Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Vincent Genty"'
Publikováno v:
Bio tribune magazine. 34:22-32
Le systeme erythroide humain est responsable du transport d’oxygene. Il comprend la moelle osseuse, les globules rouges, le systeme reticulo-endothelial et les cellules produisant l’EPO au niveau du rein. Ces cellules renales disposent de capteur
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::916e235a9e86ee5091ac024888cbf01d
https://doi.org/10.1007/s11834-010-0139-9
https://doi.org/10.1007/s11834-010-0139-9
Autor:
Olivier Hermine, Muriel Tafflet, Flavia Guillem, Christian Palierne, Vincent Genty, Jean-Pierre Cervetti, Gabriela Fumagalli, Françoise Van Lierde, Jean-Claude Lapostolle, Philippe Noirez, Jean-François Toussaint, A Frey, Marie-Philippe Rousseaux-Blanchi, Xavier Jouven, Laurie-Anne Marquet, G. Dine
Publikováno v:
Biochimie. 119
The HFE gene encodes a protein involved in iron homeostasis; individuals with mutations in both alleles develop hemochromatosis. 27% of the French population is heterozygous for mutations in this gene. We found that 80% of the French athletes who won
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b87368aa88f28f586c976d36e00a5428
https://pubmed.ncbi.nlm.nih.gov/26416567
https://pubmed.ncbi.nlm.nih.gov/26416567
Publikováno v:
Leukemia Research. 28:307-313
Resistance to glucocorticoids (GCs) frequently appears during treatment of hematological malignancies. This study investigates the phenotypical alterations observed in human myeloma cell sublines resistant to glucocorticoids. Using the RPMI8226 cell
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::de14a65a938e99ed1f2c207fd68cc762
https://doi.org/10.1016/j.leukres.2003.08.009
https://doi.org/10.1016/j.leukres.2003.08.009
Autor:
Cecilia Montañez, Dominique Mornet, Gonzalo Aranda, Vincent Genty, Bulmaro Cisneros, Alvaro Rendon, Flor Marquez
Publikováno v:
Neuroscience Letters. 213:107-110
The expression of dystrophin-protein 71 (Dp71) was investigated during nerve growth factor (NGF) induced differentiation of PC12 cells. A semi-quantitative reverse transcription-polymerase chain reaction (RT-PCR) assay was designed to measure Dp71 mR
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::da7ef4abd839fed443216cb815f33343
https://doi.org/10.1016/0304-3940(96)12863-9
https://doi.org/10.1016/0304-3940(96)12863-9
Publikováno v:
Bio Tribune Magazine. 26
Les outils biologiques permettent d’explorer de maniere significative la physiologie et la pathologie du muscle. Le demembrement progressif du fonctionnement musculaire, grâce aux travaux realises pour comprendre les myopathies, a fait progresser
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::a651c218da0f783cfaf3a2bd46b727b0
https://doi.org/10.1007/s11834-008-0053-6
https://doi.org/10.1007/s11834-008-0053-6
Publikováno v:
Cancer Biology and Therapy
Cancer Biology and Therapy, Taylor & Francis, 2005, 4 (8), pp.832-839
Cancer Biology and Therapy, Taylor & Francis, 2005, 4 (8), pp.832-839
Nuclear morphological alterations associated with glucocorticoid resistance in human myeloma were evaluated by image cytometry in three human myeloma RPMI 8226 cell sub-lines. Resistance was induced by drug selection using prednisone (8226p), methylp
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c65909206ad61fec6664c9db8a36ea42
https://hal.archives-ouvertes.fr/hal-00110888
https://hal.archives-ouvertes.fr/hal-00110888
Autor:
Yves Rehn, William Mendes, Nadia Ali Ammar, Vincent Genty, Albert Najman, Said Brahimi, G. Dine
Publikováno v:
Blood. 106:1331-1331
The potential cardiotoxicity of chemotherapic drugs is well known. For example anthracycline-based regimens are extremely effective for various hematological malignancies. The main disadvantage is cardiotoxicity particularly, in elderly patients who
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::4c7d6d9802be4e05cc2543e486b168ff
https://doi.org/10.1182/blood.v106.11.1331.1331
https://doi.org/10.1182/blood.v106.11.1331.1331