Immunohematology. 13:1-8
Aims: To assess potential changes of pharmacological activities of a novel normal immunoglobulin for intravenous administration from pooled normal plasma (IVIG). Methods: We assessed the impact of the SARS-CoV-2 pandemic on the level and activity of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::11af618dca9eedc81780995886b1a678
https://doi.org/10.5348/100076z02ch2023ra
https://doi.org/10.5348/100076z02ch2023ra
Autor:
Simone Fulda, Albert C. Ludolph, Klaus-Michael Debatin, Franz Rödel, Stephanie Hehlgans, Sri HariKrishna Vellanki, Liane Wagner, Silvia Cristofanon, Sabine Karl, Viola Marschall, Claudia Jennewein, Rebecca Berger
PDF file-54KB
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5944f77f33dad565e1758c59fb6563d0
https://doi.org/10.1158/1535-7163.22497226
https://doi.org/10.1158/1535-7163.22497226
Autor:
Simone Fulda, Albert C. Ludolph, Klaus-Michael Debatin, Franz Rödel, Stephanie Hehlgans, Sri HariKrishna Vellanki, Liane Wagner, Silvia Cristofanon, Sabine Karl, Viola Marschall, Claudia Jennewein, Rebecca Berger
PDF file-82KB
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::049e96508f920250e7fd2cf555adcd50
https://doi.org/10.1158/1535-7163.22497232.v1
https://doi.org/10.1158/1535-7163.22497232.v1
Autor:
Valeska Helfinger, Bernhard Brüne, Tobias Schmid, Nina Henke, Viola Marschall, Ajay M. Shah, Michael M. Kunze, Flávia Rezende, Karen E. Anderson, Simone Fulda, Ilka Wittig, Ralf P. Brandes, Juliana Heidler, Florian Freiherr von Gall, Katrin Schröder, Heinfried H. Radeke
Publikováno v:
Proceedings of the National Academy of Sciences of the United States of America
Significance The stereotype of ROS produced by NADPH oxidases as cause of malignant diseases persists in a generalized manner. In fact, high levels of ROS formation could be harmful in the context of a disease process. This study demonstrates that lo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8a3edddb41ffc6e6854f63e77f262ef7
https://doi.org/10.1073/pnas.2020152118
https://doi.org/10.1073/pnas.2020152118
Autor:
Jaromir Sykora, Mauricio Redondo Müller, Meinolf Thiemann, Oliver Hill, David M. Richards, Christian Gieffers, Christian Merz, Karl Heinonen, Harald Fricke, Tim Schnyder, Viola Marschall
Publikováno v:
Journal of Immunotherapy. 41:385-398
CD40 ligand (TNFSF5/CD154/CD40L), a member of the tumor necrosis factor (TNF) superfamily is a key regulator of the immune system. The cognate receptor CD40 (TNFRSF5) is expressed broadly on antigen-presenting cells and many tumor types, and has emer
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6d0a8811004b002a8c10842d1f6b4e4d
https://doi.org/10.1097/cji.0000000000000246
https://doi.org/10.1097/cji.0000000000000246
Autor:
Oliver Hill, Christian Merz, Meinolf Thiemann, Christian Gieffers, David M. Richards, Julian P. Sefrin, Katharina Billian-Frey, Mauricio Redondo Müller, Karl Heinonen, Harald Fricke, Jaromir Sykora, Matthias Schröder, Viola Marschall
Publikováno v:
Journal for ImmunoTherapy of Cancer, Vol 7, Iss 1, Pp 1-13 (2019)
Journal for Immunotherapy of Cancer
Journal for Immunotherapy of Cancer
Background Glucocorticoid-induced TNFR-related protein (TNFRSF18, GITR, CD357), expressed by T cells, and its ligand (TNFSF18, GITRL), expressed by myeloid populations, provide co-stimulatory signals that boost T cell activity. Due to the important r
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::26a4544601aef382ac432bcd4c5d9ff0
https://doi.org/10.1186/s40425-019-0671-4
https://doi.org/10.1186/s40425-019-0671-4
Autor:
Heinfried H Radecke, Juliana Heidler, Valeska Helfinger, Tobias Schmid, Nina Henke, Bernhard Brüne, Viola Marschall, Florian Freiherr von Gall, Katrin Schröder, Simone Fulda, Michael M. Kunze, Karen S. Anderson, Ajay M. Shah, Ralf P. Brandes, Ilka Wittig
Reactive oxygen species (ROS) can cause cellular damage and are thought to promote cancer-development. Nevertheless, under physiological conditions, all cells constantly produce ROS, either as chemical by-products or for signaling purpose. During dif
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f5e084fac162a4b467b8411d32f07a3c