Zobrazeno 1 - 10
of 24
pro vyhledávání: '"Vincent Dammai"'
Publikováno v:
BioTechniques, Vol 38, Iss 6, Pp 864-868 (2005)
Externí odkaz:
https://doaj.org/article/7f020d8f29924b40801e345b681341df
Supplementary Figure 2 from Endothelial Function of von Hippel-Lindau Tumor Suppressor Gene: Control of Fibroblast Growth Factor Receptor Signaling
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5c89a0c20eeeba9182b6cb6177e2a1ac
https://doi.org/10.1158/0008-5472.22370933.v1
https://doi.org/10.1158/0008-5472.22370933.v1
Supplementary Figure 4 from Endothelial Function of von Hippel-Lindau Tumor Suppressor Gene: Control of Fibroblast Growth Factor Receptor Signaling
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9fd2c3d47ae31d8ba9fcf1804719d092
https://doi.org/10.1158/0008-5472.22370927.v1
https://doi.org/10.1158/0008-5472.22370927.v1
Supplementary Figure 1 from Endothelial Function of von Hippel-Lindau Tumor Suppressor Gene: Control of Fibroblast Growth Factor Receptor Signaling
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1339cd7ce86ab96cbb08d27c25e3c07a
https://doi.org/10.1158/0008-5472.22370936.v1
https://doi.org/10.1158/0008-5472.22370936.v1
Supplementary Figure 3 from Endothelial Function of von Hippel-Lindau Tumor Suppressor Gene: Control of Fibroblast Growth Factor Receptor Signaling
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::21205feb8019d989d250cf3f20866fed
https://doi.org/10.1158/0008-5472.22370930
https://doi.org/10.1158/0008-5472.22370930
Publikováno v:
Naunyn-Schmiedeberg's Archives of Pharmacology. 388:143-152
Among the anti-tumor genes (tumor suppressors and metastasis suppressors), the von-Hippel Lindau gene and the Nm23 family of genes are among the more intriguing ones. Both are small (long and short forms of VHL are 30 and 19 kD, respectively, and Nm2
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::81b27002639674a47779e6f2b1d3f490
https://doi.org/10.1007/s00210-014-1002-4
https://doi.org/10.1007/s00210-014-1002-4
Publikováno v:
Molecular and Cellular Biology. 30:3779-3794
Mutations in the human von Hippel-Lindau (VHL) gene are the cause of VHL disease that displays multiple benign and malignant tumors. The VHL gene has been shown to regulate angiogenic potential and glycolic metabolism via its E3 ubiquitin ligase func
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::28790dd11ff448dec03cecd174cba3b1
https://doi.org/10.1128/mcb.01578-09
https://doi.org/10.1128/mcb.01578-09
Publikováno v:
Molecular and Cellular Biochemistry. 329:35-44
The metastasis suppressor gene Nm23 is highly conserved from yeast to human, implicating a critical developmental function. Studies in cultured mammalian cells have identified several potential functions, but many have not been directly verified in v
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b9ebac200e5102ae7ede85e4fd6737be
https://doi.org/10.1007/s11010-009-0112-7
https://doi.org/10.1007/s11010-009-0112-7
Publikováno v:
Carcinogenesis
To understand the role of hypoxia-inducible factor (HIF)-2alpha in regulating sensitivity of renal cancer cells to tumor necrosis factor-related apoptosis inducing ligand (TRAIL)-induced apoptosis, we transfected wild-type and mutant von Hippel Linda
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::42cf8414d05f4bbce8f264f8f81bc52c
https://doi.org/10.1093/carcin/bgn132
https://doi.org/10.1093/carcin/bgn132
Publikováno v:
Journal of Biological Chemistry. 281:12069-12080
The tumor suppressor VHL (von Hippel-Lindau protein) serves as a negative regulator of hypoxia-inducible factor-alpha subunits. However, accumulated evidence indicates that VHL may play additional roles in other cellular functions. We report here a n
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a8907a89a65642a008ee5e028df1eb68
https://doi.org/10.1074/jbc.m511621200
https://doi.org/10.1074/jbc.m511621200